Pre-eclampsia causes: Difference between revisions
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{{CMG}}; {{AE}} {{Sara.Zand}} {{Ochuko}} | {{CMG}}; {{AE}} {{Sara.Zand}} {{Ochuko}} | ||
==Overview== | ==Overview== | ||
Common cause of [[preeclampsia]] include [[uteroplacental ischemia]] and [[genetic]] predisposition following The formation of [[atheromatous plaques]] and [[fibrinoid necrosis]] of the [[ spiral ]] [[vessel walls]], [[ | Common cause of [[preeclampsia]] include [[uteroplacental ischemia]] and [[genetic]] predisposition following The formation of [[atheromatous plaques]] and [[fibrinoid necrosis]] of the [[spiral]] [[vessel walls]], [[oxidative stress]] in [[trophoblast cells]], [[apoptosis]] in [[trophoblast cells]],[[systemic inflammatory response]], [[vasospasm]], [[platelet]] aggregation, [[thrombin]] formation, deposition of the [[fibrin]] in multiple [[organs]]. | ||
==Causes== | ==Causes== | ||
Common cause of [[preeclampsia]] include [[uteroplacental]] [[ischemia]] and [[genetic predisposition]] due to the following:<ref name="LyeBloise2013">{{cite journal|last1=Lye|first1=P.|last2=Bloise|first2=E.|last3=Dunk|first3=C.|last4=Javam|first4=M.|last5=Gibb|first5=W.|last6=Lye|first6=S.J.|last7=Matthews|first7=S.G.|title=Effect of oxygen on multidrug resistance in the first trimester human placenta|journal=Placenta|volume=34|issue=9|year=2013|pages=817–823|issn=01434004|doi=10.1016/j.placenta.2013.05.010}}</ref><ref name="MayrinkCosta2018">{{cite journal|last1=Mayrink|first1=J.|last2=Costa|first2=M. L.|last3=Cecatti|first3=J. G.|title=Preeclampsia in 2018: Revisiting Concepts, Physiopathology, and Prediction|journal=The Scientific World Journal|volume=2018|year=2018|pages=1–9|issn=2356-6140|doi=10.1155/2018/6268276}}</ref> | Common cause of [[preeclampsia]] include [[uteroplacental]] [[ischemia]] and [[genetic predisposition]] due to the following:<ref name="LyeBloise2013">{{cite journal|last1=Lye|first1=P.|last2=Bloise|first2=E.|last3=Dunk|first3=C.|last4=Javam|first4=M.|last5=Gibb|first5=W.|last6=Lye|first6=S.J.|last7=Matthews|first7=S.G.|title=Effect of oxygen on multidrug resistance in the first trimester human placenta|journal=Placenta|volume=34|issue=9|year=2013|pages=817–823|issn=01434004|doi=10.1016/j.placenta.2013.05.010}}</ref><ref name="MayrinkCosta2018">{{cite journal|last1=Mayrink|first1=J.|last2=Costa|first2=M. L.|last3=Cecatti|first3=J. G.|title=Preeclampsia in 2018: Revisiting Concepts, Physiopathology, and Prediction|journal=The Scientific World Journal|volume=2018|year=2018|pages=1–9|issn=2356-6140|doi=10.1155/2018/6268276}}</ref> | ||
* The formation of [[atheromatous plaques]] and [[fibrinoid necrosis]] of the [[ spiral ]] [[vessel walls]] | * The formation of [[atheromatous plaques]] and [[fibrinoid necrosis]] of the [[spiral]] [[vessel walls]] | ||
* [[Oxidative stress]] in [[trophoblast cells]] | * [[Oxidative stress]] in [[trophoblast cells]] | ||
* [[Apoptosis]] in [[trophoblast cells]] | * [[Apoptosis]] in [[trophoblast cells]] | ||
* [[Systemic inflammatory response]] | * [[Systemic inflammatory response]] | ||
* [[Vasospasm]] | * [[Vasospasm]] | ||
*[[Platelet]] | * [[Platelet]] aggregation | ||
* [[Thrombin]] formation | * [[Thrombin]] formation | ||
* Deposition of the [[fibrin]] in multiple [[organs]] | * Deposition of the [[fibrin]] in multiple [[organs]] | ||
| Line 25: | Line 25: | ||
[[Category:Emergency medicine]] | [[Category:Emergency medicine]] | ||
[[Category:Cardiology]] | [[Category:Cardiology]] | ||
[[Category:Needs English Review]] | |||
Revision as of 02:37, 28 January 2021
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Pre-eclampsia Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Pre-eclampsia causes On the Web |
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American Roentgen Ray Society Images of Pre-eclampsia causes |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Sara Zand, M.D.[2] Ogheneochuko Ajari, MB.BS, MS [3]
Overview
Common cause of preeclampsia include uteroplacental ischemia and genetic predisposition following The formation of atheromatous plaques and fibrinoid necrosis of the spiral vessel walls, oxidative stress in trophoblast cells, apoptosis in trophoblast cells,systemic inflammatory response, vasospasm, platelet aggregation, thrombin formation, deposition of the fibrin in multiple organs.
Causes
Common cause of preeclampsia include uteroplacental ischemia and genetic predisposition due to the following:[1][2]
- The formation of atheromatous plaques and fibrinoid necrosis of the spiral vessel walls
- Oxidative stress in trophoblast cells
- Apoptosis in trophoblast cells
- Systemic inflammatory response
- Vasospasm
- Platelet aggregation
- Thrombin formation
- Deposition of the fibrin in multiple organs
References
- ↑ Lye, P.; Bloise, E.; Dunk, C.; Javam, M.; Gibb, W.; Lye, S.J.; Matthews, S.G. (2013). "Effect of oxygen on multidrug resistance in the first trimester human placenta". Placenta. 34 (9): 817–823. doi:10.1016/j.placenta.2013.05.010. ISSN 0143-4004.
- ↑ Mayrink, J.; Costa, M. L.; Cecatti, J. G. (2018). "Preeclampsia in 2018: Revisiting Concepts, Physiopathology, and Prediction". The Scientific World Journal. 2018: 1–9. doi:10.1155/2018/6268276. ISSN 2356-6140.