Hepatic hemangioma pathophysiology: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
* | * They arise from the endothelial cells that line the blood vessels and consists of multiple, large vascular channels lined by a single layer of endothelial cells and supported by collagenous walls. | ||
* They may be associated with [[focal nodular hyperplasia]]. | * They may be associated with [[focal nodular hyperplasia]]. | ||
* May be associated with [[Kasabach-Merritt syndrome]], [[ | * May be associated with [[Kasabach-Merritt syndrome]], [[hemolytic anemia]] and consumptive coagulopathy. | ||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Gastroenterology]] | |||
[[Category:Hepatology]] | |||
[[Category:Oncology]] | |||
[[Category:Surgery]] | |||
[[Category:Disease]] | [[Category:Disease]] | ||
[[Category:Needs overview]] | [[Category:Needs overview]] | ||
[[Category:needs content]] | |||
Revision as of 21:37, 7 March 2013
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Hepatic hemangioma Microchapters |
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Hepatic hemangioma pathophysiology On the Web |
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American Roentgen Ray Society Images of Hepatic hemangioma pathophysiology |
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Risk calculators and risk factors for Hepatic hemangioma pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Pathophysiology
- They arise from the endothelial cells that line the blood vessels and consists of multiple, large vascular channels lined by a single layer of endothelial cells and supported by collagenous walls.
- They may be associated with focal nodular hyperplasia.
- May be associated with Kasabach-Merritt syndrome, hemolytic anemia and consumptive coagulopathy.