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==Overview==
==Overview==

Revision as of 16:56, 28 December 2017

Duodenal Atresia Microchapters

Home

Patient Information

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Duodenal Atresia from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Study of Choice

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

X-ray

Echocardiography and Ultrasound

CT scan

MRI

Other Imaging Findings

Other Diagnostic Studies

Treatment

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Primary Prevention

Secondary Prevention

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Hamid Qazi, MD, BSc [2]

https://https://www.youtube.com/watch?v=7481ok2xsbI%7C350}}

Overview

Historical Perspective

In 1900, J. Tandler reported duodenal stenosis due to failure of recanalization of duodenum during fetal development in pregnancy. In 1961, Thomas Santulli and William Blanc described the figure 8 formation of small bowel, which is described as apple-peel intestinal atresia. In 1936, William Ladd developed a surgical procedure to correct the duodenal malrotation.

Classification

Intestinal atresia, including duodenal atresia, may be classified into four subtypes: Type I, Type II, Type III, and Type IV. Type I is the most common subtype which involves the complete mucosal membrane, with muscularis and serosa remaining intact.

Pathophysiology

It is thought that duodenal atresia is the result of failure of neural cell migration during the 8th to 10th week of duodenal re-canalization. It is associated with down syndrome, vertebral defects, anal anomalies, esophageal atresia, annular pancreas, malrotation, renal abnormalities, cardiac causes, and mandibulofacial anomalies.

Causes

The cause of duodenal atresia has not been identified.

Differentiating ((Page name)) from Other Diseases

Epidemiology and Demographics

The prevalance of duodenal atresia is 0.1 to 0.4 per 100000. It commonly affects neonates and has a male to female ratio of 2 to 1.

Risk Factors

The most potent risk factor in the development of duodenal atresia is down syndrome. Other risk factors include annular pancreas, and VACTERL syndrome.

Screening

There is insufficient evidence to recommend routine screening for duodenal atresia.

Natural History, Complications, and Prognosis

The symptoms of duodenal atresia usually develop in the first 48 hours of life, and start with symptoms such as bilious vomiting in 80% of the cases. Prognosis is generally very good, and the survival rate of patients with duodenal atresia is approximately 90%.

Diagnosis

Diagnostic Criteria

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

X-ray

Ultrasound

CT scan

MRI

Other Imaging Findings

Other Diagnostic Studies

Treatment

Medical Therapy

Surgery

Primary Prevention

Secondary Prevention

References


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