Wild-type (senile) amyloidosis electrocardiogram: Difference between revisions
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==Overview== | ==Overview== | ||
he [[The electrocardiogram|EKG]] findings in wild-type (senile) amyloidosis have low [[Sensitivity (tests)|sensitivity]] and [[Specificity (tests)|specificity]] for the condition. | |||
==Electrocardiogram== | ==Electrocardiogram== | ||
*The EKG findings in wild-type (senile) amyloidosis have low sensitivity and specificity for the condition.<ref>{{Cite journal | *The [[The electrocardiogram|EKG]] findings in wild-type (senile) amyloidosis have low [[Sensitivity (tests)|sensitivity]] and [[Specificity (tests)|specificity]] for the condition.<ref>{{Cite journal | ||
| author = [[Ilia G. Halatchev]], [[Jingsheng Zheng]] & [[Jiafu Ou]] | | author = [[Ilia G. Halatchev]], [[Jingsheng Zheng]] & [[Jiafu Ou]] | ||
| title = Wild-type transthyretin cardiac amyloidosis (ATTRwt-CA), previously known as senile cardiac amyloidosis: clinical presentation, diagnosis, management and emerging therapies | | title = Wild-type transthyretin cardiac amyloidosis (ATTRwt-CA), previously known as senile cardiac amyloidosis: clinical presentation, diagnosis, management and emerging therapies | ||
| Line 18: | Line 19: | ||
| pmid = 29707360 | | pmid = 29707360 | ||
}}</ref> | }}</ref> | ||
*This is because the EKG findings are also common in aging patients | *This is because the [[The electrocardiogram|EKG]] findings are also common in [[Ageing|aging]] patients suffering from comorbidities. | ||
*EKG findings encountered during the evaluation of a patient with wild-type (senile) amyloidosis are given below: | *[[The electrocardiogram|EKG]] findings encountered during the evaluation of a [[patient]] with wild-type (senile) amyloidosis are given below: | ||
:*Pseudoinfarct pattern<ref>{{Cite journal | |||
:*[[Pseudoinfarct pattern]]<ref>{{Cite journal | |||
| author = [[Thibaud Damy]], [[Mathew S. Maurer]], [[Claudio Rapezzi]], [[Violaine Plante-Bordeneuve]], [[Onur N. Karayal]], [[Rajiv Mundayat]], [[Ole B. Suhr]] & [[Arnt V. Kristen]] | | author = [[Thibaud Damy]], [[Mathew S. Maurer]], [[Claudio Rapezzi]], [[Violaine Plante-Bordeneuve]], [[Onur N. Karayal]], [[Rajiv Mundayat]], [[Ole B. Suhr]] & [[Arnt V. Kristen]] | ||
| title = Clinical, ECG and echocardiographic clues to the diagnosis of TTR-related cardiomyopathy | | title = Clinical, ECG and echocardiographic clues to the diagnosis of TTR-related cardiomyopathy | ||
| Line 43: | Line 45: | ||
| pmid = 28329248 | | pmid = 28329248 | ||
}}</ref> | }}</ref> | ||
:*Poor R wave progression<ref>{{Cite journal | :*[[Poor R Wave Progression|Poor R wave progression]]<ref>{{Cite journal | ||
| author = [[Esther Gonzalez-Lopez]], [[Christian Gagliardi]], [[Fernando Dominguez]], [[Cristina Candida Quarta]], [[F. Javier de Haro-Del Moral]], [[Agnese Milandri]], [[Clara Salas]], [[Mario Cinelli]], [[Marta Cobo-Marcos]], [[Massimiliano Lorenzini]], [[Enrique Lara-Pezzi]], [[Serena Foffi]], [[Luis Alonso-Pulpon]], [[Claudio Rapezzi]] & [[Pablo Garcia-Pavia]] | | author = [[Esther Gonzalez-Lopez]], [[Christian Gagliardi]], [[Fernando Dominguez]], [[Cristina Candida Quarta]], [[F. Javier de Haro-Del Moral]], [[Agnese Milandri]], [[Clara Salas]], [[Mario Cinelli]], [[Marta Cobo-Marcos]], [[Massimiliano Lorenzini]], [[Enrique Lara-Pezzi]], [[Serena Foffi]], [[Luis Alonso-Pulpon]], [[Claudio Rapezzi]] & [[Pablo Garcia-Pavia]] | ||
| title = Clinical characteristics of wild-type transthyretin cardiac amyloidosis: disproving myths | | title = Clinical characteristics of wild-type transthyretin cardiac amyloidosis: disproving myths | ||
| Line 55: | Line 57: | ||
| pmid = 28329248 | | pmid = 28329248 | ||
}}</ref> | }}</ref> | ||
:*Atrial fibrillation<ref>{{Cite journal | :*[[Atrial fibrillation]]<ref>{{Cite journal | ||
| author = [[Christoph Rocken]], [[Brigitte Peters]], [[Gina Juenemann]], [[Wolfgang Saeger]], [[Helmut U. Klein]], [[Christof Huth]], [[Albert Roessner]] & [[Andreas Goette]] | | author = [[Christoph Rocken]], [[Brigitte Peters]], [[Gina Juenemann]], [[Wolfgang Saeger]], [[Helmut U. Klein]], [[Christof Huth]], [[Albert Roessner]] & [[Andreas Goette]] | ||
| title = Atrial amyloidosis: an arrhythmogenic substrate for persistent atrial fibrillation | | title = Atrial amyloidosis: an arrhythmogenic substrate for persistent atrial fibrillation | ||
| Line 77: | Line 79: | ||
| doi = 10.1016/j.amjcard.2014.07.026 | | doi = 10.1016/j.amjcard.2014.07.026 | ||
| pmid = 25212550 | | pmid = 25212550 | ||
}}</ref> | }}</ref> | ||
:*First degree AV block<ref>{{Cite journal | :*[[First degree AV block]]<ref>{{Cite journal | ||
| author = [[Blaithnead Murtagh]], [[Stephen C. Hammill]], [[Morie A. Gertz]], [[Robert A. Kyle]], [[A. Jamil Tajik]] & [[Martha Grogan]] | | author = [[Blaithnead Murtagh]], [[Stephen C. Hammill]], [[Morie A. Gertz]], [[Robert A. Kyle]], [[A. Jamil Tajik]] & [[Martha Grogan]] | ||
| title = Electrocardiographic findings in primary systemic amyloidosis and biopsy-proven cardiac involvement | | title = Electrocardiographic findings in primary systemic amyloidosis and biopsy-proven cardiac involvement | ||
Revision as of 22:24, 17 December 2019
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Sabawoon Mirwais, M.B.B.S, M.D.[2]
Overview
he EKG findings in wild-type (senile) amyloidosis have low sensitivity and specificity for the condition.
Electrocardiogram
- The EKG findings in wild-type (senile) amyloidosis have low sensitivity and specificity for the condition.[1]
- This is because the EKG findings are also common in aging patients suffering from comorbidities.
- EKG findings encountered during the evaluation of a patient with wild-type (senile) amyloidosis are given below:
- Pseudoinfarct pattern[2][3]
- Poor R wave progression[4]
- Atrial fibrillation[5][6]
- First degree AV block[7]
- Nonspecific ST-T wave abnormalities[8]
References
- ↑ Ilia G. Halatchev, Jingsheng Zheng & Jiafu Ou (2018). "Wild-type transthyretin cardiac amyloidosis (ATTRwt-CA), previously known as senile cardiac amyloidosis: clinical presentation, diagnosis, management and emerging therapies". Journal of thoracic disease. 10 (3): 2034–2045. doi:10.21037/jtd.2018.03.134. PMID 29707360. Unknown parameter
|month=ignored (help) - ↑ Thibaud Damy, Mathew S. Maurer, Claudio Rapezzi, Violaine Plante-Bordeneuve, Onur N. Karayal, Rajiv Mundayat, Ole B. Suhr & Arnt V. Kristen (2016). "Clinical, ECG and echocardiographic clues to the diagnosis of TTR-related cardiomyopathy". Open heart. 3 (1): e000289. doi:10.1136/openhrt-2015-000289. PMID 26870387.
- ↑ Esther Gonzalez-Lopez, Christian Gagliardi, Fernando Dominguez, Cristina Candida Quarta, F. Javier de Haro-Del Moral, Agnese Milandri, Clara Salas, Mario Cinelli, Marta Cobo-Marcos, Massimiliano Lorenzini, Enrique Lara-Pezzi, Serena Foffi, Luis Alonso-Pulpon, Claudio Rapezzi & Pablo Garcia-Pavia (2017). "Clinical characteristics of wild-type transthyretin cardiac amyloidosis: disproving myths". European heart journal. 38 (24): 1895–1904. doi:10.1093/eurheartj/ehx043. PMID 28329248. Unknown parameter
|month=ignored (help) - ↑ Esther Gonzalez-Lopez, Christian Gagliardi, Fernando Dominguez, Cristina Candida Quarta, F. Javier de Haro-Del Moral, Agnese Milandri, Clara Salas, Mario Cinelli, Marta Cobo-Marcos, Massimiliano Lorenzini, Enrique Lara-Pezzi, Serena Foffi, Luis Alonso-Pulpon, Claudio Rapezzi & Pablo Garcia-Pavia (2017). "Clinical characteristics of wild-type transthyretin cardiac amyloidosis: disproving myths". European heart journal. 38 (24): 1895–1904. doi:10.1093/eurheartj/ehx043. PMID 28329248. Unknown parameter
|month=ignored (help) - ↑ Christoph Rocken, Brigitte Peters, Gina Juenemann, Wolfgang Saeger, Helmut U. Klein, Christof Huth, Albert Roessner & Andreas Goette (2002). "Atrial amyloidosis: an arrhythmogenic substrate for persistent atrial fibrillation". Circulation. 106 (16): 2091–2097. doi:10.1161/01.cir.0000034511.06350.df. PMID 12379579. Unknown parameter
|month=ignored (help) - ↑ Nicole B. Cyrille, Jeff Goldsmith, Julissa Alvarez & Mathew S. Maurer (2014). "Prevalence and prognostic significance of low QRS voltage among the three main types of cardiac amyloidosis". The American journal of cardiology. 114 (7): 1089–1093. doi:10.1016/j.amjcard.2014.07.026. PMID 25212550. Unknown parameter
|month=ignored (help) - ↑ Blaithnead Murtagh, Stephen C. Hammill, Morie A. Gertz, Robert A. Kyle, A. Jamil Tajik & Martha Grogan (2005). "Electrocardiographic findings in primary systemic amyloidosis and biopsy-proven cardiac involvement". The American journal of cardiology. 95 (4): 535–537. doi:10.1016/j.amjcard.2004.10.028. PMID 15695149. Unknown parameter
|month=ignored (help) - ↑ Blaithnead Murtagh, Stephen C. Hammill, Morie A. Gertz, Robert A. Kyle, A. Jamil Tajik & Martha Grogan (2005). "Electrocardiographic findings in primary systemic amyloidosis and biopsy-proven cardiac involvement". The American journal of cardiology. 95 (4): 535–537. doi:10.1016/j.amjcard.2004.10.028. PMID 15695149. Unknown parameter
|month=ignored (help)