Supraventricular tachycardia differentiating SVT from VT: Difference between revisions

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==Differentiating SVT from VT==
==Differentiating SVT from VT==
*''Rate'': More than 100 bpm and usually 150-200 bpm
For a detailed discussion of how to distinguish [[ventricular tachycardia]] ([[VT]]) from [[supraventricular tachycardia]] ([[SVT]]), please visit the [[wide complex tachycardia differential diagnosis]] page.
*''Rhythm'': Generally regular
 
*''[[P waves]]'': Normal morphology, upright, but dissociated from the QRS complex (i.e. "march through" the [[QRS complex]])
In brief, the diagnosis of [[VT]] is more likely if:
*''[[PR interval]]'': Variable PR interval
*There is a history of [[myocardial infarction]], [[congestive heart failure]] or [[structural heart disease]]
*''[[QRS complex]]'': Wide and greater than 0.12 seconds
*[[VT]] is more common in the elderly
*''Response to Maneuvers'': Does not terminate in response to [[adenosine]] or [[vagal maneuvers]]
*The [[electrical axis]] is -90 to -180 degrees (a “northwest” or “superior” axis)
*''Epidemiology and Demographics'': Ventricular tachycardia is a major cause of [[sudden cardiac death]]. Common in elderly men compared to women.
*The [[QRS]] is > 140 msec
*''Risk Factors'': Occurs in the context of [[myocardial ischemia]], [[myocardial infarction]], [[congestive heart failure]], drug toxicity, and inhereted [[channelopathies]]
*There is [[AV dissociation]]. [[P waves]] are normal in morphology, upright, but dissociated from the QRS complex (i.e. "march through" the [[QRS complex]])
*There are positive or negative [[QRS]] complexes in all the precordial leads
*The morphology of the [[QRS]] complexes resembles that of a previous [[premature ventricular contraction]] ([[PVC]]).
*Rate: More than 100 bpm and usually 150-200 bpm
*Rhythm: The rhythm is regular
*[[PR interval]]: Variable PR interval
*Response to Maneuvers: VT does not terminate in response to [[adenosine]] or [[vagal maneuvers]]


==References==
==References==

Latest revision as of 15:31, 20 August 2013

Supraventricular tachycardia Microchapters

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Overview

Historical Perspective

Classification

Pathophysiology

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Differentiating Among the Different Types of Supraventricular Tachycardia

Differentiating Supraventricular Tachycardia from Ventricular Tachycardia

Epidemiology and Demographics

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Acute Treatment of SVT of Unknown Mechanism
Ongoing Management of SVT of Unknown Mechanism
Ongoing Management of IST
Acute Treatment of Suspected Focal Atrial Tachycardia
Acute Treatment of Multifocal Atria Tachycardia
Ongoing Management of Multifocal Atrial Tachycardia
Acute Treatment of AVNRT
Ongoing Management of AVNRT
Acute Treatment of Orthodromic AVRT
Ongoing Management of Orthodromic AVRT
Asymptomatic Patients With Pre-Excitation
Management of Symptomatic Patients With Manifest Accessory Pathways
Acute Treatment of Atrial Flutter
Ongoing Management of Atrial Flutter
Acute Treatment of Junctional Tachycardia
Ongoing Management of Junctional Tachycardia
Acute Treatment of SVT in ACHD Patients
Ongoing Management of SVT in ACHD Patients
Acute Treatment of SVT in Pregnant Patients
Acute Treatment and Ongoing Management of SVT in Older Population

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

See also: Wide complex tachycardia

Overview

Most supraventricular tachycardias have a narrow QRS complex on the EKG. It is not infrequent, however, for aberrant conduction to be be present, sometimes as a result of the more rapid rate of conduction. This widening of the QRS complex yields supraventricular tachycardia with aberrant conduction (SVTAC) which produces a wide-complex tachycardia that may mimic ventricular tachycardia (VT). In the clinical setting, it is important to determine whether a wide-complex tachycardia is an SVT or a ventricular tachycardia, since they are treated differently. Ventricular tachycardia has to be treated appropriately, since it can quickly degenerate to ventricular fibrillation and death. A number of different algorithms have been devised to determine whether a wide complex tachycardia is supraventricular or ventricular in origin.[1]

In general, a history of structural heart disease, ischemic heart disease or congestive heart failure increases the likelihood that the tachycardia is ventricular in origin.

Differentiating SVT from VT

For a detailed discussion of how to distinguish ventricular tachycardia (VT) from supraventricular tachycardia (SVT), please visit the wide complex tachycardia differential diagnosis page.

In brief, the diagnosis of VT is more likely if:

References

  1. Lau EW, Ng GA (2002). "Comparison of the performance of three diagnostic algorithms for regular broad complex tachycardia in practical application". Pacing and clinical electrophysiology : PACE. 25 (5): 822–7. PMID 12049375.


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