Atrial fibrillation differential diagnosis: Difference between revisions

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{{Atrial fibrillation}}
{{Atrial fibrillation}}
{{CMG}}
{{CMG}}; {{AE}} {{HK}} {{Anahita}}


==Overview==
==Overview==
Atrial fibrillation must be distinguished from other common [[atrial arrhythmias]], which include [[atrial flutter]], [[atrial tachycardia]], [[paroxysmal supraventricular tachycardia]], [[Wolff-Parkinson-White syndrome]], and [[atrioventricular nodal reentry tachycardia]].
[[Atrial fibrillation]] has to be differentiated from other [[diseases]] such as [[atrial flutter]], [[atrial tachycardia]], [[atrioventricular nodal reentry tachycardia]] ([[Atrioventricular nodal reentry tachycardia|AVNRT]]), [[multifocal atrial tachycardia]], [[paroxysmal supraventricular tachycardia]] and [[Wolff-Parkinson-White syndrome]]. The differentiating features are largely based on both [[The electrocardiogram|EKG]] findings and [[Circulatory system|cardiovascular]] [[Physical examination|examinations]].


==Differentiating Atrial Fibrillation from other Diseases==
==Differentiating Atrial Fibrillation from other Diseases==
Atrial fibrillation has to be differnetiated from other diseases like:
[[Atrial fibrillation]] has to be differentiated from other [[diseases]] such as:<ref name="pmid28835836">{{cite journal |vauthors=Cosío FG |title=Atrial Flutter, Typical and Atypical: A Review |journal=Arrhythm Electrophysiol Rev |volume=6 |issue=2 |pages=55–62 |date=June 2017 |pmid=28835836 |pmc=5522718 |doi=10.15420/aer.2017.5.2 |url=}}</ref><ref name="pmid27617092">{{cite journal |vauthors=Katritsis DG, Josephson ME |title=Classification, Electrophysiological Features and Therapy of Atrioventricular Nodal Reentrant Tachycardia |journal=Arrhythm Electrophysiol Rev |volume=5 |issue=2 |pages=130–5 |date=August 2016 |pmid=27617092 |pmc=5013176 |doi=10.15420/AER.2016.18.2 |url=}}</ref>
*[[Atrial flutter]]
*[[Atrial flutter]]
*[[Atrial tachycardia]]
*[[Atrial tachycardia]]
*[[Atrioventricular nodal reentry tachycardia]] ([[AVNRT]])
*[[Atrioventricular nodal reentry tachycardia]] ([[Atrioventricular nodal reentry tachycardia|AVNRT]])
*[[Multifocal atrial tachycardia]]
*[[Multifocal atrial tachycardia]]
*[[Paroxysmal supraventricular tachycardia]]
*[[Paroxysmal supraventricular tachycardia]]
*[[Wolff-Parkinson-White syndrome]]
*[[Wolff-Parkinson-White syndrome]]


The differentiating features are largely based on both EKG findings and cardiovascular examination.  
The differentiating features are largely based on both [[The electrocardiogram|EKG]] findings and [[Circulatory system|cardiovascular]] [[Physical examination|examinations]].<ref name="pmid24837984">{{cite journal |vauthors=Lankveld TA, Zeemering S, Crijns HJ, Schotten U |title=The ECG as a tool to determine atrial fibrillation complexity |journal=Heart |volume=100 |issue=14 |pages=1077–84 |date=July 2014 |pmid=24837984 |doi=10.1136/heartjnl-2013-305149 |url=}}</ref><ref name="pmid22518390">{{cite journal |vauthors=Harris K, Edwards D, Mant J |title=How can we best detect atrial fibrillation? |journal=J R Coll Physicians Edinb |volume=42 Suppl 18 |issue= |pages=5–22 |date=2012 |pmid=22518390 |doi=10.4997/JRCPE.2012.S02 |url=}}</ref>
* Atrial fibrillation is irregularly irregular, while the other rhythms such as [[atrial flutter]], [[sinus tachycardia]], [[AV nodal reentry tachycardia]] and [[paroxysmal supraventricular tachycardia]] are all much more regular.
*[[Atrial fibrillation]] is irregularly irregular, while the other conditions such as [[atrial flutter]], [[sinus tachycardia]], [[AV nodal reentry tachycardia]] and [[paroxysmal supraventricular tachycardia]] are all much more regular.
* An atrioventricular nodal reentry tachycardia will often break with either carotid sinus massage or AV nodal blocking agents.
*An [[atrioventricular nodal reentry tachycardia]] will often break with either [[Carotid sinus|carotid sinus massage]] or [[Atrioventricular node|AV nodal]] blocking agents.
* If the patient has [[Wolff-Parkinson-White syndrome]] there may be much more rapid conduction. The presence of the [[delta wave]] on EKG is characteristic.
*If the [[patient]] has [[Wolff-Parkinson-White syndrome]] there may be much more rapid conduction. The presence of the [[delta wave]] on [[The electrocardiogram|EKG]] is characteristic for [[Wolff-Parkinson-White syndrome]].


<br />
<br />
{| class="wikitable"
{| class="wikitable"
|+
|+
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Arrhythmia
! align="center" style="background:#4479BA; color: #FFFFFF;" + |[[Arrhythmia]]
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Rhythm
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Rhythm
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Rate
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Rate
! align="center" style="background:#4479BA; color: #FFFFFF;" + |P wave
! align="center" style="background:#4479BA; color: #FFFFFF;" + |[[P wave]]
! align="center" style="background:#4479BA; color: #FFFFFF;" + |PR Interval
! align="center" style="background:#4479BA; color: #FFFFFF;" + |[[PR Interval]]
! align="center" style="background:#4479BA; color: #FFFFFF;" + |QRS Complex
! align="center" style="background:#4479BA; color: #FFFFFF;" + |[[QRS Complex]]
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Response to Maneuvers
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Response to Maneuvers
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Epidemiology
! align="center" style="background:#4479BA; color: #FFFFFF;" + |[[Epidemiology]]
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Co-existing Conditions
! align="center" style="background:#4479BA; color: #FFFFFF;" + |Co-existing Conditions
|-
|-
|'''Atrial Fibrillation (AFib)<ref name="pmid24837984">{{cite journal |vauthors=Lankveld TA, Zeemering S, Crijns HJ, Schotten U |title=The ECG as a tool to determine atrial fibrillation complexity |journal=Heart |volume=100 |issue=14 |pages=1077–84 |date=July 2014 |pmid=24837984 |doi=10.1136/heartjnl-2013-305149 |url=}}</ref><ref name="pmid22518390">{{cite journal |vauthors=Harris K, Edwards D, Mant J |title=How can we best detect atrial fibrillation? |journal=J R Coll Physicians Edinb |volume=42 Suppl 18 |issue= |pages=5–22 |date=2012 |pmid=22518390 |doi=10.4997/JRCPE.2012.S02 |url=}}</ref>'''
|'''[[Atrial Fibrillation]] ([[Atrial Fibrillation|AFib]])<ref name="pmid24837984">{{cite journal |vauthors=Lankveld TA, Zeemering S, Crijns HJ, Schotten U |title=The ECG as a tool to determine atrial fibrillation complexity |journal=Heart |volume=100 |issue=14 |pages=1077–84 |date=July 2014 |pmid=24837984 |doi=10.1136/heartjnl-2013-305149 |url=}}</ref><ref name="pmid22518390">{{cite journal |vauthors=Harris K, Edwards D, Mant J |title=How can we best detect atrial fibrillation? |journal=J R Coll Physicians Edinb |volume=42 Suppl 18 |issue= |pages=5–22 |date=2012 |pmid=22518390 |doi=10.4997/JRCPE.2012.S02 |url=}}</ref>'''
|
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* Irregularly irregular
* Irregularly irregular
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* Does not break with [[adenosine]] or [[vagal maneuvers]]
* Does not break with [[adenosine]] or [[vagal maneuvers]]
|
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* 2.7–6.1 million people in the United States have AFib
* 2.7–6.1 million people in the United States have [[atrial fibrillation]]
* 2% of people younger than age 65 have AFib, while about 9% of people aged 65 years or older have AFib
* 2% of people younger than age 65 have [[atrial fibrillation]], while about 9% of people aged 65 years or older have [[atrial fibrillation]]
|
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* Elderly
* [[Old age|Elderly]]
* Following [[Coronary artery bypass surgery|bypass surgery]]
* Following [[Coronary artery bypass surgery|bypass surgery]]
*[[Mitral valve disease]]
*[[Mitral valve disease]]
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* 75 (4:1 block), 100 (3:1 block) and 150 (2:1 block) beats per minute (bpm), but 150 is more common
* 75 (4:1 block), 100 (3:1 block) and 150 (2:1 block) beats per minute (bpm), but 150 is more common
|
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* Sawtooth pattern of P waves at 250 to 350 bpm
* Sawtooth pattern of [[P waves]] at 250 to 350 bpm
*Biphasic deflection in V1
*Biphasic deflection in V1
|
|
* Varies depending upon the magnitude of the block, but is short
* Varies depending upon the magnitude of the block, but it is usually short
|
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* Less than 0.12 seconds, consistent, and normal in morphology
* Less than 0.12 seconds, consistent, and normal in morphology
|
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* Conduction may vary in response to drugs and maneuvers dropping the rate from 150 to 100 or to 75 bpm
* Conduction may vary in response to [[medications]] and maneuvers dropping the rate from 150 to 100 or to 75 bpm
|
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*[[Incidence]]: 88 per 100,000 individuals
*[[Incidence]]: 88 per 100,000 individuals
|
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*[[Elderly]]
*[[old age|Elderly]]
*[[Alcohol]]
*[[Alcohol]]
|-
|-
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* 140-280 bpm
* 140-280 bpm
|
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*Slow-Fast AVNRT:
*Slow-Fast [[AV nodal reentrant tachycardia]] ([[AV nodal reentrant tachycardia|AVNRT]]):
**Pseudo-S wave in leads II, III, and AVF
**Pseudo-[[QRS complex|S wave]] in leads II, III, and AVF
**Pseudo-R' in lead V1.
**Pseudo-[[QRS complex|R wave]] in lead V1.
*Fast-Slow AVNRT
*Fast-Slow [[AV nodal reentrant tachycardia]] ([[AV nodal reentrant tachycardia|AVNRT]])
**[[P waves]] between the [[QRS complex|QRS]] and [[T waves]] (QRS-P-T complexes)
**[[P waves]] between the [[QRS complex|QRS]] and [[T waves]] (QRS-P-T complexes)
*Slow-Slow AVNRT
*Slow-Slow [[AV nodal reentrant tachycardia]] ([[AV nodal reentrant tachycardia|AVNRT]])
**Late [[P waves]] after a [[QRS complex|QRS]]
**Late [[P waves]] after a [[QRS complex|QRS]]
**Often appears as [[atrial tachycardia]].
**Often appears as [[atrial tachycardia]].
*Inverted, superimposed on or buried within the [[QRS complex]] (pseudo R prime in V1/pseudo S wave in inferior leads)
*Inverted, superimposed on or buried within the [[QRS complex]] (pseudo R prime in V1/pseudo [[QRS complex|S wave]] in inferior leads)
|
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* Absent ([[P wave]] can appear after the QRS complex and before the T wave, and in atypical AVNRT, the [[P wave]] can appear just before the [[QRS complex]])
* Absent ([[P wave]] can appear after the [[QRS complex]] and before the [[T wave]], and in atypical [[AV nodal reentrant tachycardia]], the [[P wave]] can appear just before the [[QRS complex]])
|
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* Less than 0.12 seconds, consistent, and normal in morphology in the absence of aberrant conduction
* Less than 0.12 seconds, consistent, and normal in morphology in the absence of aberrant conduction
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* Absence of one dominant atrial pacemaker, can be mistaken for [[atrial fibrillation]] if the [[P waves]] are of low amplitude
* Absence of one dominant atrial pacemaker, can be mistaken for [[atrial fibrillation]] if the [[P waves]] are of low amplitude
|
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* Variable [[PR interval|PR intervals]], RR intervals, and PP intervals
* Variable [[PR interval|PR intervals]], [[RR interval|RR intervals]], and [[PP interval|PP intervals]]
|
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* Less than 0.12 seconds, consistent, and normal in morphology
* Less than 0.12 seconds, consistent, and normal in morphology
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* 0.05% to 0.32% of [[electrocardiograms]] in general hospital admissions
* 0.05% to 0.32% of [[electrocardiograms]] in general hospital admissions
|
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*[[Elderly]]
*[[old age|Elderly]]
*[[Chronic obstructive pulmonary disease]] ([[Chronic obstructive pulmonary disease|COPD]])
*[[Chronic obstructive pulmonary disease]] ([[Chronic obstructive pulmonary disease|COPD]])
|-
|-
|'''Paroxysmal Supraventricular Tachycardia'''
|'''[[Paroxysmal Supraventricular Tachycardia]]'''
|
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* Regular
* Regular
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* Absent
* Absent
* Hidden in [[QRS complex|QRS]]
* Hidden in [[QRS complex]]
|
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* Absent
* Absent
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|'''[[Premature atrial contraction|Premature Atrial Contractrions]] ([[Premature atrial contraction|PAC]])'''<ref name="pmid26316525">{{cite journal |vauthors=Lin CY, Lin YJ, Chen YY, Chang SL, Lo LW, Chao TF, Chung FP, Hu YF, Chong E, Cheng HM, Tuan TC, Liao JN, Chiou CW, Huang JL, Chen SA |title=Prognostic Significance of Premature Atrial Complexes Burden in Prediction of Long-Term Outcome |journal=J Am Heart Assoc |volume=4 |issue=9 |pages=e002192 |date=August 2015 |pmid=26316525 |pmc=4599506 |doi=10.1161/JAHA.115.002192 |url=}}</ref><ref name="pmid18063110">{{cite journal |vauthors=Strasburger JF, Cheulkar B, Wichman HJ |title=Perinatal arrhythmias: diagnosis and management |journal=Clin Perinatol |volume=34 |issue=4 |pages=627–52, vii–viii |date=December 2007 |pmid=18063110 |pmc=3310372 |doi=10.1016/j.clp.2007.10.002 |url=}}</ref>
|'''[[Premature atrial contraction|Premature Atrial Contractrions]] ([[Premature atrial contraction|PAC]])'''<ref name="pmid26316525">{{cite journal |vauthors=Lin CY, Lin YJ, Chen YY, Chang SL, Lo LW, Chao TF, Chung FP, Hu YF, Chong E, Cheng HM, Tuan TC, Liao JN, Chiou CW, Huang JL, Chen SA |title=Prognostic Significance of Premature Atrial Complexes Burden in Prediction of Long-Term Outcome |journal=J Am Heart Assoc |volume=4 |issue=9 |pages=e002192 |date=August 2015 |pmid=26316525 |pmc=4599506 |doi=10.1161/JAHA.115.002192 |url=}}</ref><ref name="pmid18063110">{{cite journal |vauthors=Strasburger JF, Cheulkar B, Wichman HJ |title=Perinatal arrhythmias: diagnosis and management |journal=Clin Perinatol |volume=34 |issue=4 |pages=627–52, vii–viii |date=December 2007 |pmid=18063110 |pmc=3310372 |doi=10.1016/j.clp.2007.10.002 |url=}}</ref>
|
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* Regular except when disturbed by premature beat(s)
* Regular except when disturbed by [[Premature atrial contraction|premature beat(s)]]
|
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* 80-120 bpm
* 80-120 bpm
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* > 0.12 second  
* > 0.12 second  
* May be shorter than that in normal sinus rhythm (NSR) if the origin of PAC is located closer to the AV node
* May be shorter than that in normal [[sinus rhythm]] ([[[[sinus rhythm|SR]]) if the origin of [[premature atrial contraction]] ([[Premature atrial contraction|PAC]]) is located closer to the [[Atrioventricular node|AV node]]
*Ashman’s Phenomenon:
*[[Ashman phenomenon]]:
**[[Premature atrial contraction|PAC]] displaying a [[right bundle branch block]] pattern
**[[Premature atrial contraction|PAC]] displaying a [[right bundle branch block]] pattern
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* Regular
* Regular
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* Atrial rate is nearly 300 bpm and ventricular rate is at 150 bpm
* [[atrium|Atrial]] rate is nearly 300 bpm and [[ventricle|ventricular]] rate is at 150 bpm
|
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* With [[orthodromic]] conduction due to a bypass tract, the [[P wave]] generally follows the [[QRS complex]], whereas in [[AVNRT]], the [[P wave]] is generally buried in the [[QRS complex]].
* With [[orthodromic]] conduction due to a bypass tract, the [[P wave]] generally follows the [[QRS complex]], whereas in [[AVNRT]], the [[P wave]] is generally buried in the [[QRS complex]].
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* Less than 0.12 seconds
* Less than 0.12 seconds
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* A [[delta wave]] and evidence of [[ventricular]] pre-excitation if there is conduction to the ventricle via ante-grade conduction down an accessory pathway
* A [[delta wave]] and evidence of [[ventricular]] pre-excitation if there is conduction to the [[ventricle]] via ante-grade conduction down an accessory pathway
* A [[delta wave]] and pre-excitation may not be present because bypass tracts do not conduct ante-grade.
* A [[delta wave]] and pre-excitation may not be present because bypass tracts do not conduct ante-grade.
|
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*[[Ebstein's anomaly]]
*[[Ebstein's anomaly]]
*[[Mitral valve prolapse]]: This cardiac disorder, if present, is associated with left-sided accessory pathways.
*[[Mitral valve prolapse]]: This [[heart|cardiac]] disorder, if present, is associated with left-sided accessory pathways.
*[[Hypertrophic cardiomyopathy]]: This disorder is associated with familial/inherited form of [[Wolff-Parkinson-White syndrome|WPW syndrome]].
*[[Hypertrophic cardiomyopathy]]: This disorder is associated with familial/inherited form of [[Wolff-Parkinson-White syndrome|WPW syndrome]].
*[[Hypokalemic periodic paralysis]]
*[[Hypokalemic periodic paralysis]]
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*[[Tuberous sclerosis]]
*[[Tuberous sclerosis]]
|-
|-
|'''[[Ventricular fibrillation|Ventricular Fibrillation]] (VF)'''<ref name="pmid27899944">{{cite journal |vauthors=Glinge C, Sattler S, Jabbari R, Tfelt-Hansen J |title=Epidemiology and genetics of ventricular fibrillation during acute myocardial infarction |journal=J Geriatr Cardiol |volume=13 |issue=9 |pages=789–797 |date=September 2016 |pmid=27899944 |pmc=5122505 |doi=10.11909/j.issn.1671-5411.2016.09.006 |url=}}</ref><ref name="pmid11334828">{{cite journal |vauthors=Samie FH, Jalife J |title=Mechanisms underlying ventricular tachycardia and its transition to ventricular fibrillation in the structurally normal heart |journal=Cardiovasc. Res. |volume=50 |issue=2 |pages=242–50 |date=May 2001 |pmid=11334828 |doi=10.1016/s0008-6363(00)00289-3 |url=}}</ref><ref name="pmid20142817">{{cite journal |vauthors=Adabag AS, Luepker RV, Roger VL, Gersh BJ |title=Sudden cardiac death: epidemiology and risk factors |journal=Nat Rev Cardiol |volume=7 |issue=4 |pages=216–25 |date=April 2010 |pmid=20142817 |pmc=5014372 |doi=10.1038/nrcardio.2010.3 |url=}}</ref>
|'''[[Ventricular fibrillation|Ventricular Fibrillation]] ([[Ventricular fibrillation|VF]])'''<ref name="pmid27899944">{{cite journal |vauthors=Glinge C, Sattler S, Jabbari R, Tfelt-Hansen J |title=Epidemiology and genetics of ventricular fibrillation during acute myocardial infarction |journal=J Geriatr Cardiol |volume=13 |issue=9 |pages=789–797 |date=September 2016 |pmid=27899944 |pmc=5122505 |doi=10.11909/j.issn.1671-5411.2016.09.006 |url=}}</ref><ref name="pmid11334828">{{cite journal |vauthors=Samie FH, Jalife J |title=Mechanisms underlying ventricular tachycardia and its transition to ventricular fibrillation in the structurally normal heart |journal=Cardiovasc. Res. |volume=50 |issue=2 |pages=242–50 |date=May 2001 |pmid=11334828 |doi=10.1016/s0008-6363(00)00289-3 |url=}}</ref><ref name="pmid20142817">{{cite journal |vauthors=Adabag AS, Luepker RV, Roger VL, Gersh BJ |title=Sudden cardiac death: epidemiology and risk factors |journal=Nat Rev Cardiol |volume=7 |issue=4 |pages=216–25 |date=April 2010 |pmid=20142817 |pmc=5014372 |doi=10.1038/nrcardio.2010.3 |url=}}</ref>
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* Irregular
* Irregular
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* Absent
* Absent
|
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* Absent (R on T phenomenon in the setting of ischemia)
* Absent (R on T phenomenon in the setting of [[ischemia]])
|
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* Does not break in response to [[procainamide]], [[adenosine]], [[vagal maneuvers]]
* Does not break in response to [[procainamide]], [[adenosine]], [[vagal maneuvers]]
|
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* 3-12% cases of [[acute myocardial infarction]] (AMI)
* 3-12% cases of [[acute myocardial infarction]] ([[acute myocardial infarction|AMI]])
* Out of 356,500 out of hospital cardiac arrests, 23% have VF as initial rhythm
* Out of 356,500 out of hospital [[Cardiac arrest resident survival guide|cardiac arrests]], 23% have [[ventricular fibrillation]] ([[Ventricular fibrillation|VF]]) as initial rhythm
|
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*[[Myocardial ischemia]] / [[Myocardial infarction|infarction]]
*[[Myocardial ischemia]] / [[Myocardial infarction|infarction]]
*[[Cardiomyopathy]]
*[[Cardiomyopathy]]
* Channelopathies e.g. Long QT (acquired / congenital)
*[[Channelopathy|Channelopathies]] such as [[long QT syndrome]] (acquired / congenital)
*Electrolyte abnormalities ([[hypokalemia]]/[[hyperkalemia]], [[hypomagnesemia]])
*[[Electrolyte disturbance|Electrolyte abnormalities]] ([[hypokalemia]]/[[hyperkalemia]], [[hypomagnesemia]])
*[[Aortic stenosis]]
*[[Aortic stenosis]]
*[[Aortic dissection]]
*[[Aortic dissection]]
*[[Myocarditis]]
*[[Myocarditis]]
*[[Cardiac tamponade]]
*[[Cardiac tamponade]]
* Blunt trauma (Commotio Cordis)
* [[Blunt trauma]] [[Commotio Cordis]])
*[[Sepsis]]
*[[Sepsis]]
*[[Hypothermia]]
*[[Hypothermia]]
Line 260: Line 260:


*Absent
*Absent
*Initial [[R wave]] in V1, initial r > 40 ms in V1/V2, notched S in V1, initial R in aVR, lead II R wave peak time ≥50 ms, no RS in V1-V6, and atrioventricular dissociation
*Initial [[R wave]] in V1, initial r > 40 ms in V1/V2, notched S in V1, initial R in aVR, lead II R wave peak time ≥50 ms, no RS in V1-V6, and [[atrioventricular dissociation]]|
|
* Wide complex, [[QRS complex|QRS]] duration > 120 milliseconds
* Wide complex, [[QRS complex|QRS]] duration > 120 milliseconds
|
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* Does not break in response to [[procainamide]], [[adenosine]], [[vagal maneuvers]]
* Does not break in response to [[procainamide]], [[adenosine]], [[vagal maneuvers]]
|
|
* 5-10% of patients presenting with AMI
* 5-10% of patients presenting with [[acute myocardial ifarction]]|  
|
*[[Coronary artery disease]]
*[[Coronary artery disease]]
*[[Aortic stenosis]]
*[[Aortic stenosis]]

Latest revision as of 23:53, 29 August 2021
Resident
Survival
Guide


Sinus rhythm
Atrial fibrillation

Atrial Fibrillation Microchapters

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Risk calculators and risk factors for Atrial fibrillation differential diagnosis

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Syed Hassan A. Kazmi BSc, MD [2] Anahita Deylamsalehi, M.D.[3]

Overview

Atrial fibrillation has to be differentiated from other diseases such as atrial flutter, atrial tachycardia, atrioventricular nodal reentry tachycardia (AVNRT), multifocal atrial tachycardia, paroxysmal supraventricular tachycardia and Wolff-Parkinson-White syndrome. The differentiating features are largely based on both EKG findings and cardiovascular examinations.

Differentiating Atrial Fibrillation from other Diseases

Atrial fibrillation has to be differentiated from other diseases such as:[1][2]

The differentiating features are largely based on both EKG findings and cardiovascular examinations.[3][4]


Arrhythmia Rhythm Rate P wave PR Interval QRS Complex Response to Maneuvers Epidemiology Co-existing Conditions
Atrial Fibrillation (AFib)[3][4]
  • Irregularly irregular
  • Absent
  • Fibrillatory waves
  • Absent
  • Less than 0.12 seconds, consistent, and normal in morphology in the absence of aberrant conduction
Atrial Flutter[1]
  • Regular or Irregular
  • 75 (4:1 block), 100 (3:1 block) and 150 (2:1 block) beats per minute (bpm), but 150 is more common
  • Sawtooth pattern of P waves at 250 to 350 bpm
  • Biphasic deflection in V1
  • Varies depending upon the magnitude of the block, but it is usually short
  • Less than 0.12 seconds, consistent, and normal in morphology
  • Conduction may vary in response to medications and maneuvers dropping the rate from 150 to 100 or to 75 bpm
Atrioventricular nodal reentry tachycardia (AVNRT)[2][5][6][7]
  • Regular
  • 140-280 bpm
  • Less than 0.12 seconds, consistent, and normal in morphology in the absence of aberrant conduction
  • QRS alternans may be present
Multifocal Atrial Tachycardia[8][9]
  • Irregular
  • Atrial rate is > 100 beats per minute
  • Varying morphology from at least three different foci
  • Absence of one dominant atrial pacemaker, can be mistaken for atrial fibrillation if the P waves are of low amplitude
  • Less than 0.12 seconds, consistent, and normal in morphology
Paroxysmal Supraventricular Tachycardia
  • Regular
  • 150 and 240 bpm
  • Absent
  • Narrow complexes (< 0.12 s)
Premature Atrial Contractrions (PAC)[10][11]
  • 80-120 bpm
  • Upright
  • Usually narrow (< 0.12 s)
Wolff-Parkinson-White Syndrome[12][13]
  • Regular
  • Less than 0.12 seconds
  • A delta wave and evidence of ventricular pre-excitation if there is conduction to the ventricle via ante-grade conduction down an accessory pathway
  • A delta wave and pre-excitation may not be present because bypass tracts do not conduct ante-grade.
Ventricular Fibrillation (VF)[14][15][16]
  • Irregular
  • 150 to 500 bpm
  • Absent
  • Absent
  • Absent (R on T phenomenon in the setting of ischemia)
Ventricular Tachycardia[17][18]
  • Regular
  • > 100 bpm (150-200 bpm common)
  • Absent
  • Absent
  • Initial R wave in V1, initial r > 40 ms in V1/V2, notched S in V1, initial R in aVR, lead II R wave peak time ≥50 ms, no RS in V1-V6, and atrioventricular dissociation|
  • Wide complex, QRS duration > 120 milliseconds

References

  1. ↑ 1.0 1.1 Cosío FG (June 2017). "Atrial Flutter, Typical and Atypical: A Review". Arrhythm Electrophysiol Rev. 6 (2): 55–62. doi:10.15420/aer.2017.5.2. PMC 5522718. PMID 28835836.
  2. ↑ 2.0 2.1 Katritsis DG, Josephson ME (August 2016). "Classification, Electrophysiological Features and Therapy of Atrioventricular Nodal Reentrant Tachycardia". Arrhythm Electrophysiol Rev. 5 (2): 130–5. doi:10.15420/AER.2016.18.2. PMC 5013176. PMID 27617092.
  3. ↑ 3.0 3.1 Lankveld TA, Zeemering S, Crijns HJ, Schotten U (July 2014). "The ECG as a tool to determine atrial fibrillation complexity". Heart. 100 (14): 1077–84. doi:10.1136/heartjnl-2013-305149. PMID 24837984.
  4. ↑ 4.0 4.1 Harris K, Edwards D, Mant J (2012). "How can we best detect atrial fibrillation?". J R Coll Physicians Edinb. 42 Suppl 18: 5–22. doi:10.4997/JRCPE.2012.S02. PMID 22518390.
  5. ↑ Letsas KP, Weber R, Siklody CH, Mihas CC, Stockinger J, Blum T, Kalusche D, Arentz T (April 2010). "Electrocardiographic differentiation of common type atrioventricular nodal reentrant tachycardia from atrioventricular reciprocating tachycardia via a concealed accessory pathway". Acta Cardiol. 65 (2): 171–6. doi:10.2143/AC.65.2.2047050. PMID 20458824.
  6. ↑ "Atrioventricular Nodal Reentry Tachycardia (AVNRT) - StatPearls - NCBI Bookshelf".
  7. ↑ Schernthaner C, Danmayr F, Strohmer B (2014). "Coexistence of atrioventricular nodal reentrant tachycardia with other forms of arrhythmias". Med Princ Pract. 23 (6): 543–50. doi:10.1159/000365418. PMC 5586929. PMID 25196716.
  8. ↑ Scher DL, Arsura EL (September 1989). "Multifocal atrial tachycardia: mechanisms, clinical correlates, and treatment". Am. Heart J. 118 (3): 574–80. doi:10.1016/0002-8703(89)90275-5. PMID 2570520.
  9. ↑ Goodacre S, Irons R (March 2002). "ABC of clinical electrocardiography: Atrial arrhythmias". BMJ. 324 (7337): 594–7. doi:10.1136/bmj.324.7337.594. PMC 1122515. PMID 11884328.
  10. ↑ Lin CY, Lin YJ, Chen YY, Chang SL, Lo LW, Chao TF, Chung FP, Hu YF, Chong E, Cheng HM, Tuan TC, Liao JN, Chiou CW, Huang JL, Chen SA (August 2015). "Prognostic Significance of Premature Atrial Complexes Burden in Prediction of Long-Term Outcome". J Am Heart Assoc. 4 (9): e002192. doi:10.1161/JAHA.115.002192. PMC 4599506. PMID 26316525.
  11. ↑ Strasburger JF, Cheulkar B, Wichman HJ (December 2007). "Perinatal arrhythmias: diagnosis and management". Clin Perinatol. 34 (4): 627–52, vii–viii. doi:10.1016/j.clp.2007.10.002. PMC 3310372. PMID 18063110.
  12. ↑ Rao AL, Salerno JC, Asif IM, Drezner JA (July 2014). "Evaluation and management of wolff-Parkinson-white in athletes". Sports Health. 6 (4): 326–32. doi:10.1177/1941738113509059. PMC 4065555. PMID 24982705.
  13. ↑ Rosner MH, Brady WJ, Kefer MP, Martin ML (November 1999). "Electrocardiography in the patient with the Wolff-Parkinson-White syndrome: diagnostic and initial therapeutic issues". Am J Emerg Med. 17 (7): 705–14. doi:10.1016/s0735-6757(99)90167-5. PMID 10597097.
  14. ↑ Glinge C, Sattler S, Jabbari R, Tfelt-Hansen J (September 2016). "Epidemiology and genetics of ventricular fibrillation during acute myocardial infarction". J Geriatr Cardiol. 13 (9): 789–797. doi:10.11909/j.issn.1671-5411.2016.09.006. PMC 5122505. PMID 27899944.
  15. ↑ Samie FH, Jalife J (May 2001). "Mechanisms underlying ventricular tachycardia and its transition to ventricular fibrillation in the structurally normal heart". Cardiovasc. Res. 50 (2): 242–50. doi:10.1016/s0008-6363(00)00289-3. PMID 11334828.
  16. ↑ Adabag AS, Luepker RV, Roger VL, Gersh BJ (April 2010). "Sudden cardiac death: epidemiology and risk factors". Nat Rev Cardiol. 7 (4): 216–25. doi:10.1038/nrcardio.2010.3. PMC 5014372. PMID 20142817.
  17. ↑ Koplan BA, Stevenson WG (March 2009). "Ventricular tachycardia and sudden cardiac death". Mayo Clin. Proc. 84 (3): 289–97. doi:10.1016/S0025-6196(11)61149-X. PMC 2664600. PMID 19252119.
  18. ↑ Levis JT (2011). "ECG Diagnosis: Monomorphic Ventricular Tachycardia". Perm J. 15 (1): 65. doi:10.7812/tpp/10-130. PMC 3048638. PMID 21505622.

CME Category::Cardiology

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