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15625_lores.jpg | This image depicts the soles of both feet of a syphilis patient revealing the presence of secondary syphilitic lesions consisting of erosive dermal regions of the toes, mainly involving the intertriginous spaces between the toes. Secondary syphilitic lesions consist of skin rashes and/or sores in the mouth, vagina, or anus (also called mucous membrane lesions) mark the secondary stage of symptoms. This stage usually starts with a rash on one or more areas of the body. Rashes associated with secondary syphilis can appear from the time when the primary sore is healing to several weeks after the sore has healed. The rash usually does not cause itching. This rash may appear as rough, red, or reddish brown spots both on the palms of the hands and/or the bottoms of the feet. However, this rash may look different on other parts of the body and can look like rashes caused by other diseases Large, raised, gray or white lesions may develop in warm, moist areas such as the mouth, underarm or groin region. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. Other symptoms of secondary syphilis include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The symptoms of secondary syphilis will go away with or without treatment. Without appropriate treatment, the infection will progress to the latent and possibly late stages of disease. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
15625_lores.jpg | This image depicts the soles of both feet of a syphilis patient revealing the presence of secondary syphilitic lesions consisting of erosive dermal regions of the toes, mainly involving the intertriginous spaces between the toes. Secondary syphilitic lesions consist of skin rashes and/or sores in the mouth, vagina, or anus (also called mucous membrane lesions) mark the secondary stage of symptoms. This stage usually starts with a rash on one or more areas of the body. Rashes associated with secondary syphilis can appear from the time when the primary sore is healing to several weeks after the sore has healed. The rash usually does not cause itching. This rash may appear as rough, red, or reddish brown spots both on the palms of the hands and/or the bottoms of the feet. However, this rash may look different on other parts of the body and can look like rashes caused by other diseases Large, raised, gray or white lesions may develop in warm, moist areas such as the mouth, underarm or groin region. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. Other symptoms of secondary syphilis include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The symptoms of secondary syphilis will go away with or without treatment. Without appropriate treatment, the infection will progress to the latent and possibly late stages of disease. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
15566_lores.jpg | This image depicted the umbilicus of an infant, which displayed an inflamed lesion that under a darkfield examination revealed the presence of Treponema pallidum spirochetes, and hence, a diagnosis of congenital syphilis. Syphilis is a sexually transmitted disease (STD) caused by the bacterium ''Treponema pallidum''. It has often been called "the great imitator" because so many of the signs and symptoms are indistinguishable from those of other diseases. It is passed from person to person through direct contact with a syphilis sore. Sores occur mainly on the external genitals, vagina, anus, or in the rectum. Sores also can occur on the lips and in the mouth. Transmission of the organism occurs during vaginal, anal, or oral sex. Pregnant women with the disease can pass it to the babies they are carrying. Syphilis cannot be spread through contact with toilet seats, doorknobs, swimming pools, hot tubs, bathtubs, shared clothing, or eating utensils.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
15566_lores.jpg | This image depicted the umbilicus of an infant, which displayed an inflamed lesion that under a darkfield examination revealed the presence of ''Treponema pallidum'' spirochetes, and hence, a diagnosis of congenital syphilis. Syphilis is a sexually transmitted disease (STD) caused by the bacterium ''Treponema pallidum''. It has often been called "the great imitator" because so many of the signs and symptoms are indistinguishable from those of other diseases. It is passed from person to person through direct contact with a syphilis sore. Sores occur mainly on the external genitals, vagina, anus, or in the rectum. Sores also can occur on the lips and in the mouth. Transmission of the organism occurs during vaginal, anal, or oral sex. Pregnant women with the disease can pass it to the babies they are carrying. Syphilis cannot be spread through contact with toilet seats, doorknobs, swimming pools, hot tubs, bathtubs, shared clothing, or eating utensils.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
15022_lores.jpg | With a VDRL (Venereal Disease Research Laboratory) titer of 1:128, this syphilis patient displayed symptoms indicative of the onset of the secondary stage of this disease, which included generalized lymphadenopathy, and accompanying lingual (tongue) mucous patches. Syphilis is a sexually transmitted disease (STD) caused by the bacterium ''Treponema pallidum''. It has often been called "the great imitator" because so many of the signs and symptoms are indistinguishable from those of other diseases. It is passed from person to person through direct contact with a syphilis sore. Sores occur mainly on the external genitals, vagina, anus, or in the rectum. Sores also can occur on the lips and in the mouth. Transmission of the organism occurs during vaginal, anal, or oral sex. Pregnant women with the disease can pass it to the babies they are carrying. Syphilis cannot be spread through contact with toilet seats, doorknobs, swimming pools, hot tubs, bathtubs, shared clothing, or eating utensils.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
15022_lores.jpg | With a VDRL (Venereal Disease Research Laboratory) titer of 1:128, this syphilis patient displayed symptoms indicative of the onset of the secondary stage of this disease, which included generalized lymphadenopathy, and accompanying lingual (tongue) mucous patches. Syphilis is a sexually transmitted disease (STD) caused by the bacterium ''Treponema pallidum''. It has often been called "the great imitator" because so many of the signs and symptoms are indistinguishable from those of other diseases. It is passed from person to person through direct contact with a syphilis sore. Sores occur mainly on the external genitals, vagina, anus, or in the rectum. Sores also can occur on the lips and in the mouth. Transmission of the organism occurs during vaginal, anal, or oral sex. Pregnant women with the disease can pass it to the babies they are carrying. Syphilis cannot be spread through contact with toilet seats, doorknobs, swimming pools, hot tubs, bathtubs, shared clothing, or eating utensils.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12627_lores.jpg | This image depicts the dorsal surface of the tongue in the case of an elderly African-American male, due to what was determined to be a secondary syphilitic infection. Note the furrowed appearance, and the papillae-free, i.e., desquamated, smooth lingual surface. Rashes associated with secondary syphilis can appear as the chancre is healing or several weeks after the chancre has healed. The characteristic rash of secondary syphilis may appear as rough, red, or reddish brown spots both on the palms of the hands and the bottoms of the feet. However, rashes with a different appearance may occur on other parts of the body, sometimes resembling rashes caused by other diseases. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. In addition to rashes, symptoms of secondary syphilis may include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The signs and symptoms of secondary syphilis will resolve with or without treatment, but without treatment, the infection will progress to the latent and possibly late stages of disease. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12627_lores.jpg | This image depicts the dorsal surface of the tongue in the case of an elderly African-American male, due to what was determined to be a secondary syphilitic infection. Note the furrowed appearance, and the papillae-free, i.e., desquamated, smooth lingual surface. Rashes associated with secondary syphilis can appear as the chancre is healing or several weeks after the chancre has healed. The characteristic rash of secondary syphilis may appear as rough, red, or reddish brown spots both on the palms of the hands and the bottoms of the feet. However, rashes with a different appearance may occur on other parts of the body, sometimes resembling rashes caused by other diseases. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. In addition to rashes, symptoms of secondary syphilis may include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The signs and symptoms of secondary syphilis will resolve with or without treatment, but without treatment, the infection will progress to the latent and possibly late stages of disease. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12626_lores.jpg | This close-up view demonstrates the interior oral cavity of an elderly African-American male patient, revealing a perforated hard palate due to what was a congenital syphilis infection. At the time of this photograph, the patient was being treated for both active syphilis, and gonorrhea infections. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12626_lores.jpg | This close-up view demonstrates the interior oral cavity of an elderly African-American male patient, revealing a perforated hard palate due to what was a congenital syphilis infection. At the time of this photograph, the patient was being treated for both active syphilis, and gonorrhea infections. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12625_lores.jpg | This close-up view demonstrates the dentition within the oral cavity of a young African-American female patient, revealing the triangular-shaped deformity of her right lateral incisor, and the left central incisor, which is known as Hutchinson incisors, and is caused by a congenital syphilitic infection. In this particular case, at the time of her birth, one of this woman’s parents tested positive for syphilis. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12625_lores.jpg | This close-up view demonstrates the dentition within the oral cavity of a young African-American female patient, revealing the triangular-shaped deformity of her right lateral incisor, and the left central incisor, which is known as Hutchinson incisors, and is caused by a congenital syphilitic infection. In this particular case, at the time of her birth, one of this woman’s parents tested positive for syphilis. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12624_lores.jpg | This image depicts a close view of the right corner, i.e., angle, of the mouth of an African-American female, upon which one can see a circular lesion that was diagnosed as a primary syphilitic chancre. The primary stage of syphilis is usually marked by the appearance of a single sore (called a chancre), but there may be multiple sores. The time between infection with syphilis and the start of the first symptom can range from 10 to 90 days (average 21 days). The chancre is usually firm, round, small, and painless. It appears at the spot where syphilis entered the body. The chancre lasts 3 to 6 weeks, and it heals without treatment. However, if adequate treatment is not administered, the infection progresses to the secondary stage.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12624_lores.jpg | This image depicts a close view of the right corner, i.e., angle, of the mouth of an African-American female, upon which one can see a circular lesion that was diagnosed as a primary syphilitic chancre. The primary stage of syphilis is usually marked by the appearance of a single sore (called a chancre), but there may be multiple sores. The time between infection with syphilis and the start of the first symptom can range from 10 to 90 days (average 21 days). The chancre is usually firm, round, small, and painless. It appears at the spot where syphilis entered the body. The chancre lasts 3 to 6 weeks, and it heals without treatment. However, if adequate treatment is not administered, the infection progresses to the secondary stage.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12623_lores.jpg | This image depicts a close view of the surface of an African-American female’s tongue, upon which one can see a circular lesion that was diagnosed as a primary syphilitic chancre. The primary stage of syphilis is usually marked by the appearance of a single sore (called a chancre), but there may be multiple sores. The time between infection with syphilis and the start of the first symptom can range from 10 to 90 days (average 21 days). The chancre is usually firm, round, small, and painless. It appears at the spot where syphilis entered the body. The chancre lasts 3 to 6 weeks, and it heals without treatment. However, if adequate treatment is not administered, the infection progresses to the secondary stage.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12623_lores.jpg | This image depicts a close view of the surface of an African-American female’s tongue, upon which one can see a circular lesion that was diagnosed as a primary syphilitic chancre. The primary stage of syphilis is usually marked by the appearance of a single sore (called a chancre), but there may be multiple sores. The time between infection with syphilis and the start of the first symptom can range from 10 to 90 days (average 21 days). The chancre is usually firm, round, small, and painless. It appears at the spot where syphilis entered the body. The chancre lasts 3 to 6 weeks, and it heals without treatment. However, if adequate treatment is not administered, the infection progresses to the secondary stage.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12605_lores.jpg | This photograph depicts the destruction of a patient’s left knee joint, which was determined to be a case of neuropathic arthropathy, also known as Charcot’s joint, brought on by a tertiary syphilitic infection. See PHIL 12606, for a radiographic view (x-ray) of a patient's knee with this arthritic deformity. Late and Latent Stages: The latent (hidden) stage of syphilis begins when primary and secondary symptoms disappear. Without treatment, the infected person will continue to have syphilis even though there are no signs or symptoms; infection remains in the body. This latent stage can last for years. The late stages of syphilis can develop in about 15% of people who have not been treated for syphilis, and can appear 10-20 years after infection was first acquired. In the late stages of syphilis, the disease may subsequently damage the internal organs, including the brain, nerves, eyes, heart, blood vessels, liver, bones, and joints. Signs and symptoms of the late stage of syphilis include difficulty coordinating muscle movements, paralysis, numbness, gradual blindness, and dementia. This damage may be serious enough to cause death.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12605_lores.jpg | This photograph depicts the destruction of a patient’s left knee joint, which was determined to be a case of neuropathic arthropathy, also known as Charcot’s joint, brought on by a tertiary syphilitic infection. See PHIL 12606, for a radiographic view (x-ray) of a patient's knee with this arthritic deformity. Late and Latent Stages: The latent (hidden) stage of syphilis begins when primary and secondary symptoms disappear. Without treatment, the infected person will continue to have syphilis even though there are no signs or symptoms; infection remains in the body. This latent stage can last for years. The late stages of syphilis can develop in about 15% of people who have not been treated for syphilis, and can appear 10-20 years after infection was first acquired. In the late stages of syphilis, the disease may subsequently damage the internal organs, including the brain, nerves, eyes, heart, blood vessels, liver, bones, and joints. Signs and symptoms of the late stage of syphilis include difficulty coordinating muscle movements, paralysis, numbness, gradual blindness, and dementia. This damage may be serious enough to cause death.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12604_lores.jpg | This photograph depicts a patient’s face revealing pathologic cutaneous changes in the region around the nose and mouth, consisting of noduloulcerative lesions, known as syphilids, due to a syphilitic infection. The secondary stage of syphilis is characterized by the manifestation of a skin rash and mucous membrane lesions. This stage typically starts with the development of a rash on one or more areas of the body. The rash usually does not cause itching. Rashes associated with secondary syphilis can appear as the chancre is healing or several weeks after the chancre has healed. The characteristic rash of secondary syphilis may appear as rough, red, or reddish brown spots both on the palms of the hands and the bottoms of the feet. However, rashes with a different appearance may occur on other parts of the body, sometimes resembling rashes caused by other diseases. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. In addition to rashes, symptoms of secondary syphilis may include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The signs and symptoms of secondary syphilis will resolve with or without treatment, but without treatment, the infection will progress to the latent and possibly late stages of disease.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12604_lores.jpg | This photograph depicts a patient’s face revealing pathologic cutaneous changes in the region around the nose and mouth, consisting of noduloulcerative lesions, known as syphilids, due to a syphilitic infection. The secondary stage of syphilis is characterized by the manifestation of a skin rash and mucous membrane lesions. This stage typically starts with the development of a rash on one or more areas of the body. The rash usually does not cause itching. Rashes associated with secondary syphilis can appear as the chancre is healing or several weeks after the chancre has healed. The characteristic rash of secondary syphilis may appear as rough, red, or reddish brown spots both on the palms of the hands and the bottoms of the feet. However, rashes with a different appearance may occur on other parts of the body, sometimes resembling rashes caused by other diseases. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. In addition to rashes, symptoms of secondary syphilis may include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The signs and symptoms of secondary syphilis will resolve with or without treatment, but without treatment, the infection will progress to the latent and possibly late stages of disease.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12603_lores.jpg | This photograph depicts the a patient’s opened mouth revealing pathologic changes in the superior mucosal surface of his tongue known as syphilitic glossitis, due to a congenital syphilitic infection. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12603_lores.jpg | This photograph depicts the a patient’s opened mouth revealing pathologic changes in the superior mucosal surface of his tongue known as syphilitic glossitis, due to a congenital syphilitic infection. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12602_lores.jpg | This photograph depicts the wrinkled skin around a patient’s nose and mouth known as “rhagades”, due to a congenital syphilitic infection. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12602_lores.jpg | This photograph depicts the wrinkled skin around a patient’s nose and mouth known as “rhagades”, due to a congenital syphilitic infection. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12601_lores.jpg | This image depicts an inferior, intraoral view of a patient’s hard palate revealing the pathologic changes in palatal anatomy, which resulted in a perforation into the nasal cavity, and was due to a congenital syphilitic infection. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12601_lores.jpg | This image depicts an inferior, intraoral view of a patient’s hard palate revealing the pathologic changes in palatal anatomy, which resulted in a perforation into the nasal cavity, and was due to a congenital syphilitic infection. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12600_lores.jpg | This image depicts a close view of the right eye of a patient revealing the pathologic changes in her cornea known as interstitial corneal keratitis, which was due to a congenital syphilitic infection, and is a chronic progressive keratitis of the corneal stroma, i.e., connective tissue matrix, often resulting in blindness and frequently associated with congenital syphilis. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12600_lores.jpg | This image depicts a close view of the right eye of a patient revealing the pathologic changes in her cornea known as interstitial corneal keratitis, which was due to a congenital syphilitic infection, and is a chronic progressive keratitis of the corneal stroma, i.e., connective tissue matrix, often resulting in blindness and frequently associated with congenital syphilis. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12599_lores.jpg | This image depicts the dentition of a congenital syphilis patient, who due to this disease, went on to develop what are known as Hutchinson’s teeth, in which case the teeth are widely spaced, and the bite surfaces of the incisors are notched. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12599_lores.jpg | This image depicts the dentition of a congenital syphilis patient, who due to this disease, went on to develop what are known as Hutchinson’s teeth, in which case the teeth are widely spaced, and the bite surfaces of the incisors are notched. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12598_lores.jpg | This photograph depicts a lateral view of a patient's right knee, who'd been diagnosed with "Clutton’s joints" due to what was determined to be congenital syphilis. See PHIL 4102, for a view of the patient's knees, from an anterior persoective. ”Clutton's joints”, or symmetrical hydrarthrosis of the knee joints, is a painless condition that often occurs during the late stages of congenital syphilis. It involes synovitis, or swelling of a joint, accompanied by collections of fluid within the joint capsule.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12598_lores.jpg | This photograph depicts a lateral view of a patient's right knee, who'd been diagnosed with "Clutton’s joints" due to what was determined to be congenital syphilis. See PHIL 4102, for a view of the patient's knees, from an anterior persoective. ”Clutton's joints”, or symmetrical hydrarthrosis of the knee joints, is a painless condition that often occurs during the late stages of congenital syphilis. It involes synovitis, or swelling of a joint, accompanied by collections of fluid within the joint capsule.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12597_lores.jpg | This image depicts the perineal region and upper thighs of an infant born with what was diagnosed as congenital syphilis. In this particular case, one will note the presence of early cutaneous syphilids. How does syphilis affect a pregnant woman and her baby: The syphilis bacterium can infect the baby of a woman during her pregnancy. Depending on how long a pregnant woman has been infected, she may have a high risk of having a stillbirth (a baby born dead) or of giving birth to a baby who dies shortly after birth. An infected baby may be born without signs or symptoms of disease. However, if not treated immediately, the baby may develop serious problems within a few weeks. Untreated babies may become developmentally delayed, have seizures, or die. Infants born to mothers who have reactive nontreponemal and treponemal test results should be evaluated with a quantitative nontreponemal serologic test (RPR or VDRL) performed on infant serum because umbilical cord blood can become contaminated with maternal blood and could yield a false-positive result. Conducting a treponemal test (i.e., TP-PA or FTA-ABS) on a newborn’s serum is not necessary. No commercially available immunoglobulin (IgM) test can be recommended. All infants born to women who have reactive serologic tests for syphilis should be examined thoroughly for evidence of congenital syphilis (e.g., nonimmune hydrops, jaundice, hepatosplenomegaly, rhinitis, skin rash, and/or pseudoparalysis of an extremity). Pathologic examination of the umbilical cord by using specific fluorescent antitreponemal antibody staining is suggested. Darkfield microscopic examination or DFA staining of suspicious lesions or body fluids (e.g., nasal discharge) also should be performed.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
12597_lores.jpg | This image depicts the perineal region and upper thighs of an infant born with what was diagnosed as congenital syphilis. In this particular case, one will note the presence of early cutaneous syphilids. How does syphilis affect a pregnant woman and her baby: The syphilis bacterium can infect the baby of a woman during her pregnancy. Depending on how long a pregnant woman has been infected, she may have a high risk of having a stillbirth (a baby born dead) or of giving birth to a baby who dies shortly after birth. An infected baby may be born without signs or symptoms of disease. However, if not treated immediately, the baby may develop serious problems within a few weeks. Untreated babies may become developmentally delayed, have seizures, or die. Infants born to mothers who have reactive nontreponemal and treponemal test results should be evaluated with a quantitative nontreponemal serologic test (RPR or VDRL) performed on infant serum because umbilical cord blood can become contaminated with maternal blood and could yield a false-positive result. Conducting a treponemal test (i.e., TP-PA or FTA-ABS) on a newborn’s serum is not necessary. No commercially available immunoglobulin (IgM) test can be recommended. All infants born to women who have reactive serologic tests for syphilis should be examined thoroughly for evidence of congenital syphilis (e.g., nonimmune hydrops, jaundice, hepatosplenomegaly, rhinitis, skin rash, and/or pseudoparalysis of an extremity). Pathologic examination of the umbilical cord by using specific fluorescent antitreponemal antibody staining is suggested. Darkfield microscopic examination or DFA staining of suspicious lesions or body fluids (e.g., nasal discharge) also should be performed.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
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<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5341_lores.jpg | This patient presented with a Moon’s Molar or Mulberry Molar due to a congenital syphilitic infection. Moon's- or Mulberry Molar is a condition where the first lower molar tooth has become dome-shaped due to malformation by congenital syphilis.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5341_lores.jpg | This patient presented with a Moon’s Molar or Mulberry Molar due to a congenital syphilitic infection. Moon's- or Mulberry Molar is a condition where the first lower molar tooth has become dome-shaped due to malformation by congenital syphilis.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5339_lores.jpg | This patient presented with a primary vulvar syphilitic chancre due to ''Treponema pallidum'' bacteria. The primary stage of syphilis is usually marked by the appearance of a single sore known as a chancre, but there may be multiple sores. The chancre is usually firm, round, small, and painless.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5339_lores.jpg | This patient presented with an anal chancre due to ''Treponema pallidum'' bacteria. The primary stage of syphilis is usually marked by the appearance of a single sore known as a chancre, but there may be multiple sores. The chancre is usually firm, round, small, and painless.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5339_lores.jpg | This patient presented with an anal chancre due to ''Treponema pallidum'' bacteria. The primary stage of syphilis is usually marked by the appearance of a single sore known as a chancre, but there may be multiple sores. The chancre is usually firm, round, small, and painless.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5338_lores.jpg | This patient presented with a case of syphilis during the secondary stage with the appearance of a papular forearm lesion. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment the rash clears up on its own. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5338_lores.jpg | This patient presented with a case of syphilis during the secondary stage with the appearance of a papular forearm lesion. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment the rash clears up on its own. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
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5336_lores.jpg | This patient presented with a case of alopecia during the secondary stage of syphilis. Second-stage symptoms can include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and tiredness. The disease can easily be passed to sex partners during the primary or secondary stages.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5336_lores.jpg | This patient presented with a case of alopecia during the secondary stage of syphilis. Second-stage symptoms can include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and tiredness. The disease can easily be passed to sex partners during the primary or secondary stages.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5334_lores.jpg | This patient presented with a case of alopecia due to what was determined to be secondary syphilis. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment the rash clears up on its own.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5334_lores.jpg | This patient presented with a case of alopecia due to what was determined to be secondary syphilis. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment the rash clears up on its own.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5330_lores.jpg | This patient presented with a gumma of nose due to a long standing tertiary syphilitic Treponema palliduminfection. Without treatment, an infected person still has syphilis even though there are no signs or symptoms. It remains in the body, and it may begin to damage the internal organs, including the brain, nerves, eyes, heart, blood vessels, liver, bones, and joints.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5330_lores.jpg | This patient presented with a gumma of nose due to a long standing tertiary syphilitic ''Treponema pallidum'' infection. Without treatment, an infected person still has syphilis even though there are no signs or symptoms. It remains in the body, and it may begin to damage the internal organs, including the brain, nerves, eyes, heart, blood vessels, liver, bones, and joints.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5328_lores.jpg | This 16 year old patient presented with a "saddle nose" deformity due to a congenital syphilitic condition. See PHIL 17626, for a color version of this image. The presence of the ''Treponema pallidum'' bacterium detrimentally affects the normal cytoarchitectural development of the soft, boney precursor tissues such as cartilage, giving rise to boney malformations like this “saddle nose” deformity.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
5328_lores.jpg | This 16 year old patient presented with a "saddle nose" deformity due to a congenital syphilitic condition. See PHIL 17626, for a color version of this image. The presence of the ''Treponema pallidum'' bacterium detrimentally affects the normal cytoarchitectural development of the soft, boney precursor tissues such as cartilage, giving rise to boney malformations like this “saddle nose” deformity.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
4816_lores.jpg | This image depicts a lingual mucous patch on the tongue of a patient who was subsequently diagnosed with secondary syphilis, due to the ''Treponema pallidum'' bacterium. Secondary syphilis is the most contagious of all the stages of this disease, and is characterized by a systemic spread of the ''Treponema pallidum'' bacterial spirochetes. Skin rash and mucous membrane lesions characterize the secondary stage. The characteristic rash of secondary syphilis may appear as rough, red, or reddish brown spots both on the palms of the hands and the bottoms of the feet. However, rashes with a different appearance may occur on other parts of the body, sometimes resembling rashes caused by other diseases. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. In addition to rashes, symptoms of secondary syphilis may include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The signs and symptoms of secondary syphilis will resolve with or without treatment, but without treatment, the infection will progress to the latent and late stages of disease.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
4816_lores.jpg | This image depicts a lingual mucous patch on the tongue of a patient who was subsequently diagnosed with secondary syphilis, due to the ''Treponema pallidum'' bacterium. Secondary syphilis is the most contagious of all the stages of this disease, and is characterized by a systemic spread of the ''Treponema pallidum'' bacterial spirochetes. Skin rash and mucous membrane lesions characterize the secondary stage. The characteristic rash of secondary syphilis may appear as rough, red, or reddish brown spots both on the palms of the hands and the bottoms of the feet. However, rashes with a different appearance may occur on other parts of the body, sometimes resembling rashes caused by other diseases. Sometimes rashes associated with secondary syphilis are so faint that they are not noticed. In addition to rashes, symptoms of secondary syphilis may include fever, swollen lymph glands, sore throat, patchy hair loss, headaches, weight loss, muscle aches, and fatigue. The signs and symptoms of secondary syphilis will resolve with or without treatment, but without treatment, the infection will progress to the latent and late stages of disease.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
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PHIL_3482_lores.jpg | This patient presented with a primary syphilitic chancre in the right inguinal region. The primary stage of syphilis is usually marked by the appearance of a single sore known as a chancre, but there may be multiple sores. The chancre is usually firm, round, small, and painless. These serpiginous, nodular ulcerative lesions are due to late syphilitic disease. The signs and symptoms of late stage syphilis include not being able to coordinate muscle movements, paralysis, numbness, gradual blindness and dementia. This damage may be serious enough to cause death.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3482_lores.jpg | This patient presented with a primary syphilitic chancre in the right inguinal region. The primary stage of syphilis is usually marked by the appearance of a single sore known as a chancre, but there may be multiple sores. The chancre is usually firm, round, small, and painless. These serpiginous, nodular ulcerative lesions are due to late syphilitic disease. The signs and symptoms of late stage syphilis include not being able to coordinate muscle movements, paralysis, numbness, gradual blindness and dementia. This damage may be serious enough to cause death.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3481_lores.jpg | This patient presented with a papulo-squamous rash on the sole of the foot due to secondary syphilis. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment, rashes clear up on their own.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3481_lores.jpg | This patient presented with a papulo-squamous rash on the sole of the foot due to secondary syphilis. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment, rashes clear up on their own.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3480_lores.jpg | This patient presented with a papular rash on the sole of the foot due to secondary syphilis. The second stage of syphilis starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of hands and on the bottoms of feet. Even without treatment, rashes clear up by itself.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3479_lores.jpg | This patient presented with phimosis and inguinal lymphadenopathy due to primary syphilis. One of the symptoms of primary syphilis is the presence of lymphadenopathy, i.e. the swelling of the inguinal lymph nodes, either bilaterally or unilaterally, as well as the presence of a primary chancre.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3479_lores.jpg | This patient presented with phimosis and inguinal lymphadenopathy due to primary syphilis. One of the symptoms of primary syphilis is the presence of lymphadenopathy, i.e. the swelling of the inguinal lymph nodes, either bilaterally or unilaterally, as well as the presence of a primary chancre.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3478_lores.jpg | This patient presented with a secondary syphilitic maculopapular rash of the right hand and forearm. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment, rashes clear up on their own.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3478_lores.jpg | This patient presented with a secondary syphilitic maculopapular rash of the right hand and forearm. The second stage starts when one or more areas of the skin break into a rash that appears as rough, red or reddish brown spots both on the palms of the hands and on the bottoms of the feet. Even without treatment, rashes clear up on their own.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
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PHIL_2389_lores.jpg | Photograph of rhagades resulting from congenital syphilis. This patient with congenital syphilis is exhibiting rhagades, which are cracks or fissures in the skin around the mouth. Such a rare type of facial disfigurement, results from persistent infantile syphilitic rhinitis. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2389_lores.jpg | Photograph of rhagades resulting from congenital syphilis. This patient with congenital syphilis is exhibiting rhagades, which are cracks or fissures in the skin around the mouth. Such a rare type of facial disfigurement, results from persistent infantile syphilitic rhinitis. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2388_lores.jpg | This photograph depicts a perforated hard palate on a patient with congenital syphilis. This patient with congenital syphilis has developed a perforation of hard palate due to gummatous destruction. These destructive tumors can also attack the skin, long bones, eyes, mucous membranes, throat, liver, or stomach lining.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2388_lores.jpg | This photograph depicts a perforated hard palate on a patient with congenital syphilis. This patient with congenital syphilis has developed a perforation of hard palate due to gummatous destruction. These destructive tumors can also attack the skin, long bones, eyes, mucous membranes, throat, liver, or stomach lining.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
2386_lores.jpg | This image depicts the dentition of a congenital syphilis patient, who due to this disease, went on to develop what are known as mulberry molars. “Moon's“, or mulberry molars, is a condition where the bite surface of the permanent first lower molar teeth develops rounded surfaces to its cusps, resembling the surface of a mulberry. Congenital syphilis, is a condition caused by infection in utero with Treponema pallidum. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
2386_lores.jpg | This image depicts the dentition of a congenital syphilis patient, who due to this disease, went on to develop what are known as mulberry molars. “Moon's", or mulberry molars, is a condition where the bite surface of the permanent first lower molar teeth develops rounded surfaces to its cusps, resembling the surface of a mulberry. Congenital syphilis, is a condition caused by infection in utero with ''Treponema pallidum''. A wide spectrum of severity exists, and only severe cases are clinically apparent at birth. An infant or child (aged less than 2 years) may have signs such as hepatosplenomegaly, rash, condyloma lata, snuffles, jaundice (nonviral hepatitis), pseudoparalysis, anemia, or edema (nephrotic syndrome and/or malnutrition). An older child may have stigmata (e.g., interstitial keratitis, nerve deafness, anterior bowing of shins, frontal bossing, mulberry molars, Hutchinson teeth, saddle nose, rhagades, or Clutton joints).<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2385_lores.jpg | A photograph of Hutchinson’s Teeth resulting from congenital syphilis. Hutchinson’s Teeth is a congenital anomaly in which the permanent incisor teeth are narrow and notched. Note the notched edges and "screwdriver" shape of the central incisors.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2385_lores.jpg | A photograph of Hutchinson’s Teeth resulting from congenital syphilis. Hutchinson’s Teeth is a congenital anomaly in which the permanent incisor teeth are narrow and notched. Note the notched edges and "screwdriver" shape of the central incisors.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2384_lores.jpg | A photograph of a patient with congenital syphilis exhibiting interstitial corneal keratitis. This patient’s congenital syphilitic disease resulted in the onset of interstitial keratitis, an inflammation of the connective tissue structure of the cornea. Syphilis is the most common cause for this condition.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2384_lores.jpg | A photograph of a patient with congenital syphilis exhibiting interstitial corneal keratitis. This patient’s congenital syphilitic disease resulted in the onset of interstitial keratitis, an inflammation of the connective tissue structure of the cornea. Syphilis is the most common cause for this condition.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
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PHIL_2382_lores.jpg | A photograph of a young child with congenital syphilis exhibiting intraoral mucous patches and facial skin lesions. An infant demonstrating mucous patches and skin lesions resulting from congenital syphilis. In 1998, 81.3% of reported cases of CS occurred because the mother received no penicillin treatment or inadequate treatment before or during pregnancy.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2382_lores.jpg | A photograph of a young child with congenital syphilis exhibiting intraoral mucous patches and facial skin lesions. An infant demonstrating mucous patches and skin lesions resulting from congenital syphilis. In 1998, 81.3% of reported cases of CS occurred because the mother received no penicillin treatment or inadequate treatment before or during pregnancy.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3481_lores.jpg | A photograph of a patient with tertiary syphilis resulting in gummatous lesions on the dorsal surface of the left hand. Gummatous lesions due to tertiary syphilis occur many years after initial untreated primary syphilis. The tumors are benign and if properly treated, the gummas will heal and the patient will recover in most cases.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3481_lores.jpg | A photograph of a patient with tertiary syphilis resulting in gummatous lesions on the dorsal surface of the left hand. Gummatous lesions due to tertiary syphilis occur many years after initial untreated primary syphilis. The tumors are benign and if properly treated, the gummas will heal and the patient will recover in most cases.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_3480_lores.jpg | A photograph of a patient with tertiary syphilis resulting in gummas seen here on the scalp. Tertiary syphilis occurs many years after initial untreated primary syphilis. Gummas, or internal tissue granulation, form and result in severe damage to the skin, bone, and liver.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2379_lores.jpg | A photograph of a patient with tertiary syphilis depicting gummatous lesions on the volar surface of the right arm. Tertiary syphilis occurs many years after initial untreated primary syphilis. Gummas, or internal tissue granulation, form and result in severe damage to the skin, bone, liver and other bodily organs, or regions.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2379_lores.jpg | A photograph of a patient with tertiary syphilis depicting gummatous lesions on the volar surface of the right arm. Tertiary syphilis occurs many years after initial untreated primary syphilis. Gummas, or internal tissue granulation, form and result in severe damage to the skin, bone, liver and other bodily organs, or regions.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2378_lores.jpg | The onset of this aortic aneurysm occurred during the tertiary stage of syphilis. This patient with late tertiary syphilis has developed an aortic aneurysm, which is eroding supraclavicularly through the ribs and clavicle. Cardiovascular syphilis can begin 5 to 10 years after initial infection.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2378_lores.jpg | The onset of this aortic aneurysm occurred during the tertiary stage of syphilis. This patient with late tertiary syphilis has developed an aortic aneurysm, which is eroding supraclavicularly through the ribs and clavicle. Cardiovascular syphilis can begin 5 to 10 years after initial infection.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
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  <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
  <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2357_lores.jpg | Patient with secondary syphilitic macular rash on the medial right foot. The rash often appears as rough, red or reddish brown spots, and can appear on both the palms of the hands as well as on the plantar surface (bottom) of the feet.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2357_lores.jpg | Patient with secondary syphilitic macular rash on the medial right foot. The rash often appears as rough, red or reddish brown spots, and can appear on both the palms of the hands as well as on the plantar surface (bottom) of the feet.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2353_lores.jpg | Chancres on the penile shaft due to a primary syphilitic infection caused by ''Treponema pallidum'' bacteria. The primary stage of syphilis is usually marked by the appearance of a single sore called a chancre. The chancre is usually firm, round, small, and painless.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2353_lores.jpg | A chancre of the posterior vaginal fourchette during the primary stage of syphilis. This chancre is located on the posterior vaginal fourchette (where labia minora meet). The primary stage of syphilis is often marked by the appearance of a single sore – called a chancre, which is usually firm, round, small, and painless.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2353_lores.jpg | A chancre of the posterior vaginal fourchette during the primary stage of syphilis. This chancre is located on the posterior vaginal fourchette (where labia minora meet). The primary stage of syphilis is often marked by the appearance of a single sore – called a chancre, which is usually firm, round, small, and painless.<br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2352_lores.jpg | A chancre on the penis due to primary syphilis. The primary stage of syphilis is usually marked by the appearance of a single sore called a chancre. The chancre is usually firm, round, small, and painless. This image shows a chancre located at the coronal sulcus of the penis. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
PHIL_2352_lores.jpg | A chancre on the penis due to primary syphilis. The primary stage of syphilis is usually marked by the appearance of a single sore called a chancre. The chancre is usually firm, round, small, and painless. This image shows a chancre located at the coronal sulcus of the penis. <br>[http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
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=====Amebiasis=====
=====Amebiasis=====
'''Amebiasis cysts'''
'''''Entamoeba'' sp. cysts'''
<gallery widths=200px>
<gallery widths=200px>


Ehistdisp_cyst_tric.jpg | Cyst of ''E. histolytica/E. dispar'' stained with trichrome. Note the chromatoid body with blunt ends (red arrow).
Ehistdisp_cyst_tric.jpg | Cyst of ''Entamoeba'' sp. stained with trichrome. Note the chromatoid body with blunt ends (red arrow).
  <br> [http://www.cdc.gov/dpdx/amebiasis/index.html <font size="-2">''Adapted from CDC''</font>]
  <br> [http://www.cdc.gov/dpdx/amebiasis/index.html <font size="-2">''Adapted from CDC''</font>]


</gallery>
</gallery>


'''Amebiasis trophozioites'''
'''''Entamoeba'' sp. trophozioites'''
<gallery widths=200px>


<gallery widths=200px>
Ehisto_troph_tric3.jpg | Trophozoites of ''E. histolytica'' with ingested erythrocytes stained with trichrome. The ingested erythrocytes appear as dark inclusions. The parasite above show nuclei that have the typical small, centrally located karyosome, and thin, uniform peripheral chromatin. <br> [http://www.cdc.gov/dpdx/amebiasis/index.html <font size="-2">''Adapted from CDC''</font>]
Ehisto_troph_tric3.jpg | Trophozoites of ''E. histolytic'' with ingested erythrocytes stained with trichrome. The ingested erythrocytes appear as dark inclusions. The parasite above show nuclei that have the typical small, centrally located karyosome, and thin, uniform peripheral chromatin. <br> [http://www.cdc.gov/dpdx/amebiasis/index.html <font size="-2">''Adapted from CDC''</font>]
Ehisto_trophs_he_colon.jpg | ''Entamoeba histolytica'' trophozoites in colon tissue stained with H&E. <br> [http://www.cdc.gov/dpdx/amebiasis/index.html <font size="-2">''Adapted from CDC''</font>]
Ehisto_trophs_he_colon.jpg | ''Entamoeba histolytica'' trophozoites in colon tissue stained with H&E. <br> [http://www.cdc.gov/dpdx/amebiasis/index.html <font size="-2">''Adapted from CDC''</font>]


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======''Pseudoterranova decipiens''======
======''Pseudoterranova decipiens''======
'''''Pseudoterranova'' sp. larval worms'''
'''''Pseudoterranova'' sp. larval worms'''
<gallery widths=200px>
<gallery widths=200px>


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</gallery>
</gallery>


'''Cross sections of ''Pseudoterranova'' sp. worms''' Cross sections of anisakid worms.
'''Cross sections of ''Pseudoterranova'' sp. worms'''
<gallery widths=200px>
<gallery widths=200px>


Pseudocontra_crosssection.jpg | Cross-section of Pseudoterranova sp. Note the large butterfly-shaped lateral chords (black arrows), characteristic for this genus. <br> [http://www.cdc.gov/dpdx/anisakiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Pseudocontra_crosssection.jpg | Cross-section of ''Pseudoterranova'' sp. Note the large butterfly-shaped lateral chords (black arrows), characteristic for this genus. <br> [http://www.cdc.gov/dpdx/anisakiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Codworm_10x_dic.jpg | Cross-section of Pseudoterranova sp. viewed under differential interference contrast (DIC) microscopy. <br> [http://www.cdc.gov/dpdx/anisakiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Codworm_10x_dic.jpg | Cross-section of ''Pseudoterranova'' sp. viewed under differential interference contrast (DIC) microscopy. <br> [http://www.cdc.gov/dpdx/anisakiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]


</gallery>
</gallery>
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Bmicroti_thin_giemsa.jpg | ''Babesia microti'' in a thin blood smear stained with Giemsa. ''Babesia'' sp. cannot be identified to the species level by morphology alone; additional testing, such as PCR, is always recommended. <br> [http://www.cdc.gov/dpdx/babesiosis/index.html <font size="-2">''Adapted from CDC''</font>]
Bmicroti_thin_giemsa.jpg | ''Babesia microti'' in a thin blood smear stained with Giemsa. ''Babesia'' sp. cannot be identified to the species level by morphology alone; additional testing, such as PCR, is always recommended. <br> [http://www.cdc.gov/dpdx/babesiosis/index.html <font size="-2">''Adapted from CDC''</font>]
Bmicroti_thin_giemsa_tetrad.jpg | ''Babesia microti'' in a thin blood smear stained with Giemsa. Babesia sp. cannot be identified to the species level by morphology alone; additional testing, such as PCR, is always recommended. Note the tetrad form in this image. <br> [http://www.cdc.gov/dpdx/babesiosis/index.html <font size="-2">''Adapted from CDC''</font>]
Bmicroti_thin_giemsa_tetrad.jpg | ''Babesia microti'' in a thin blood smear stained with Giemsa. ''Babesia'' sp. cannot be identified to the species level by morphology alone; additional testing, such as PCR, is always recommended. Note the tetrad form in this image. <br> [http://www.cdc.gov/dpdx/babesiosis/index.html <font size="-2">''Adapted from CDC''</font>]
Bmicroti_thin_giemsa2_arrows.jpg | ''Babesia microti'' in a thin blood smear stained with Giemsa. Note the intra-erythrocytic vacuolated forms indicated by the black arrows. <br> [http://www.cdc.gov/dpdx/babesiosis/index.html <font size="-2">''Adapted from CDC''</font>]
Bmicroti_thin_giemsa2_arrows.jpg | ''Babesia microti'' in a thin blood smear stained with Giemsa. Note the intra-erythrocytic vacuolated forms indicated by the black arrows. <br> [http://www.cdc.gov/dpdx/babesiosis/index.html <font size="-2">''Adapted from CDC''</font>]


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<gallery widths=200px>
<gallery widths=200px>


CercarialDermatitisA.jpg | Cercaria of ''Austrobilharzia variglandis'', which can cause cercarial dermatitis. <br> [http://www.cdc.gov/dpdx/cercarialDermatitis/gallery.html <font size="-2">''Adapted from CDC''</font>]
CercarialDermatitisA.jpg | Cercaria of ''Austrobilharzia variglandis'', which can cause cercarial dermatitis. Note the forked 'tail' and a pair of 'eye spots' near the anterior end. <br> [http://www.cdc.gov/dpdx/cercarialDermatitis/gallery.html <font size="-2">''Adapted from CDC''</font>]
CercarialDermatitisA.jpg | Cercaria of ''Austrobilharzia variglandis'', which can cause cercarial dermatitis. Note the forked 'tail' and a pair of 'eye spots' near the anterior end. <br> [http://www.cdc.gov/dpdx/cercarialDermatitis/gallery.html <font size="-2">''Adapted from CDC''</font>]


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<gallery widths=200px>
<gallery widths=200px>


Coenurus_BAM2.jpg | Large, polycephalic coenurus removed from the shoulder of a baboon (Papio sp.). <br> [http://www.cdc.gov/dpdx/coenurosis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Coenurus_BAM2.jpg | Large, polycephalic coenurus removed from the shoulder of a baboon (''Papio'' sp.). <br> [http://www.cdc.gov/dpdx/coenurosis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Coenurus_BAM3.jpg | Close-up of a coenurus of ''T. multiceps'' removed from the eye of a patient, broken open to show multiple protoscoleces. <br> [http://www.cdc.gov/dpdx/coenurosis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Coenurus_BAM3.jpg | Close-up of a coenurus of ''T. multiceps'' removed from the eye of a patient, broken open to show multiple protoscoleces. <br> [http://www.cdc.gov/dpdx/coenurosis/gallery.html <font size="-2">''Adapted from CDC''</font>]
</gallery>
</gallery>
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=====Dioctophymiasis=====
=====Dioctophymiasis=====
======''Dioctophyme renale''======
======''Dioctophyme renale''======
'''Larvae of Dioctotphyme renale in human tissue'''
'''Larvae of ''Dioctotphyme renale'' in human tissue'''
<gallery widths=200px>
<gallery widths=200px>


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</gallery>
</gallery>
======''Echinococcus oligarthrus''======
======''Echinococcus vogeli''======


=====Echinostomiasis=====
=====Echinostomiasis=====
======''Echinostoma spp.''======
======''Echinostoma spp.''======
'''''Echinostoma spp.'' egg in wet mounts'''
'''''Echinostoma'' spp. egg in wet mounts'''


<gallery widths=200px>
<gallery widths=200px>
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Echinostoma_tissue_BAM2.jpg | Higher magnification of the anterior end of the specimen in Figure 1. Notice the acetabulum (ventral sucker, AC). <br> [http://www.cdc.gov/dpdx/echinostomiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Echinostoma_tissue_BAM2.jpg | Higher magnification of the anterior end of the specimen in Figure 1. Notice the acetabulum (ventral sucker, AC). <br> [http://www.cdc.gov/dpdx/echinostomiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Echinostoma_tissue_BAM3.jpg | Higher magnification of the posterior end of the specimen in Figure 1. Notice the vitelline glands (VT) and lobed testes (TE). <br> [http://www.cdc.gov/dpdx/echinostomiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Echinostoma_tissue_BAM3.jpg | Higher magnification of the posterior end of the specimen in Figure 1. Notice the vitelline glands (VT) and lobed testes (TE). <br> [http://www.cdc.gov/dpdx/echinostomiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Echinostoma_tissue_BAM4.jpg | Higher magnification of the specimen in Figures 1-3. Shown here are eggs (EG) within the size range forEchinostoma spp. (roughly 100 micrometers in length, taking into account they are sections and may not be cut in a perfect horizontal plane). <br> [http://www.cdc.gov/dpdx/echinostomiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Echinostoma_tissue_BAM4.jpg | Higher magnification of the specimen in Figures 1-3. Shown here are eggs (EG) within the size range for ''Echinostoma'' spp. (roughly 100 micrometers in length, taking into account they are sections and may not be cut in a perfect horizontal plane). <br> [http://www.cdc.gov/dpdx/echinostomiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]


</gallery>
</gallery>


'''Intermediate hosts of ''Echinostoma spp.'''''
'''Intermediate hosts of ''Echinostoma'' spp.'''
<gallery widths=200px>
<gallery widths=200px>


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Ehartmanni_troph_tric3.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
Ehartmanni_troph_tric3.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
Ehartmanni_troph_tric4.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. In the upper-right of the image is a cyst-like body of ''Blastocystis hominis''. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
Ehartmanni_troph_tric4.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. In the upper-right of the image is a cyst-like body of ''Blastocystis hominis''. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
E_hartmanni_cyst_trich_BAM1.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
E_hartmanni_troph_trich_BAM1.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
E_hartmanni_cyst_trich_BAM2.jpg | Two trophozoites of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
E_hartmanni_troph_trich_BAM2.jpg | Two trophozoites of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
E_hartmanni_cyst_trich_BAM3.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]
E_hartmanni_troph_trich_BAM3.jpg | Trophozoite of ''E. hartmanni'' stained with trichrome. <br> [http://www.cdc.gov/dpdx/intestinalAmebae/gallery.html <font size="-2">''Adapted from CDC''</font>]


</gallery>
</gallery>
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</gallery>
</gallery>
======''Nosema'' spp.======
======''Pleistophora'' sp.======
======''Trachipleistophora'' spp.======
======''Vittaforma corneae''======


=====Myiasis (Bot Flies)=====
=====Myiasis (Bot Flies)=====
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O_volvulus_filaria_BAM1.jpg | Microfilariae of ''O. volvulus'' from a skin nodule of a patient from Zambia, stained with hematoxylin and eosin (H&E). Image taken at 1000x oil magnification. Microfilariae of ''O. volvulus'' within the uterus of an adult female. The specimen was taken from the same patient as in Figure 1. Image taken at 500x magnification, oil. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
O_volvulus_filaria_BAM1.jpg | Microfilariae of ''O. volvulus'' from a skin nodule of a patient from Zambia, stained with hematoxylin and eosin (H&E). Image taken at 1000x oil magnification. Microfilariae of ''O. volvulus'' within the uterus of an adult female. The specimen was taken from the same patient as in Figure 1. Image taken at 500x magnification, oil. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]


O_volvulus_filaria_BAM2.jpg | Microfilariae of ''O. volvulus'' within the uterus of an adult female. The specimen was taken from the same patient as in Figure A. Image taken at 500x magnification, oil. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
O_volvulus_filaria_BAM2.jpg | Microfilariae of ''O. volvulus'' within the uterus of an adult female. The specimen was taken from the same patient as in Figure 1. Image taken at 500x magnification, oil. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
O_volvulus_filaria_BAM3.jpg | Microfilariae of ''O. volvulus'' from a skin nodule of a patient from Zambia, stained with H&E. Image taken at 1000x oil magnification. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
O_volvulus_filaria_BAM3.jpg | Microfilariae of ''O. volvulus'' from a skin nodule of a patient from Zambia, stained with H&E. Image taken at 1000x oil magnification. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
O_volvulus_filaria_BAM4.jpg | Coiled microfilaria of ''O. volvulus'', in a skin nodule from a patient from Zambia, stained with H&E. Image taken at 1000x oil magnification. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
O_volvulus_filaria_BAM4.jpg | Coiled microfilaria of ''O. volvulus'', in a skin nodule from a patient from Zambia, stained with H&E. Image taken at 1000x oil magnification. <br> [http://www.cdc.gov/dpdx/onchocerciasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
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Paragonimus_oral_sucker.jpg | Adult of ''Paragonimus'' sp., taken from a lung biopsy specimen stained with hematoxylin and eosin (H&E). Note the presence of the oral sucker. The species was not identified in this case. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Paragonimus_oral_sucker.jpg | Adult of ''Paragonimus'' sp., taken from a lung biopsy specimen stained with hematoxylin and eosin (H&E). Note the presence of the oral sucker. The species was not identified in this case. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Paragonimus_cuticle_close-up.jpg | Higher magnification of Figure 3, showing a close-up of the cuticle. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
Paragonimus_cuticle_close-up.jpg | Higher magnification of Figure 3, showing a close-up of the cuticle. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
P_kellicotti_lung2.jpg | Adult of P. kellicotti taken from a lung biopsy specimen stained with H&E. This worm is in poor condition, indicating it was probably an old infection. Image courtesy of Dr. Miguel Madariaga, University of Nebraska Medical Center. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
P_kellicotti_lung2.jpg | Adult of ''P. kellicotti'' taken from a lung biopsy specimen stained with H&E. This worm is in poor condition, indicating it was probably an old infection. Image courtesy of Dr. Miguel Madariaga, University of Nebraska Medical Center. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
P_kellicotti_lung1.jpg | Sections of several adults of ''P. kellicotti'' taken from a pleural biopsy stained with H&E. Numbers 1-5 show the individual worms. The integument is gone from most of the worms, as this was probably an old infection, but remnants of the gonad (A, dart) and uterine tubes (B) can be seen. Image courtesy of Dr. Miguel Madariaga, University of Nebraska Medical Center. Image first appeared in: Madariaga, MD, M. G., T. Ruma, MD, and J. H. Theis, MD. 2007. Autochthonous human paragonimiasis in North America. Wilderness & Environmental Medicine. 18(3): 203-205. Image used with permission of © Allen Press Publishing Services. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]
P_kellicotti_lung1.jpg | Sections of several adults of ''P. kellicotti'' taken from a pleural biopsy stained with H&E. Numbers 1-5 show the individual worms. The integument is gone from most of the worms, as this was probably an old infection, but remnants of the gonad (A, dart) and uterine tubes (B) can be seen. Image courtesy of Dr. Miguel Madariaga, University of Nebraska Medical Center. Image first appeared in: Madariaga, MD, M. G., T. Ruma, MD, and J. H. Theis, MD. 2007. Autochthonous human paragonimiasis in North America. Wilderness & Environmental Medicine. 18(3): 203-205. Image used with permission of © Allen Press Publishing Services. <br> [http://www.cdc.gov/dpdx/paragonimiasis/gallery.html <font size="-2">''Adapted from CDC''</font>]


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====S====
====S====
=====Sarcocystosis=====
=====Sarcocystosis=====
======''Sarcocystis hominis''======
======''Sarcocystis suihominis''======
======''Sarcocystis'' spp.======
======''Sarcocystis'' spp.======
'''''Sarcocystis'' oocysts in wet mounts'''
'''''Sarcocystis'' oocysts in wet mounts'''
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Taenia_egg_wtmt_OR.jpg |  Iodine-stained wet mount of a ''Taenia'' sp. egg. Image courtesy of the Oregon State Public Health Laboratory. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_egg_wtmt_OR.jpg |  Iodine-stained wet mount of a ''Taenia'' sp. egg. Image courtesy of the Oregon State Public Health Laboratory. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_egg1.jpg | Iodine-stained wet mount of a ''Taenia'' sp. egg. Image courtesy of the Oregon State Public Health Laboratory. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_egg1.jpg | Iodine-stained wet mount of a ''Taenia'' sp. egg. Image courtesy of the Oregon State Public Health Laboratory. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_solium_scolex1.jpg | Unstained ''Taenia'' sp. egg, teased from a proglottid of an adult. Four hooks can easily be seen in this image. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]


</gallery>
</gallery>
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Taenia_solium_scolex1.jpg | Scolex of ''T. solium''. Note the four large suckers and rostellum containing two rows of hooks. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_solium_scolex1.jpg | Scolex of ''T. solium''. Note the four large suckers and rostellum containing two rows of hooks. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_solium_scolex2.jpg | Scolex of ''T. solium''. Note the four large suckers and rostellum containing two rows of hooks. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_solium_scolex2.jpg | Scolex of ''T. solium''. Note the four large suckers and rostellum containing two rows of hooks. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_solium_scolex.jpg | Scolex of ''T. saginata''. Note the four large suckers and lack of rostellum and rostellar hooks. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]


</gallery>
</gallery>
Line 2,540: Line 2,522:
Taenia_eggs_HE.jpg | Higher magnification of the image in Figure 2, showing a close-up of the eggs. Note the characteristic striations, typical for the taeniids. Not visible in these images are the hooks commonly seen in cestode eggs. Hooks do not stain with H&E but are refractile and are visible with fine focusing of the microscope <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_eggs_HE.jpg | Higher magnification of the image in Figure 2, showing a close-up of the eggs. Note the characteristic striations, typical for the taeniids. Not visible in these images are the hooks commonly seen in cestode eggs. Hooks do not stain with H&E but are refractile and are visible with fine focusing of the microscope <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_eggs_HE2.jpg | Higher magnification of the image in Figure 2, showing a close-up of the eggs. Note the characteristic striations, typical for the taeniids. Not visible in these images are the hooks commonly seen in cestode eggs. Hooks do not stain with H&E but are refractile and are visible with fine focusing of the microscope. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
Taenia_eggs_HE2.jpg | Higher magnification of the image in Figure 2, showing a close-up of the eggs. Note the characteristic striations, typical for the taeniids. Not visible in these images are the hooks commonly seen in cestode eggs. Hooks do not stain with H&E but are refractile and are visible with fine focusing of the microscope. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
ImageName.jpg | Close-up of a cross-section of a ''Taenia'' sp. proglottid stained with H&E, showing numerous calcareous corpuscles (yellow arrows). Image courtesy of the Michael E. DeBakey V. A. Medical Center in Houston, TX. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
ImageName.jpg | Close-up of a cross-section of a ''Taenia'' sp. proglottid stained with H&E, showing numerous calcareous corpuscles. Image courtesy of Ameripath. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
</gallery>
'''''Taenia'' spp. adults'''
<gallery widths=200px>
ImageName.jpg | Taenia saginata adult worm. The adult in this image is approximately 4 meters in length. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]
ImageName.jpg | ''Taenia saginata'' adult worm. <br> [http://www.cdc.gov/dpdx/taeniasis/gallery.html# <font size="-2">''Adapted from CDC''</font>]


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Latest revision as of 12:29, 28 July 2015

Template

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ImageName.jpg | Description <br> [http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
ImageName.jpg | Description <br> [http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]
ImageName.jpg | Description <br> [http://phil.cdc.gov/Phil/ <font size="-2">''Adapted from CDC''</font>]


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Sexually Transmitted Disease Gallery

Bacterial vaginosis

Chlamydia

Genital Herpes

Gonorrhea

Human papillomavirus

Lymphogranuloma venereum

Pubic lice infestation

Scabies

Syphilis

Trichomoniasis


Parasite Gallery

A

Acanthocephaliasis
Bolbosoma spp.
Macracanthorhynchus hirudinaceous

Eggs of M. hirudinaceous

Adults of M. hirudinaceous

Moniliformis moniliformis

Eggs of M. moniliformis

Adults of M. moniliformis

African trypanosomiasis (Sleeping sickness)
Trypansoma brucei

Trypansoma brucei ssp. in thick blood smear stained with Giemsa

Trypansoma brucei ssp. in thin blood smear stained with Giemsa

Trypansoma brucei ssp. in thin blood smears stained with Wright-Giemsa

Trypansoma brucei ssp. in thin blood smear, beginning to divide

Amebiasis

Entamoeba sp. cysts

Entamoeba sp. trophozioites

American trypanosomiasis (Chagas disease)
Trypanosoma cruzi

Trypanosoma cruzi in thick blood smears stained with Giemsa

T. cruzi in thin blood smears stained with Giemsa

T. cruzi in cerebrospinal fluid (CSF) stained with Giemsa

T. cruzi amastigotes in heart tissue

T. cruzi epimastigotes, from culture

Ancylostomiasis (Hookworm)
Ancylostoma braziliense
Ancylostoma caninum
Ancylostoma ceylanicum
Ancylostoma duodenale
Necator americanus
Angiostrongyliasis
Angiostrongylus cantonensis
Angiostrongylus costaricensis

A. costaricensis Eggs

A. costaricensis adult female in tissue sections stained with H&E

Anisakiasis
Anisakis simplex
Pseudoterranova decipiens

Pseudoterranova sp. larval worms

Cross sections of Pseudoterranova sp. worms

Ascariasis
Ascaris lumbricoides

Adult A. lumbricoides

Unfertilized egg of A. lumbricoides

Fertilized egg of A. lumbricoides

A. lumbricoides in tissue specimen

B

Babesiosis
Babesia divergens
Babesia microti
Balantidiasis
Balantidium coli

B. coli cysts

B. coli trophozoites

Baylisascariasis
Baylisascaris procyonis

Baylisascaris procyonis eggs

Baylisascaris procyonis larvae

Baylisascaris procyonis larvae

Bed Bugs
Cimex hemipterus
Cimex lectularius
Bertiella infection
Bertiella mucronata
Bertiella struderi
Blastocystis hominis infection
Blastocystis hominis

Blastocystis hominis cyst-like forms in wet mounts

B. hominis cyst-like forms in wet mounts under differential interference contrast (DIC) microscopy

B. hominis cyst-like forms in wet mounts stained with iodine

B. hominis cyst-like forms stained with trichrome

C

Cercarial dermatitis (Swimmer’s itch)
Austrobilharzia variglandis

Cercaria of Austrobilharzia variglandis

Clinical manifestations of Austrobilharzia variglandis

Chilomastix mesnili

Chilomastix mesnili trophozoites, trichrome stain

Chilomastix mesnili cysts, trichrome stain

Chilomastix mesnili cysts in wet mounts

Clonorchiasis
Clonorchis sinensis

Clonorchis sinensis eggs

Clonorchis sinensis adults

Clonorchis sinensis eggs

Coenurosis
Taenia spp.

Gross coenurus specimens

Coenuri in tissue specimens, stained with hematoxylin and eosin (H&E)

Coenurus in an eye specimens, stained with hematoxylin and eosin (H&E)

Cryptosporidiosis
Cryptosporidium spp.

Cryptosporidium sp. oocysts in a wet mount

Cryptosporidium sp. oocysts stained with trichrome

Cryptosporidium sp. oocysts stained with modified acid-fast

Cryptosporidium sp. oocysts unstained on a slide stained with modified acid-fast

Cryptosporidium sp. oocysts stained with safranin

Cryptosporidium sp. oocysts stained with Ziehl-Neelsen modified acid-fast

Cryptosporidium parvum oocysts stained with the fluorescent stain auramine-rhodamine

Oocysts of C. parvum' and cysts of Giardia duodenalis labeled with immunofluorescent antibodies

Cyclosporiasis
Cyclospora cayetanensis

Cyclospora cayetanensis oocysts in wet mounts

Cyclospora cayetanensis oocysts stained with trichrome

C. cayetanensis oocysts viewed under ultraviolet (UV) microscopy

C. cayetanensis oocysts stained with modified acid-fast

C. cayetanensis oocysts stained with safranin (SAF)

C. cayetanensis oocysts viewed under differential interference contrast (DIC) microscopy

Cysticercosis
Taenia solium

Larval Taenia solium

Cystoisosporiasis (Isosporiasis)
Cystoisospora belli (Isospora belli)

Cystoisospora belli oocysts

Cystoisospora belli oocysts, stained with hematoxylin and eoisin (H&E)

D

Dicrocoeliasis
Dicrocoelium dendriticum

Dicrocoelium dendriticum eggs in wet mounts

Dicrocoelium dendriticum adults

Intermediate hosts of Dicrocoelium dendriticum

Dientamoeba fragilis infection
Dientamoeba fragilis

Dientamoeba fragilis binucleate trophozoites stained with trichrome

Dientamoeba fragilis uninucleate trophozoites stained with trichrome

Dioctophymiasis
Dioctophyme renale

Larvae of Dioctotphyme renale in human tissue

Eggs of D. renale in animal tissue

Diphyllobothriasis
Diphyllobothrium latum

Diphyllobothrium latum eggs in wet mounts

Eggs of Diphyllobothrium latum eggs in wet mounts

Proglottids of Diphyllobothrium latum

Dipylidium caninum infection

Dipylidium caninum egg packets in wet mounts

D. caninum eggs in wet mounts under conventional and differential interference contrast microscopy

D. caninum proglottids

Cross-section of a D. caninum proglottid stained with hematoxylin and eosin (H&E)

D. caninum scolex

Adult tapeworm of D. caninum

Dirofilariasis
Dirofilaria sp.
Dracunculiasis (Guinea Worm Disease)
Drancunculus medinensis

A female Dracuncunculus medinensis in a human host

E

Echinococcosis (Hydatid disease)
Echinococcus granulosus

Echinococcus granulosus in tissue

Echinococcus granulosus adults

Echinococcus multilocularis
Echinostomiasis
Echinostoma spp.

Echinostoma spp. egg in wet mounts

Echinostoma spp. adults

Echinostoma sp. in tissue, stained with hematoxylin and eosin (H&E)

Intermediate hosts of Echinostoma spp.

Enterobiasis (Pinworm Infection)
Enterobius vermicularis

Enterobius vermicularis eggs

Enterobius vermicularis adult worms

Enterobius vermicularis in tissue, stained with hematoxylin and eosin (H&E)

Enteromonas hominis

Enteromonas hominis cysts

F

Fascioliasis
Fasciola hepatica

Fasciola hepatica eggs

F. hepatica adults

F. hepatica adults observed in endoscopic retrograde cholangiopancreatography (ERCP)

Intermediate hosts of Fasciola spp.

Fasciolopsiasis
Fasciolopsis buski

Fasciolopsis buski eggs

Fasciolopsis buski adults

Intermediate hosts of F. buski

Fleas
Ctenocephalides canis
Ctenocephalides felis
Free-living amebic infections
Acanthamoeba

Acanthamoeba spp. cysts

Acanthamoeba spp. trophozoites

Balamuthia mandrillaris

Balamuthia mandrillaris cysts

Balamuthia mandrillaris trophozoites

Naegleria fowleri

Naegleria fowleri cysts

Naegleria fowleri trophozoites

Sappinia pedata

G

Giardiasis
Giardia duodenalis (syn. G. lamblia, G. intestinalis)

Giardia duodenalis cysts in wet mounts stained with iodine

Giardia duodenalis cysts in wet mounts under differential interference contrast (DIC) microscopy

G. duodenalis cysts in trichrome stain

G. duodenalis trophozoites in wet mounts

G. duodenalis trophozoites stained with trichrome

G. duodenalis trophozoites in unique stains

Cysts of Giardia duodenalis and oocysts of Cryptosporidium parvum

Gnathostomiasis
Gnathostoma hispidum
Gnathostoma spinigerum

Head bulb and cuticular spines of Gnathostoma spinigerum

Detail of cuticular spines of the anterior body part of G. spinigerum

Detail of nondendiculated cuticular spines of G. spinigerum

H

Hepatic capillariasis
Capillaria hepatica

Capillaria hepatica eggs

Capillaria hepatica adults

Heterophyiasis
Heterophyes heterophyes

Adult of Heterophyes heterophyes

Snail intermediate hosts of Heterophyes heterophyes

Hymenolepiasis
Hymenolepis diminuta

Hymenolepis diminuta eggs in wet mounts

Hymenolepis diminuta proglottids

Hymenolepis nana

Hymenolepis nana eggs in wet mounts

Hymenolepis nana eggs, zinc PVA trichrome stain

Hymenolepis nana proglottids

Hymenolepis nana adults

I

Intestinal amebae
Entamoeba coli

E. coli cysts in concentrated wet mounts

E. coli cysts stained with trichrome

E. coli trophozoites stained with trichrome

Entamoeba gingivalis

E. gingivalis trophozoites stained with trichrome

Entamoeba hartmanni

E. hartmanni cyst in a wet mount

E. hartmanni cysts stained with trichrome

E. hartmanni trophozoites stained with trichrome

Entamoeba histolytica
Entamoeba polecki

E. polecki cyst in a concentrated wet mount, stained with iodine

E. polecki cysts stained with trichrome

E. polecki trophozoites stained with trichrome

Endolimax nana

Endolimax nana cysts in concentrated wet mounts

E. nana cyst stained with trichrome

E. nana trophozoites stained with trichrome

Iodamoeba buetschlii

Iodamoeba buetschlii cysts in concentrated wet mounts

I. buetschlii cysts stained with trichrome

I. buetschlii trophozoite stained with trichrome

Intestinal capillariasis
Capillaria philippinensis

Capillaria philippinensis eggs

Capillaria philippinensis adults

J

K

L

Leishmaniasis (Visceral leishmaniasis, Kala-azar)
Leishmania sp.

Leishmania amastigotes

Leishmania mexicana in tissue stained with hematoxylin and eosin (H&E)

Leishmania sp. promastigotes from culture

Loaiasis
Loa loa

Microfilariae of Loa loa

Adults of L. loa

Lymphatic filariasis (Bancroftian filariasis)
Brugia malayi
Brugia timori
Wuchereria bancrofti

Microfilariae of Wuchereria bancrofti

Adults of W. bancrofti

M

Malaria
Plasmodium falciparum
Plasmodium knowlesi
Plasmodium malariae
Plasmodium ovale
Plasmodium vivax
Mansonellosis
Mansonella ozzardi

Microfilariae of Mansonella ozzardi

Mansonella perstans

Microfilariae of Mansonella perstans

Mansonella streptocerca

Microfilariae of Mansonella streptocerca

Mesocestoidiasis
Mesocestoides spp.

Mesocestoides spp. proglottids and scoleces

Mesocestoides spp. tetrathyridia

Metagonimiasis
Metagonimus yokogawai

Metagonimus yokogawai, adult fluke

Snail intermediate hosts of M. yokogawai

Microsporidiosis
Encephalitozoon cuniculi
Encephalitozoon hellem
Encephalitozoon intestinalis
Enterocytozoon bieneusi
Myiasis (Bot Flies)
Cuterebra spp.
Dermatobia hominis
Oestrus ovis
Phormia regina

N

O

Oesophagostomiasis
Oesophagostomum spp.

Eggs of Oesophagostomum spp.

L3 infective larvae of Oesophagostomum spp.

Adults of Oesophagostomum spp.

Oesophagostomum spp. in tissue specimens

Onchocerciasis (River Blindness)
Onchocerca volvulus

Microfilariae of Onchocerca volvulus in tissue

Adults of Onchocerca volvulus in tissue

Opisthorchiasis
Opisthorchis felineus

Adults of Opisthorchis felineus

Intermediate hosts of Opisthorchis spp.

Opisthorchis viverrini

Eggs of Opisthorchis viverrini in wet mounts

Adults of O. viverrini

P

Paragonimiasis
Paragonimus spp.

Eggs of Paragonimus spp. in unstained wet mounts

Eggs of Paragonimus spp. in tissue

Eggs of Paragonimus kellicotti

Adults of Paragonimus spp.

Pediculosis
Pediculosis sp.

Head and Body Lice adults

Head and Body Lice nits

Pentatrichomonas hominis

Pentatrichomonas hominis trophozoites

Philophthalmiasis
Philophthalmus spp.

Philophthalmus spp, adult flukes

Snail intermediate hosts of Philophthalmus spp.

Pneumocystis pneumonia (PCP)
Pneumocystis jirovecii (previously Pneumocystis carinii)

Pneumocystis jirovecii trophozoites

Pneumocystis jirovecii cysts

Indirect immunofluorescence using monoclonal antibodies against Pneumocystis jirovecii

Q

R

Retortamonas intestinalis

Retortamonas intestinalis, trophozoites

Retortamonas intestinalis, cysts

S

Sarcocystosis
Sarcocystis spp.

Sarcocystis oocysts in wet mounts

Sarcocystis oocysts in wet mounts viewed under differential interference contrast (DIC)

Sarcocystis oocysts in wet mounts viewed under ultraviolet (UV) microscopy

Sarcocystis sarcocysts in tissue

Schistosomiasis (Bilharziasis)
Schistosoma haematobium
Schistosoma intercalatum
Schistosoma japonicum
Schistosoma mansoni
Schistosoma mekongi
Sparganosis

Proliferating spargana in groin tissue

Proliferating spargana in lung tissue

Spargana removed from tissue

Sparganum proliferum
Spirometra erinacei
Spirometra mansoni
Spirometra mansonoides
Spirometra ranarum
Strongyloidiasis
Strongyloides stercoralis

Strongyloides stercoralis first-stage rhabditiform (L1) larvae

Strongyloides stercoralis third-stage filariform (L3) larvae

Strongyloides stercoralis free-living adults

Strongyloides stercoralis in tissue

T

Taeniasis
Taenia spp.

Taenia spp. eggs

Taenia spp. scoleces

Taenia spp. proglottids

Cross-sections of Taenia spp. stained with hematoxylin and eosin (H&E)

Thelaziasis
Thelazia spp.

Thelazia spp. adults

Intermediate hosts of Thelazia spp.

Toxocariasis
Toxocara canis

Toxocara canis larva hatching

Toxocara cati

Adult Toxocara cati worms

Toxoplasmosis
Toxoplasma gondii
Trichinellosis (Trichinosis)
Trichinella spp.

Encysted larvae of Trichinella in tissue, stained with hematoxylin and eosin (H&E)

Trichinella larvae in tongue tissue of a rat, stained with H&E

Larvae of Trichinella from bear meat

Trichostrongylosis
Trichostrongylus spp.

Trichostrongylus spp. eggs in wet mounts

Trichostrongyle eggs in wet mounts

Trichostrongylus adults

Trichuriasis (Whip Worm)
Trichuris trichiura
Tungiasis
Tunga penetrans

Tunga penetrans

Tunga penetrans lesions and biopsy specimens

U

V

W

X

Y

Z