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{{CMG}}
{{CMG}}
{| border="1"
|+ The table's caption
! Differentiating features !! Leukemia !! Leukemoid reaction
|-
! Causes
| Carcinogens || Infectious agents, biologically active substances and
products of tissue destruction
|-
! Pathogenesis
| The transformation of normal haematopoietic
cells to a tumor
| Activation of normal hematopoiesis and exit of immature leukocytes
into the bloodstream. 
|-
! Duration
| Chronic
| Temporary and reversible
|-
! Genetic level
| Defect
| No defect
|-
! Splenomegaly
| Present
| Not present
|-
! Peripheral blood
| Immature cells, pancytopenia
| Mature and immature granulocytes with left shift
|-
! Bone marrow
| Increase in blasts and immature cells
| Myeloid hyperplasia and normal morphology
|-
! LAP score
| Low
| High
|-
! Toxic granules
(suggestive of infection)
| Absent
| Present
|-
! Dohle inclusion bodies
| Absent
| Present

Latest revision as of 14:25, 9 October 2018


Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]


The table's caption
Differentiating features Leukemia Leukemoid reaction
Causes Carcinogens Infectious agents, biologically active substances and

products of tissue destruction

Pathogenesis The transformation of normal haematopoietic

cells to a tumor

Activation of normal hematopoiesis and exit of immature leukocytes

into the bloodstream.

Duration Chronic Temporary and reversible
Genetic level Defect No defect
Splenomegaly Present Not present
Peripheral blood Immature cells, pancytopenia Mature and immature granulocytes with left shift
Bone marrow Increase in blasts and immature cells Myeloid hyperplasia and normal morphology
LAP score Low High
Toxic granules
(suggestive of infection)
Absent Present
Dohle inclusion bodies Absent Present