Norovirus: Difference between revisions

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===Less Common Causes===
===Less Common Causes===


==Virology==
==Life Cycle==
==Life Cycle==
Norovirus has a cytoplasmic replication. It attaches to the host receptors and enters the cell through endocytosis. Since, it is a positive sense virus, replication and transcription follows the corresponding models for positive stranded RNA viruses. Translation occurs by leaky scanning, and RNA termination-reinitiation.<ref name="ViralZone" />
Norovirus has a cytoplasmic replication. It attaches to the host receptors and enters the cell through endocytosis. Since, it is a positive sense virus, replication and transcription follows the corresponding models for positive stranded RNA viruses. Translation occurs by leaky scanning, and RNA termination-reinitiation.<ref name="ViralZone" />

Revision as of 18:54, 19 February 2021

Norovirus infection Microchapters

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Overview

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Classification

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Causes

Differentiating Norovirus infection from other Diseases

Epidemiology and Demographics

Risk Factors

Natural History, Complications and Prognosis

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Risk calculators and risk factors for Norovirus

This page is about microbiologic aspects of the organism(s).  For clinical aspects of the disease, see Norovirus infection.

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

Overview

Norovirus is the cause of norovirus infection. Noroviruses (genus Norovirus) are a group of related, single-stranded RNA, nonenveloped viruses that cause acute gastroenteritis in humans. Noroviruses belong to the family Caliciviridae.

Causes

Common Causes

Less Common Causes

Life Cycle

Norovirus has a cytoplasmic replication. It attaches to the host receptors and enters the cell through endocytosis. Since, it is a positive sense virus, replication and transcription follows the corresponding models for positive stranded RNA viruses. Translation occurs by leaky scanning, and RNA termination-reinitiation.[1]

Genus Host Details Tissue Tropism Entry Details Release Details Replication Site Assembly Site Transmission
Norovirus Humans; mammals Intestinal epithelium Cell receptor endocytosis Lysis Cytoplasm Cytoplasm Oral-fecal

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References

  1. 2.0 2.1 2.2 2.3 2.4 2.5 2.6 "Public Health Image Library (PHIL)".


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