Sandbox:Aneeza: Difference between revisions
Jump to navigation
Jump to search
m (ADDED MICROCHAPTER) |
m (added data) |
||
| Line 7: | Line 7: | ||
===HISTORICAL PERSPECTIVES:=== | ===HISTORICAL PERSPECTIVES:=== | ||
Libman and Sack in 1924, first coined this term for the sterile verrucous vegetations of the mitral and aortic valves in patients | Libman and Sack in 1924, first coined this term for the sterile verrucous vegetations of the mitral and aortic valves in four patients in New York<ref name="pmidPMID: 19089657">{{cite journal| author=Lee JL, Naguwa SM, Cheema GS, Gershwin ME| title=Revisiting Libman-Sacks endocarditis: a historical review and update. | journal=Clin Rev Allergy Immunol | year= 2009 | volume= 36 | issue= 2-3 | pages= 126-30 | pmid=PMID: 19089657 | doi=10.1007/s12016-008-8113-y | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=19089657 }} </ref>. | ||
<br /> | <br /> | ||
=== PATHOPHYSIOLOGY: === | ===PATHOPHYSIOLOGY:=== | ||
Endothelial dysfunction and hypercoagulable state are the two most important events in the process of non-thrombotic non-bacterial endocarditis (NTBE). | |||
- | |||
{| class="infobox bordered" style="width: 15em; text-align: left; font-size: 90%; background:AliceBlue" | {| class="infobox bordered" style="width: 15em; text-align: left; font-size: 90%; background:AliceBlue" | ||
| Line 81: | Line 51: | ||
|- bgcolor="Pink" | |- bgcolor="Pink" | ||
! | ! | ||
[[Libman-Sacks endocarditis# | [[Libman-Sacks endocarditis#Etiology|Etiology]] | ||
|- | |- | ||
! | ! | ||
Revision as of 12:31, 4 April 2021
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aneeza Jamshed, M.B.B.S.[2]
LIBMAN-SACK ENDOCARDITIS:
OVERVIEW:
Libman-sack endocarditis is the one of the cardiac manifestations of autoimmune systemic diseases most commonly Systemic Lupus Erythematosus. It may also be a finding in malignancy with the non-bacterial non-thrombotic involvement of the valves, most commonly the aortic and mitral valve. The most common complications include embolic phenomenon like cerebrovascular disease and infective endocarditis with valvular insufficeincy. The disease activity and titer of the antibodies in Systemic Lupus Erythematosus can be linked to Libman-sack Endocarditis[1].
HISTORICAL PERSPECTIVES:
Libman and Sack in 1924, first coined this term for the sterile verrucous vegetations of the mitral and aortic valves in four patients in New York[2].
PATHOPHYSIOLOGY:
Endothelial dysfunction and hypercoagulable state are the two most important events in the process of non-thrombotic non-bacterial endocarditis (NTBE).
-
|
Libman-Sacks endocarditis Microchapters |
|
Differentiating Libman-Sacks Endocarditis from other Diseases |
|---|
Reference
- ↑ Carrillo-Esper R, Carrillo-Córdova JR, Carrillo-Córdova LD, Carrillo-Córdova CA (2010). "[Libman-Sacks endocarditis]". Gac Med Mex. 146 (1): 67–9. PMID 20422938 PMID: 20422938 Check
|pmid=value (help). - ↑ Lee JL, Naguwa SM, Cheema GS, Gershwin ME (2009). "Revisiting Libman-Sacks endocarditis: a historical review and update". Clin Rev Allergy Immunol. 36 (2–3): 126–30. doi:10.1007/s12016-008-8113-y. PMID 19089657 PMID: 19089657 Check
|pmid=value (help).