Search by property

Jump to navigation Jump to search

This page provides a simple browsing interface for finding entities described by a property and a named value. Other available search interfaces include the page property search, and the ask query builder.

Search by property

A list of all pages that have property "AnswerCExp" with value "Hypophosphatemic rickets is an X-linked dominant disorder.". Since there have been only a few results, also nearby values are displayed.

Showing below up to 26 results starting with #1.

View (previous 50 | next 50) (20 | 50 | 100 | 250 | 500)


    

List of results

  • WBR0785  + (HUS is not associated with rouleaux formation on peripheral smear. Schistocytes are classically found in patients with HUS.)
  • WBR0204  + (Hairy leukoplakia is caused by Epstein-Barr virus (EBV) and is most commonly seen in HIV-infected patients.)
  • WBR1131  + (Haloperidol has no role in the treatment of convulsion.)
  • WBR0440  + (Heart failure is not directly associated with TS or hypertension.)
  • WBR0674  + (Heart, kidney, and spleen undergo white (pale or anemic) infarction.)
  • WBR0893  + (Helical CT angiography (CTA) provides a rapid evaluation of intracranial and extracranial vessels in acute, subacute, and chronic stroke settings. It is used to determine the presence of vessel occlusions or stenoses.)
  • WBR0331  + (Helicase unwinds DNA templates at the replication fork.)
  • WBR0394  + (Helminth infection in the lungs is not associated with Curschmann's spirals. Eosinophils are usually abundant on BAL.)
  • WBR0803  + (Hemangiosarcoma is a rare, rapidly growingHemangiosarcoma is a rare, rapidly growing, highly invasive variety of cancer. It is a blood-fed sarcoma; that is, blood vessels grow directly into the tumor and it is typically filled with blood. A frequent cause of death is the rupturing of this tumor, causing the victim to rapidly bleed to death.sing the victim to rapidly bleed to death.)
  • WBR0176  + (Hemorrhoids may be observed among patients with portal hypertension due to back-up of blood flow in the portal circulation.)
  • WBR0013  + (Hemosiderin-laden macrophages in BAL may bHemosiderin-laden macrophages in BAL may be helpful in the diagnosis of diffuse pulmonary hemorrhage syndromes and diffuse interstitial pulmonary diseases (DIPD). They are also known as "heart failure cells since they may be observed in patients with congestive heart failure.in patients with congestive heart failure.)
  • WBR0235  + (Heparin activates antithrombin III. Heparin is indicated for thromboembolic diseases, which are less likely than Lyme disease in a young patient who has recently returned from a camping trip.)
  • WBR0349  + (Heparin causes activation of antithrombin III. Rat poison works by a different mechanism.)
  • WBR0671  + (Hepatic cirrhosis is characterized by the Hepatic cirrhosis is characterized by the presence of diffuse fibrosis and nodular regeneration in the liver. In advanced cirrhosis, patients have a shrunken liver. Cirrhosis is the final result of primary biliary cirrhosis, but is not the primary disease in this patient.s not the primary disease in this patient.)
  • WBR252  + (Hepatic stellate cells, also known as eithHepatic stellate cells, also known as either perisinusoidal cells or Ito cells (earlier lipocytes or fat-storing cells), are pericytes found in the perisinusoidal space of the liver also known as the space of Disse. Following ingestion, vitamin A, a lipid soluble vitamin, undergoes hepatic metabolism, whereby hepatic parenchymal cells absorb vitamin A in the form of retinyl esters. Parenchymal cells metabolize the retinyl esters, which is subsequently transferred to the stellate cells for either storage with other long-chain fatty acids or mobilization into the plasma (bound to RBP).bilization into the plasma (bound to RBP).)
  • WBR0882  + (Hepatitis B surface antigen and hepatitic C serologies should be considered in all patients with PAN due to the association between hepatitis and the development of PAN.)
  • WBR0772  + (Hereditary spherocytosis is an autosomal dominant hereditary disorder characterized by mutated cytoskeletal components that render the red blood cell membrane susceptible to osmotic fragility.)
  • WBR0591  + (Hirschsprung's disease is characterized by the lack of ganglion cells of the enteric nervous plexus.)
  • WBR0989  + (Homocystinuria is associated with lens subluxation that is downwards and inwards.)
  • WBR0293  + (Huntington's disease, hereditary hemorrhagic telangiectasia, Marfan syndrome, neurofibromatosis types 1 and 2, tuberous sclerosis, and von-Hippel Lindau are all examples of autosomal dominant disorders.)
  • WBR0327  + (Hydroxyurea inhibits ribonucleotide reductase, which is not beneficial to patients with SCD.)
  • WBR0681  + (Hyper-IgE syndrome is characterized by the failure of Th1 cells to produce IFN-gamma.)
  • WBR0637  + (Hyperphosphorylation of the Rb protein renders it inactive and allows the progression into neoplasia as observed in Plate A.)
  • WBR0494  + (Hypersensitivity type III disorders include conditions where an antigen-antibody complex forms and activates the complement system. A PPD skin test is not a type III hypersensitivity immune reaction.)
  • WBR0493  + (Hypersensitivity type III disorders include conditions where an antigen-antibody complex forms and activates the complement system. Hashimoto's disease is not a type III hypersensitivity disorder.)
  • WBR0244  + (Hypophosphatemic rickets is an X-linked dominant disorder.)
  • WBR0563  + (Hypoplasia is defined as the presence of primordial tissue and incomplete tissue development.)
  • WBR0680  + (IL-4 induces differentiation into Th2 cells and growth of B cells. It also enhances class switching into IgE)
  • WBR0773  + (IL-4 is not a main mediator of cachexia syndrome. IL-4 normally functions to induce the differentiation of naive T-helper cells in lymph nodes.)
  • WBR0402  + (Identification is an immature defense mechanism in which the individual models his/her behavior after a figure of authority.)
  • WBR0256  + (Idiopathic thrombocytopenic purpura (ITP) Idiopathic thrombocytopenic purpura (ITP) is an acquired bleeding disorder caused by decreased production and increased destruction of platelets due to formation of auto-antibodies against GpIIb/IIIa. ITP is associated with several environmental exposures, including hematological malignancies, HIV infection, HCV infection, and lupus erythematosus. The long history of bleeding and normal platelet counts make ITP a less likely diagnosis in this patient.P a less likely diagnosis in this patient.)
  • WBR0081  + (If the infection occurs within 13–26 weeks after conception there is a 23% chance the infant will be affected by the disease. This is not the trimester during which the patient is at highest risk.)
  • WBR0872  + (IgE deposits are not seen in skin biopsies of patients with Henoch-Schönlein purpura.)
  • WBR0295  + (In inflammation, redness and warmth are a result of histamine-mediated vasodilation of arterioles.)
  • WBR0352  + (In patients on statins liver enzymes Aspartate and Alanine transaminases should be monitored regularly as statins cause elevation in the levels)
  • WBR235  + (In the classical muscle contraction pathwaIn the classical muscle contraction pathway, stimuli (e.g. histamine) initially bind to Gq protein-coupled receptors located on the surface of vascular smooth muscle cells. The binding process activates phospholipase C, which mediates the synthesis of both 1,4,5-triphosphate (IP3) and diacylglycerol (DAG). IP3 then binds to receptors on the sarcoplasmic reticulum to mediate the mobilization of stored calcium into the cytosol. As cytosolic calcium concentration increases, calcium/calmodulin complexes form and activate MLCK, which result in the phosphorylation of myosin light chain and smooth muscle contraction.light chain and smooth muscle contraction.)
  • WBR0336  + (In the neonatal period, Sertoli cells lack any androgen receptors. Thus, AMH is not inhibited by testosterone in the neonatal period. However, intratesticular testosterone inhibits AMH in the pubertal period.)
  • WBR1003  + (In toxic shock syndrome, platelet counts are decreased rather than being increased.)
  • WBR0883  + (Incorrect)
  • WBR0710  + (Incorrect CNS lymphoma are common with AIDS, but associated with ring-enhancing lesions and show a mass effect on MRI)
  • WBR1064  + (Incorrect According to AHA recommendationsIncorrect</br>According to AHA recommendations, a combination of gram positive and gram negative antibiotics (intravenous penicillin G sodium+ gentamicin sulfate), should be used in resistant viridans group streptococci. Ceftriaxone does not provide the gram negative selectivity that gentamicin offers.gative selectivity that gentamicin offers.)
  • WBR0994  + (Incorrect Amiodarone is the recommended first line antiarrhythmic drug to maintain the normal sinus rhythm after being controlled in ED first.)
  • WBR1026  + (Incorrect Amiodarone-induced hypothyroidism (AIH) needs to be treated with thyroid hormone replacement therapy)
  • WBR1027  + (Incorrect Amiodarone-induced thyrotoxicosis (AIT) is the cause in this patient.)
  • WBR0701  + (Incorrect Antibiotics should never be stopped, although corticosteroids are used sometimes to minimize the inflammatory response)
  • WBR0897  + (Incorrect Aspirin comes in the management of the case, but the diagnosis of CAD should be confirmed using stress test)
  • WBR1060  + (Incorrect Blood cultures have to be drawn first)
  • WBR1061  + (Incorrect Blood cultures have to be drawn first)
  • WBR0706  + (Incorrect Chest X-Ray is done only if there are respiratory symptoms)
  • WBR0851  + (Incorrect Continue the antibiotics without reaching a diagnosis is in appropriate, and can miss potential complications (e.g. abscess or gas formation) which may need further management)
  • WBR0898  + (Incorrect Coronary bypass is done when the result of the angiogram suggest the need to it.)