Accelerated idioventricular rhythm

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Accelerated idioventricular rhythm
Classification and external resources
12 lead EKG shows a slow and wide complexes with intermittent narrow complex beats. The 5th and 10th beats are sinus node originated complexes (sinus rhythm) and close examination of these beats will give a clue to understand the cause of the wide complex rhythm. ST elevation indicates an acute myocardial infarction. The wide complex beats represent accelerated idioventricular rhythm which is usually seen following reperfusion in acute MI
MeSH D016170

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Overview

The rate of cardiac contraction is determined by the intrinsic rate of depolarisation of the cardiac cells. In normal hearts the sinoatrial node in the atria depolarises at a rate of 70 beats per minute. This suppresses the intrinsic depolarisation of the other parts of the heart.

The accelerated idioventricular rhythm occurs when depolarisation rate of a normally suppressed focus increases to above that of the "higher order" focuses (the sinoatrial node and the atrioventricular node). This most commonly occurs in the setting of a sinus bradycardia.[4]

Accelerated Idioventricular Rhythms are ectopic ventricular rhythms at rates between 40 bpm and 100 to 120 bpm. The ventricular origin of this rhythm can be demonstrated by the usual EKG criteria which include AV dissociation, fusion, and capture complexes.

The incidence of Accelerated Idioventricular Rhythms following acute MI is reported to be between 8 and 36%. This rhythm can also be seen in patients with primary myocardial disease, hypertensive, rheumatic, and congenital heart disease. Digoxin may cause of accelerated idioventricular rhythm.

EKG characteristics

  • Regular rhythm at a rate of 40 to 100-120 BPM.
  • QRS complexes are abnormal and wide.
  • The ventricular complexes are usually but not necessarily dissociated from the P waves.
  • Ventricular capture and fusion beats are common.

Differential Diagnosis

  1. Because of its slower rate it may resemble NSR. Look for numerous fusion beats. The term accelerated isorhythmic ventricular rhythm has been suggested.
  2. Must be distinguished from junctional tachycardia with preexisting IVCDs. But in these patients there are no fusion or capture beats.

Clinical Correlation

  1. Seen in both AMIs and IMIs.
  2. Commonly seen following reperfusion.
  3. Usually occurs during sinus bradycardia.
  4. May also be caused by digitalis.

AIVR appears similar to ventricular tachycardia but is benign and doesn't need any treatment.

References

  1. Hammill S. C. Electrocardiographic diagnoses: Criteria and definitions of abnormalities, Chapter 18, MAYO Clinic, Concise Textbook of Cardiology, 3rd edition, 2007 ISBN 0-8493-9057-5
  2. Engelen DJ, Gressin V, Krucoff MW, Theuns DA, Green C, Cheriex EC, Maison-Blanche P, Dassen WR, Wellens HJ, and Gorgels AP. Usefulness of frequent arrhythmias after epicardial recanalization in anterior wall acute myocardial infarction as a marker of cellular injury leading to poor recovery of left ventricular function. Am J Cardiol 2003 Nov 15; 92(10) 1143-9. PMID 14609586.
  3. Ilia R, Zahger D, Cafri C, Abu-Ful A, Weinstein JM, Yaroslavtsev S, Gilutz H, and Amit G. Predicting survival with reperfusion arrhythmias during primary percutaneous coronary intervention for acute myocardial infarction. Isr Med Assoc J 2007 Jan; 9(1) 21-3. PMID 17274350
  4. http://www.emedicine.com/med/topic12.htm
  5. Hampton, John (1997). The ECG Made Easy.

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Acknowledgement and Attribution Regarding Sources of Content

Some of the initial content on this page may be incorporated in part from copyleft sources in the public domain including wikis such as Wikipedia and AskDrWiki. Drug information for patients came from the The National Library of Medicine. Infectious disease information may have come from the Centers for Disease Control (CDC). Differential Diagnoses are drawn from clinicians as well as an amalgamation of 3 sources: 1.The Disease Database; 2. Kahan, Scott, Smith, Ellen G. In A Page: Signs and Symptoms. Malden, Massachusetts: Blackwell Publishing, 2004:3; 3. Sailer, Christian, Wasner, Susanne. Differential Diagnosis Pocket. Hermosa Beach, CA: Borm Bruckmeir Publishing LLC, 2002:7 .

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