Endometriosis pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
==Pathophysiology== | |||
The exact pathogenesis of endometriosis is still unknown, however several theories were put forward to explain the the presence the of viable and hormonally active endometrium outside the uterine cavity. The theories proposed include the following: | |||
*'''Sampson's theory of retrograde menstruation:''' The theory explains that the viable endometrial tissue passes in a retrograde fashion via the Fallopian tubes to reach the peritoneal cavity and subsequently implants onto the pelvic structures and organs. | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
Revision as of 20:11, 13 June 2017
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Endometriosis Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Endometriosis pathophysiology On the Web |
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American Roentgen Ray Society Images of Endometriosis pathophysiology |
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Risk calculators and risk factors for Endometriosis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aravind Kuchkuntla, M.B.B.S[2]
Overview
Pathophysiology
The exact pathogenesis of endometriosis is still unknown, however several theories were put forward to explain the the presence the of viable and hormonally active endometrium outside the uterine cavity. The theories proposed include the following:
- Sampson's theory of retrograde menstruation: The theory explains that the viable endometrial tissue passes in a retrograde fashion via the Fallopian tubes to reach the peritoneal cavity and subsequently implants onto the pelvic structures and organs.