Ogilvie syndrome laboratory findings: Difference between revisions
Jump to navigation
Jump to search
No edit summary |
|||
| Line 4: | Line 4: | ||
==Overview== | ==Overview== | ||
There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may | There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include [[leukocytosis]] due to the underlying disease not due to the pseudo-obstruction itself. Many patients with Ogilvie syndrome may have [[Metabolic|metabolic imbalance]] which include [[hypokalemia]]<nowiki/>and [[hypocalcemia]]. Other laboratory tests that can be performed to exclude other causes include [[complete blood count]], [[Lactate levels raised|lactate levels]], and [[thyroid hormone]]<nowiki/>levels. | ||
==Laboratory Findings== | ==Laboratory Findings== | ||
*There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include leukocytosis due to the underlying disease not due to the pseudo-obstruction itself.<ref name="pmid3753674">{{cite journal| author=Vanek VW, Al-Salti M| title=Acute pseudo-obstruction of the colon (Ogilvie's syndrome). An analysis of 400 cases. | journal=Dis Colon Rectum | year= 1986 | volume= 29 | issue= 3 | pages= 203-10 | pmid=3753674 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=3753674 }}</ref> | *There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include [[leukocytosis]] due to the underlying disease not due to the pseudo-obstruction itself.<ref name="pmid3753674">{{cite journal| author=Vanek VW, Al-Salti M| title=Acute pseudo-obstruction of the colon (Ogilvie's syndrome). An analysis of 400 cases. | journal=Dis Colon Rectum | year= 1986 | volume= 29 | issue= 3 | pages= 203-10 | pmid=3753674 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=3753674 }}</ref> | ||
*In most of the patients metabolic imbalance may occur and it includes:<ref name="pmid1473414">{{cite journal| author=Jetmore AB, Timmcke AE, Gathright JB, Hicks TC, Ray JE, Baker JW| title=Ogilvie's syndrome: colonoscopic decompression and analysis of predisposing factors. | journal=Dis Colon Rectum | year= 1992 | volume= 35 | issue= 12 | pages= 1135-42 | pmid=1473414 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=1473414 }}</ref><ref name="pmid19892809">{{cite journal| author=Sandle GI, Hunter M| title=Apical potassium (BK) channels and enhanced potassium secretion in human colon. | journal=QJM | year= 2010 | volume= 103 | issue= 2 | pages= 85-9 | pmid=19892809 | doi=10.1093/qjmed/hcp159 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=19892809 }}</ref> | *In most of the patients [[metabolic]] imbalance may occur and it includes:<ref name="pmid1473414">{{cite journal| author=Jetmore AB, Timmcke AE, Gathright JB, Hicks TC, Ray JE, Baker JW| title=Ogilvie's syndrome: colonoscopic decompression and analysis of predisposing factors. | journal=Dis Colon Rectum | year= 1992 | volume= 35 | issue= 12 | pages= 1135-42 | pmid=1473414 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=1473414 }}</ref><ref name="pmid19892809">{{cite journal| author=Sandle GI, Hunter M| title=Apical potassium (BK) channels and enhanced potassium secretion in human colon. | journal=QJM | year= 2010 | volume= 103 | issue= 2 | pages= 85-9 | pmid=19892809 | doi=10.1093/qjmed/hcp159 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=19892809 }}</ref> | ||
**Hypokalemia | **[[Hypokalemia]] | ||
**Hypocalcemia | **[[Hypocalcemia]] | ||
**Hypomagnesemia | **[[Hypomagnesemia]] | ||
*General laboratory tests are performed to exclude other causes and disease. The laboratory tests include the following: | *General laboratory tests are performed to exclude other causes and disease. The laboratory tests include the following: | ||
**Complete blood count | **[[Complete blood count]] | ||
**Electrolytes balance | **[[Electrolyte disturbance|Electrolytes balance]] | ||
**Lactate levels | **[[Lactate levels raised|Lactate levels]] | ||
**Thyroid hormones level | **[[Thyroid hormone|Thyroid hormones level]] | ||
*To exclude acute abdominal pain diagnosis in patients suspected with perforation, the following laboratory tests are performed: | *To exclude acute [[abdominal pain]] diagnosis in patients suspected with [[perforation]], the following laboratory tests are performed: | ||
**Serum Aminotransferase | **[[Aminotransferases|Serum Aminotransferase]] | ||
**Serum Alkaline phosphatase | **[[Alkaline phosphatase|Serum Alkaline phosphatase]] | ||
**Bilirubin level | **[[Bilirubin]] level | ||
**Lipase level | **[[Lipase]] level | ||
*In patients with diarrhea, the following laboratory tests are performed:<ref name="pmid18653901">{{cite journal| author=Simon M, Duong JP, Mallet V, Jian R, MacLennan KA, Sandle GI et al.| title=Over-expression of colonic K+ channels associated with severe potassium secretory diarrhoea after haemorrhagic shock. | journal=Nephrol Dial Transplant | year= 2008 | volume= 23 | issue= 10 | pages= 3350-2 | pmid=18653901 | doi=10.1093/ndt/gfn411 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=18653901 }}</ref> | *In patients with diarrhea, the following laboratory tests are performed:<ref name="pmid18653901">{{cite journal| author=Simon M, Duong JP, Mallet V, Jian R, MacLennan KA, Sandle GI et al.| title=Over-expression of colonic K+ channels associated with severe potassium secretory diarrhoea after haemorrhagic shock. | journal=Nephrol Dial Transplant | year= 2008 | volume= 23 | issue= 10 | pages= 3350-2 | pmid=18653901 | doi=10.1093/ndt/gfn411 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=18653901 }}</ref> | ||
**Stool culture for Clostridia difficile | **[[Stool culture]] for [[Clostridia|Clostridia difficile]] | ||
==References== | ==References== | ||
Latest revision as of 16:49, 8 February 2018
| Ogilvie syndrome Microchapters
|
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Ogilvie syndrome laboratory findings On the Web |
|
American Roentgen Ray Society Images of Ogilvie syndrome laboratory findings |
|
Risk calculators and risk factors for Ogilvie syndrome laboratory findings |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ahmed Elsaiey, MBBCH [2]
Overview
There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include leukocytosis due to the underlying disease not due to the pseudo-obstruction itself. Many patients with Ogilvie syndrome may have metabolic imbalance which include hypokalemiaand hypocalcemia. Other laboratory tests that can be performed to exclude other causes include complete blood count, lactate levels, and thyroid hormonelevels.
Laboratory Findings
- There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include leukocytosis due to the underlying disease not due to the pseudo-obstruction itself.[1]
- In most of the patients metabolic imbalance may occur and it includes:[2][3]
- General laboratory tests are performed to exclude other causes and disease. The laboratory tests include the following:
- To exclude acute abdominal pain diagnosis in patients suspected with perforation, the following laboratory tests are performed:
- In patients with diarrhea, the following laboratory tests are performed:[4]
References
- ↑ Vanek VW, Al-Salti M (1986). "Acute pseudo-obstruction of the colon (Ogilvie's syndrome). An analysis of 400 cases". Dis Colon Rectum. 29 (3): 203–10. PMID 3753674.
- ↑ Jetmore AB, Timmcke AE, Gathright JB, Hicks TC, Ray JE, Baker JW (1992). "Ogilvie's syndrome: colonoscopic decompression and analysis of predisposing factors". Dis Colon Rectum. 35 (12): 1135–42. PMID 1473414.
- ↑ Sandle GI, Hunter M (2010). "Apical potassium (BK) channels and enhanced potassium secretion in human colon". QJM. 103 (2): 85–9. doi:10.1093/qjmed/hcp159. PMID 19892809.
- ↑ Simon M, Duong JP, Mallet V, Jian R, MacLennan KA, Sandle GI; et al. (2008). "Over-expression of colonic K+ channels associated with severe potassium secretory diarrhoea after haemorrhagic shock". Nephrol Dial Transplant. 23 (10): 3350–2. doi:10.1093/ndt/gfn411. PMID 18653901.