Hypoaldosteronism causes: Difference between revisions
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===Life-threatening Causes=== | ===Life-threatening Causes=== | ||
Life-threatening causes of hypoaldosteronism include:<ref name="pmid12704285">{{cite journal |vauthors=LaBan MM, Whitmore CE, Taylor RS |title=Bilateral adrenal hemorrhage after anticoagulation prophylaxis for bilateral knee arthroplasty |journal=Am J Phys Med Rehabil |volume=82 |issue=5 |pages=418–20 |year=2003 |pmid=12704285 |doi=10.1097/01.PHM.0000064741.97586.E4 |url=}}</ref> | Life-threatening causes of hypoaldosteronism include:<ref name="pmid12704285">{{cite journal |vauthors=LaBan MM, Whitmore CE, Taylor RS |title=Bilateral adrenal hemorrhage after anticoagulation prophylaxis for bilateral knee arthroplasty |journal=Am J Phys Med Rehabil |volume=82 |issue=5 |pages=418–20 |year=2003 |pmid=12704285 |doi=10.1097/01.PHM.0000064741.97586.E4 |url=}}</ref> | ||
*Adrenal infarction | *[[Adrenal gland|Adrenal]] [[infarction]] | ||
*Bilateral adrenal hemorrhage(caused by trauma, anticoagulant therapy, or coagulation disorders). | *[[Adrenal hemorrhage|Bilateral adrenal hemorrhage]](caused by [[trauma]], [[anticoagulant therapy]], or [[coagulation disorders]]). | ||
*Cancerous destruction of the adrenal gland, secondary to infiltrative or metastatic disease. | *Cancerous destruction of the [[adrenal gland]], secondary to infiltrative or [[metastatic]] [[disease]]. | ||
*Tubercular and fungal destruction of adrenal glands. | *Tubercular and [[fungal]] destruction of [[Adrenal glands|adrenal glands.]] | ||
===Common Causes=== | ===Common Causes=== | ||
Common causes of | Common causes of hypoaldosteronism include:<ref>Sailer, Christian, Wasner, Susanne. Differential Diagnosis Pocket. Hermosa Beach, CA: Borm Bruckmeir Publishing LLC, 2002:14-15</ref> | ||
* Diabetic nephropathy | * [[Diabetic nephropathy]] | ||
* Acute glomerulonephritis | * [[Acute glomerulonephritis]] | ||
* [[Autoimmune]]/[[Idiopathic]] | * [[Autoimmune]]/[[Idiopathic]] | ||
* [[Tuberculosis]] | * [[Tuberculosis]] | ||
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===Less common causes=== | ===Less common causes=== | ||
Less common causes of | Less common causes of hypoaldosteronism include: | ||
*Sarcoidosis | *[[Sarcoidosis]] | ||
*Amyloidosis | *[[Amyloidosis]] | ||
*Fungal infections | *[[Fungal infections]] | ||
*AIDS complications | *[[AIDS]] complications | ||
*Hemochromatosis | *[[Hemochromatosis]] | ||
*Polyglandular endocrine syndromes | *Polyglandular endocrine syndromes | ||
*Adrenoleukodystrophy | *Adrenoleukodystrophy | ||
*Adrenomyelodystrophy | *Adrenomyelodystrophy | ||
===Drugs causing hypoaldosteronism=== | ===Drugs causing hypoaldosteronism=== | ||
Other less common causes of hypoaldosteronism include drugs such as:<ref name="pmid2483440">{{cite journal |vauthors=Missale C, Lombardi C, De Cotiis R, Memo M, Carruba MO, Spano PF |title=Dopaminergic receptor mechanisms modulating the renin-angiotensin system and aldosterone secretion: an overview |journal=J. Cardiovasc. Pharmacol. |volume=14 Suppl 8 |issue= |pages=S29–39 |year=1989 |pmid=2483440 |doi= |url=}}</ref><ref name="pmid18331727">{{cite journal |vauthors=Akizuki O, Inayoshi A, Kitayama T, Yao K, Shirakura S, Sasaki K, Kusaka H, Matsubara M |title=Blockade of T-type voltage-dependent Ca2+ channels by benidipine, a dihydropyridine calcium channel blocker, inhibits aldosterone production in human adrenocortical cell line NCI-H295R |journal=Eur. J. Pharmacol. |volume=584 |issue=2-3 |pages=424–34 |year=2008 |pmid=18331727 |doi=10.1016/j.ejphar.2008.02.001 |url=}}</ref><ref name="IkedaIsaka2012">{{cite journal|last1=Ikeda|first1=Keiichi|last2=Isaka|first2=Tsuyoshi|last3=Fujioka|first3=Kouki|last4=Manome|first4=Yoshinobu|last5=Tojo|first5=Katsuyoshi|title=Suppression of Aldosterone Synthesis and Secretion by Channel Antagonists|journal=International Journal of Endocrinology|volume=2012|year=2012|pages=1–6|issn=1687-8337|doi=10.1155/2012/519467}}</ref><ref name="pmid744152">{{cite journal |vauthors=McKenna TJ, Island DP, Nicholson WE, Liddle GW |title=The effects of potassium on early and late steps in aldosterone biosynthesis in cells of the zona glomerulosa |journal=Endocrinology |volume=103 |issue=4 |pages=1411–6 |year=1978 |pmid=744152 |doi=10.1210/endo-103-4-1411 |url=}}</ref> | Other less common causes of hypoaldosteronism include [[drugs]] such as:<ref name="pmid2483440">{{cite journal |vauthors=Missale C, Lombardi C, De Cotiis R, Memo M, Carruba MO, Spano PF |title=Dopaminergic receptor mechanisms modulating the renin-angiotensin system and aldosterone secretion: an overview |journal=J. Cardiovasc. Pharmacol. |volume=14 Suppl 8 |issue= |pages=S29–39 |year=1989 |pmid=2483440 |doi= |url=}}</ref><ref name="pmid18331727">{{cite journal |vauthors=Akizuki O, Inayoshi A, Kitayama T, Yao K, Shirakura S, Sasaki K, Kusaka H, Matsubara M |title=Blockade of T-type voltage-dependent Ca2+ channels by benidipine, a dihydropyridine calcium channel blocker, inhibits aldosterone production in human adrenocortical cell line NCI-H295R |journal=Eur. J. Pharmacol. |volume=584 |issue=2-3 |pages=424–34 |year=2008 |pmid=18331727 |doi=10.1016/j.ejphar.2008.02.001 |url=}}</ref><ref name="IkedaIsaka2012">{{cite journal|last1=Ikeda|first1=Keiichi|last2=Isaka|first2=Tsuyoshi|last3=Fujioka|first3=Kouki|last4=Manome|first4=Yoshinobu|last5=Tojo|first5=Katsuyoshi|title=Suppression of Aldosterone Synthesis and Secretion by Channel Antagonists|journal=International Journal of Endocrinology|volume=2012|year=2012|pages=1–6|issn=1687-8337|doi=10.1155/2012/519467}}</ref><ref name="pmid744152">{{cite journal |vauthors=McKenna TJ, Island DP, Nicholson WE, Liddle GW |title=The effects of potassium on early and late steps in aldosterone biosynthesis in cells of the zona glomerulosa |journal=Endocrinology |volume=103 |issue=4 |pages=1411–6 |year=1978 |pmid=744152 |doi=10.1210/endo-103-4-1411 |url=}}</ref> | ||
{| class="wikitable" | {| class="wikitable" |
Revision as of 19:44, 23 August 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
Disease name] may be caused by [cause1], [cause2], or [cause3].
OR
Common causes of [disease] include [cause1], [cause2], and [cause3].
OR
The most common cause of [disease name] is [cause 1]. Less common causes of [disease name] include [cause 2], [cause 3], and [cause 4].
OR
The cause of [disease name] has not been identified. To review risk factors for the development of [disease name], click here.
Causes
Life-threatening Causes
Life-threatening causes of hypoaldosteronism include:[1]
- Adrenal infarction
- Bilateral adrenal hemorrhage(caused by trauma, anticoagulant therapy, or coagulation disorders).
- Cancerous destruction of the adrenal gland, secondary to infiltrative or metastatic disease.
- Tubercular and fungal destruction of adrenal glands.
Common Causes
Common causes of hypoaldosteronism include:[2]
- Diabetic nephropathy
- Acute glomerulonephritis
- Autoimmune/Idiopathic
- Tuberculosis
- AIDS
- CMV
- Hemorrhage
- Infarction
- Sarcoidosis
- Infections
Less common causes
Less common causes of hypoaldosteronism include:
- Sarcoidosis
- Amyloidosis
- Fungal infections
- AIDS complications
- Hemochromatosis
- Polyglandular endocrine syndromes
- Adrenoleukodystrophy
- Adrenomyelodystrophy
Drugs causing hypoaldosteronism
Other less common causes of hypoaldosteronism include drugs such as:[3][4][5][6]
Decreased renin release | Inhibition of
aldosterone synthase |
Decreased production
of aldosterone |
Decreased effect of
aldosterone |
Drugs that impair
adrenal function |
Direct inhibition
of zona glomerulosa |
Dopaminergic agonists |
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|
References
- ↑ LaBan MM, Whitmore CE, Taylor RS (2003). "Bilateral adrenal hemorrhage after anticoagulation prophylaxis for bilateral knee arthroplasty". Am J Phys Med Rehabil. 82 (5): 418–20. doi:10.1097/01.PHM.0000064741.97586.E4. PMID 12704285.
- ↑ Sailer, Christian, Wasner, Susanne. Differential Diagnosis Pocket. Hermosa Beach, CA: Borm Bruckmeir Publishing LLC, 2002:14-15
- ↑ Missale C, Lombardi C, De Cotiis R, Memo M, Carruba MO, Spano PF (1989). "Dopaminergic receptor mechanisms modulating the renin-angiotensin system and aldosterone secretion: an overview". J. Cardiovasc. Pharmacol. 14 Suppl 8: S29–39. PMID 2483440.
- ↑ Akizuki O, Inayoshi A, Kitayama T, Yao K, Shirakura S, Sasaki K, Kusaka H, Matsubara M (2008). "Blockade of T-type voltage-dependent Ca2+ channels by benidipine, a dihydropyridine calcium channel blocker, inhibits aldosterone production in human adrenocortical cell line NCI-H295R". Eur. J. Pharmacol. 584 (2–3): 424–34. doi:10.1016/j.ejphar.2008.02.001. PMID 18331727.
- ↑ Ikeda, Keiichi; Isaka, Tsuyoshi; Fujioka, Kouki; Manome, Yoshinobu; Tojo, Katsuyoshi (2012). "Suppression of Aldosterone Synthesis and Secretion by Channel Antagonists". International Journal of Endocrinology. 2012: 1–6. doi:10.1155/2012/519467. ISSN 1687-8337.
- ↑ McKenna TJ, Island DP, Nicholson WE, Liddle GW (1978). "The effects of potassium on early and late steps in aldosterone biosynthesis in cells of the zona glomerulosa". Endocrinology. 103 (4): 1411–6. doi:10.1210/endo-103-4-1411. PMID 744152.