Cardiac tamponade resident survival guide: Difference between revisions

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==Causes==
==Causes==
===Causes in Alphabetical Order===
*[[Amyloidosis]]
*[[Anticoagulant therapy]] <ref>Longmore, M., Wilkinson, I.B., Rajagopalan, S. (2004) (6th Ed.). Oxford Handbook of Clinical Medicine. Oxford: Oxford University Press ISBN 9780198568377 </ref>.
*[[Aortic dissection]] <ref>Isselbacher, E.M., Cigarroa, J.E., Eagle, K.A. (1994). Cardiac tamponade complicating proximal aortic dissection. Is pericardiocentesis harmful? Circulation. Vol 90, 2375-2378</ref>
*Bacterial [[pericarditis]]
*[[Bronchogenic cyst]]
*[[Cancer]]
*[[Chest trauma]] (both blunt and penetrating) <ref>Gwinnutt, C., Driscoll, P. (Eds) (2003) (2nd Ed.) Trauma Resuscitation: The Team Approach. Oxford: BIOS Scientific Publishers Ltd. ISBN 978-1859960097 </ref>
*[[Constrictive pericarditis]]
*[[Dilated cardiomyopathy]]
*[[Dissecting aortic aneurysm]]
*[[Dressler syndrome]]
*During cardiac surgery <ref>Mattson Porth, C. (Ed.) (2005) (7th Ed.) Pathophysiology: Concepts of Altered Health States. Philadelphia : Lippincott Williams & Wilkins ISBN 978-0781749886 </ref>
*[[Hypothyroidism]]
*[[Malignancy]]
*[[Mycobacterium tuberculosis]]
*[[Myocardial rupture]]. Myocardial rupture typically happens in the subacute setting after a [[myocardial infarction]] (heart attack), in which the infarcted muscle of the heart thins out and tears.  Myocardial rupture is more likely to happen in females, the elderly, patients with hypertension, and individuals without any previous[[heart|cardiac]] history who suffer from their first heart attack and are not revascularized with [[thrombolytic]] therapy, [[percutaneous coronary intervention]], or with[[coronary artery bypass graft surgery]].<ref name="rupturelikeliness">*{{cite journal
| first=A
| last=Meniconi
| authorlink=
| coauthors=C H ATTENHOFER JOST, R JENNI
| year=2000
| month=November
| title=How to survive myocardial rupture after myocardial infarction
| journal=Heart
| volume=84
| issue=5
| pages =
| id= PMID 11040020
| url=http://heart.bmj.com/cgi/content/full/84/5/552
}}</ref> These patients often have single vessel disease without the development of [[collateral]]s.
*Penetrating cardiac injury
*[[Pericarditis]]
*Physical trauma
*[[Postpericardiotomy syndrome]]
*Pyogenic [[pericarditis]]
*Rheumatoid pericarditis
*[[Scrub typhus]]
*[[Tuberculous pericarditis]]
*[[Uremia]]
*[[Uremic pericarditis]]
*[[Ventricular aneurysm]]
*Viral [[pericarditis]]
===Life Threatening Causes===
===Life Threatening Causes===
Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.
Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.

Revision as of 20:28, 17 January 2014

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ayokunle Olubaniyi, M.B,B.S [2]

Definitions

Causes

Causes in Alphabetical Order

Life Threatening Causes

Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.

Common Causes

Management

Shown below is an algorithm showing acute pericarditis management.[6]

 
 
 
 
 
 
Characterize the symptoms:

❑ Prodrome:
Fever
Malaise
Myalgia
❑ Retrosternal or left precordial chest pain:
♦Radiates to trapezius ridge
♦Can be pleuritic
♦Can simulate ischemia
♦Varies with posture

 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Auscultation:

Pericardial rub
♦Monophasic
♦Biphasic
♦Triphasic

 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Perform ECG:

❑ Convexly elevated J-ST segment
❑ Determine ECK staging:
♦ Stage I: anterior and inferior concave ST segment elevation. PR segment 7,19 deviations opposite to P polarity
♦ Early stage II: ST junctions return to the baseline, PR deviated
♦ Late stage II: T waves progressively flatten and invert
♦ Stage III: generalised T wave inversions
♦ Stage IV: ECG returns to prepericarditis state

 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Echocardiography:
❑ Effusion types B- D according to Horowitz classification:
♦ Type A: No effusion
♦ Type B: Separation of epicardium and pericardium (3–16 ml)
♦ Type C 1: Systolic and diastolic separation of epicardium and pericardium (small effusion >16 ml)
♦ Type C 2: Systolic and diastolic separation of epicardium and pericardium with attenuated pericardial motion
♦ Type D: Pronounced separation of epicardium and pericardium with large echo- free space
♦ Type E: Pericardial thickening (>4 mm)
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Evidence of pericardial effusion
 
 
 
 
 
Order lab tests:
❑ Inflammation markers:
ESR
CRP
LDH
❑ Markers of myocardial lesion:
Troponin I
CK MB
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
 
Large recurrent effusion
 
Signs of cardiac tamponade
 
 
 
Perform chest X-Ray:
❑ “water bottle” heart shadow
 
 

Dos

Don'ts

References

  1. Longmore, M., Wilkinson, I.B., Rajagopalan, S. (2004) (6th Ed.). Oxford Handbook of Clinical Medicine. Oxford: Oxford University Press ISBN 9780198568377
  2. Isselbacher, E.M., Cigarroa, J.E., Eagle, K.A. (1994). Cardiac tamponade complicating proximal aortic dissection. Is pericardiocentesis harmful? Circulation. Vol 90, 2375-2378
  3. Gwinnutt, C., Driscoll, P. (Eds) (2003) (2nd Ed.) Trauma Resuscitation: The Team Approach. Oxford: BIOS Scientific Publishers Ltd. ISBN 978-1859960097
  4. Mattson Porth, C. (Ed.) (2005) (7th Ed.) Pathophysiology: Concepts of Altered Health States. Philadelphia : Lippincott Williams & Wilkins ISBN 978-0781749886
  5. *Meniconi, A (2000). "How to survive myocardial rupture after myocardial infarction". Heart. 84 (5). PMID 11040020. Unknown parameter |coauthors= ignored (help); Unknown parameter |month= ignored (help)
  6. Maisch B, Seferović PM, Ristić AD, Erbel R, Rienmüller R, Adler Y; et al. (2004). "Guidelines on the diagnosis and management of pericardial diseases executive summary; The Task force on the diagnosis and management of pericardial diseases of the European society of cardiology". Eur Heart J. 25 (7): 587–610. doi:10.1016/j.ehj.2004.02.002. PMID 15120056.

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