Blalock-Taussig shunt

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Overview

The Blalock-Taussig shunt is a surgical procedure that are done as palliative procedures for cyanotic heart defects . This procedure is used to deviate blood flow to lungs from systemic circulation to relieve cyanosis while a definitive corrective surgery can be performed at later time. Connection between subclavian artery and pulmonary artery is usually made . There are usually two types of shunts that are used i.e classic or original Blalock-Taussig shunt and modified Blalock Taussig shunt. The classic or original BT shunt has been modified into modified Blalock-Taussig Shunt (mBTS). In this modified procedure a graft from innominate artery or subclavian artery is placed to the corresponding pulmonary artery. [1]This type of BT shunt has superior prognostic value over classical shunt leading to greater rate of shunt patency at 3-5 years as compared to classic or original BT Shunt.[2]

Historical Perspective

Classic or Original Shunt

The classic or original BT shunt procedure was named for Alfred Blalock, surgeon, Baltimore, (1899–1964) and Helen B. Taussig, cardiologist, Baltimore/Boston, (1898–1986) who, along with Blalock's African American laboratory technician Vivien Thomas (1910–1985), developed and described the procedure. Taussig noticed that the children with cyanosis along with congenital heart disease accompanied by patent ductus arteriosus have longer life time than those without PDA. Dr. Taussig approached Blalock and Thomas in their Hopkins laboratory in 1943 to work upon in this shunt as it was hypothesized that a shunt mimicking PDA can relieve the cyanosis and improve oxygenation in congential cyanotic diseases. Thomas, Vivien (1985). Partners of the heart : Vivien Thomas and his work with Alfred Blalock : an autobiography. Philadelphia: University of Pennsylvania Press. ISBN 0812216342.

Original Blalock-Taussig shunt diagram. Source: Dr Laurent Bilodeau.


Modified Blalock-Taussig Shunt

A modified method of inserting a shunt was developed in 1962 by Klinner using teflon as prosthetic graft material between the subclavian artery and the pulmonary artery is used to prevent scarring of the subclavian artery. [3]

Modified Blalock-Taussig shunt diagram. Source: Dr Laurent Bilodeau.

Indications








Procedure

Classic/Orginal Blalock-Taussig Shunt [1]

Classic/Original Blalock-Taussig Shunt
Advantages Disadvantages
The relative diameter of subclavian artery prevents excessive blood flow to lungs Thrombosis of shunt due to less diameter
Easily Reversible Risk of Dissection
Increased rate of anastomosis growth Subclavian artery is lost during the procedure

Modified Blalock Taussig Shunt [1]

It is the most commonly used procedure now a days. First described and performed by Klinner et. al in 1962, this procedure hold superb prognosis over classic one. An Interposition PTFE or Gore-Tex graft is placed between the subclavian artery and the pulmonary artery. Hence no scarring of subclavian artery results.

Modified Blalock Taussig Shunt
Advantages Disadvantages
It can be done on the same side of the arch Thrombosis
More patency than Classic BT shunt i.e >90 % at 2 years Pseudoaneurysm
Pulmonary artery is less disorted Chylothorax, Chylopericardium, chylous ascites

Complications

Case courtesy of Dr Charlie Chia-Tsong Hsu,. From the case https://radiopaedia.org/cases/18390, rID: 18390</a>

The immediate post-operative complications include [2]




Prognosis

Case courtesy of Dr Vincent Tatco,From the case https://radiopaedia.org/cases/41040,rID: 41040

Modified Blalock-Taussig Shunt has superior prognostic value over classic Blalock-Taussig Shunts. Following prognostic factors are compared between the two procedures in multiple studies. [4] [5] [6] [7] [8] [9]

  • Rise in saturation of oxygen is greater in modified than in classic shunt
  • Shunt patency is 88.8 % in modified for 3-5 years versus 90 % in first year, 62 % in two years and 78.0 % in 3 years for classic shunt.
  • The risk of early shunt faliure is 20.8 % in modified and 51.7% in classic shunt.
  • Post-shunt increase in pulmonary arterial index (mm2/m2) is 158 +/- 21 versus 117 +/- 52 in classic Blalock-Taussig shunt.










References

  1. 1.0 1.1 1.2 Kiran U, Aggarwal S, Choudhary A, Uma B, Kapoor PM (2017). "The blalock and taussig shunt revisited". Ann Card Anaesth. 20 (3): 323–330. doi:10.4103/aca.ACA_80_17. PMC 5535574. PMID 28701598.
  2. 2.0 2.1 Yuan SM, Shinfeld A, Raanani E (2009). "The Blalock-Taussig shunt". J Card Surg. 24 (2): 101–8. doi:10.1111/j.1540-8191.2008.00758.x. PMID 19040408.
  3. KLINNER W, PASINI M, SCHAUDIG A (1962). "[Anastomosis between systemic and pulmonary arteries with the aid of plastic prostheses in cyanotic heart diseases]". Thoraxchirurgie. 10: 68–75. doi:10.1055/s-0028-1096482. PMID 14457041.
  4. Al Jubair KA, Al Fagih MR, Al Jarallah AS, Al Yousef S, Ali Khan MA, Ashmeg A; et al. (1998). "Results of 546 Blalock-Taussig shunts performed in 478 patients". Cardiol Young. 8 (4): 486–90. doi:10.1017/s1047951100007150. PMID 9855103.
  5. Kim HK, Kim WH, Kim SC, Lim C, Lee CH, Kim SJ (2006). "Surgical strategy for pulmonary coarctation in the univentricular heart". Eur J Cardiothorac Surg. 29 (1): 100–4. doi:10.1016/j.ejcts.2005.10.032. PMID 16337132.
  6. Karpawich PP, Bush CP, Antillon JR, Amato JJ, Marbey ML, Agarwal KC (1985). "Modified Blalock-Taussig shunt in infants and young children. Clinical and catheterization assessment". J Thorac Cardiovasc Surg. 89 (2): 275–9. PMID 3968910.
  7. Kulkarni H, Rajani R, Dalvi B, Gupta KG, Vora A, Kelkar P (1995). "Effect of Blalock Taussig shunt on clinical parameters, left ventricular function and pulmonary arteries". J Postgrad Med. 41 (2): 34–6. PMID 10707705.
  8. Bove EL, Kohman L, Sereika S, Byrum CJ, Kavey RE, Blackman MS; et al. (1987). "The modified Blalock-Taussig shunt: analysis of adequacy and duration of palliation". Circulation. 76 (3 Pt 2): III19–23. PMID 2441893.
  9. Ullom RL, Sade RM, Crawford FA, Ross BA, Spinale F (1987). "The Blalock-Taussig shunt in infants: standard versus modified". Ann Thorac Surg. 44 (5): 539–43. doi:10.1016/s0003-4975(10)62119-4. PMID 3675059.