Amyloidosis electrocardiogram: Difference between revisions
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==Overview== | ==Overview== | ||
Electrocardiogram is particularly useful for cardiac amyloidosis. Findings on electrocardiogram include | [[Electrocardiogram]] is particularly useful for cardiac amyloidosis. Findings on [[electrocardiogram]] include low voltage [[QRS complexes]], left and right ventricular [[Hypertrophy (medical)|hypertrophy]], [[Left atrium|left atrial]] abnormalities, pathological [[Q wave|Q waves]], and [[Atrioventricular block|AV block]]. | ||
==Electrocardiogram== | ==Electrocardiogram== | ||
Electrocardiogram is particularly useful for cardiac amyloidosis. Findings on electrocardiogram include: | [[Electrocardiogram]] is particularly useful for cardiac amyloidosis. Findings on an [[electrocardiogram]] include: | ||
*Low voltage QRS complexes<ref name="pmid22058156">{{cite journal| author=Guan J, Mishra S, Falk RH, Liao R| title=Current perspectives on cardiac amyloidosis. | journal=Am J Physiol Heart Circ Physiol | year= 2012 | volume= 302 | issue= 3 | pages= H544-52 | pmid=22058156 | doi=10.1152/ajpheart.00815.2011 | pmc=3353775 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=22058156 }} </ref>: Low voltages are present due to infiltrative heart disease and the reduced electrical conduction through the cardiac muscle. | *Low voltage QRS complexes<ref name="pmid22058156">{{cite journal| author=Guan J, Mishra S, Falk RH, Liao R| title=Current perspectives on cardiac amyloidosis. | journal=Am J Physiol Heart Circ Physiol | year= 2012 | volume= 302 | issue= 3 | pages= H544-52 | pmid=22058156 | doi=10.1152/ajpheart.00815.2011 | pmc=3353775 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=22058156 }} </ref>: Low voltages are present due to infiltrative heart disease and the reduced electrical conduction through the cardiac muscle. | ||
*Left ventricular hypertrophy<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445 }} </ref> | *[[Left ventricle|Left ventricular]] [[Hypertrophy (medical)|hypertrophy]]<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445 }} </ref> | ||
*Right ventricular hypertrophy<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445 }} </ref> | *[[Right ventricle|Right ventricular]] hypertrophy<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445 }} </ref> | ||
*Left atrial abnormalities | *[[Left atrium|Left atrial]] abnormalities | ||
*Pathological Q waves<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445 }} </ref> | *Pathological [[Q waves]]<ref name="pmid25743445">{{cite journal| author=Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J| title=Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis. | journal=J Cardiovasc Transl Res | year= 2015 | volume= 8 | issue= 2 | pages= 117-27 | pmid=25743445 | doi=10.1007/s12265-015-9613-9 | pmc=4382536 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25743445 }} </ref> | ||
*Atrioventricular block: AV block occur due to deposition of amyloid fibrils in the conduction tissue. | *[[Atrioventricular block]]: [[Atrioventricular block|AV block]] occur due to deposition of amyloid fibrils in the conduction [[Tissue (biology)|tissue]]. | ||
**First-degree AV block | **[[First degree AV block|First-degree AV block]] | ||
**Second-degree AV block | **[[Second degree AV block|Second-degree AV block]] | ||
**Third-degree AV block | **[[Third degree AV block|Third-degree AV block]] | ||
== References == | == References == |
Latest revision as of 01:15, 26 October 2019
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Shyam Patel [2]
Overview
Electrocardiogram is particularly useful for cardiac amyloidosis. Findings on electrocardiogram include low voltage QRS complexes, left and right ventricular hypertrophy, left atrial abnormalities, pathological Q waves, and AV block.
Electrocardiogram
Electrocardiogram is particularly useful for cardiac amyloidosis. Findings on an electrocardiogram include:
- Low voltage QRS complexes[1]: Low voltages are present due to infiltrative heart disease and the reduced electrical conduction through the cardiac muscle.
- Left ventricular hypertrophy[2]
- Right ventricular hypertrophy[2]
- Left atrial abnormalities
- Pathological Q waves[2]
- Atrioventricular block: AV block occur due to deposition of amyloid fibrils in the conduction tissue.
References
- ↑ Guan J, Mishra S, Falk RH, Liao R (2012). "Current perspectives on cardiac amyloidosis". Am J Physiol Heart Circ Physiol. 302 (3): H544–52. doi:10.1152/ajpheart.00815.2011. PMC 3353775. PMID 22058156.
- ↑ 2.0 2.1 2.2 Damy T, Judge DP, Kristen AV, Berthet K, Li H, Aarts J (2015). "Cardiac findings and events observed in an open-label clinical trial of tafamidis in patients with non-Val30Met and non-Val122Ile hereditary transthyretin amyloidosis". J Cardiovasc Transl Res. 8 (2): 117–27. doi:10.1007/s12265-015-9613-9. PMC 4382536. PMID 25743445.