Tricuspid stenosis causes

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Causes of Tricuspid Stenosis

There are at least 4 conditions that are typically attributed with the obstruction of the nativetricuspid valve.

  1. Rheumatic tricuspid stenosis:
    • Diffuse thickening of the leaflets occur. Fusion of the commissures may or may not occur.
    • Chordae tendineae may become thickened and shortened
    • As a result of the dense collagen and elastic fibers that make up leaflet tissue, the normal leaflet layers become significantly distorted
  2. Carcinoid heart disease:
    • Fibrous white plaques located on the valvular and mural endocardium are characteristic presentations of carcinoid valve lesions
    • Valve leaflets become thick, rigid and smaller in area
    • Atrial and ventricular surfaces of the valve structure contain fibrous tissue proliferation
  3. Congenital tricuspid stenosis:
    • More common in infants
    • Lesions may present in a number of different ways, either singularly or in any combination of the following:
      • Incompletely developed leaflets
      • Shortened or malformed chordae
      • Small annuli
      • Papillary muscles of abnormal size and number
  4. Infective endocarditis:
    • Stenosis may develop as a result of large infected vegetation obstructing the opening of the tricuspid valve
    • This condition is uncommon
  • Unusual and rare causes:
    • Fabry disease
    • Giant blood cysts
  • Other conditions may mimic tricuspid stenosis, obstructing flow through the valve:
    • Supravalvular obstruction from congenital diaphragms
    • Intracardiac or extracardiac tumors
    • Thrombosis or emboli
    • Large endocarditis vegetations
    • Other conditions that impair right-sided filling
      • Constrictive pericarditis
      • Restrictive Cardiomyopathy

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