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==Overview==
==Overview==
The history of diabetes insipidus dates as far back as the early 1670s, when Thomas Willis noted that there was a difference in the taste of [[urine]] produced by different patients who presented with [[polyuria]] and [[polydipsia]]. This marked the beginning of the [[research]] into the difference between the popularly known [[diabetes mellitus]] and [[diabetes insipidus]].


==Historical Perspective==
==Historical Perspective==
*Thomas Willis was first noted the difference [[diabetes mellitus]] from [[Diabetes insipidus|diabetes insipidus]] in 1670.<ref name="pmid269138702">{{cite journal |vauthors=Valenti G, Tamma G |title=History of Diabetes Insipidus |journal=G Ital Nefrol |volume=33 Suppl 66 |issue= |pages=33.S66.1 |date=February 2016 |pmid=26913870 |doi= |url=}}</ref>
*Johann Peter Frank was first introduced the term "[[diabetes insipidus]]"; described [[Polyuria|polyuric]] patients excreting nonsaccharine urine in 1794.<ref name="pmid269138702">{{cite journal |vauthors=Valenti G, Tamma G |title=History of Diabetes Insipidus |journal=G Ital Nefrol |volume=33 Suppl 66 |issue= |pages=33.S66.1 |date=February 2016 |pmid=26913870 |doi= |url=}}</ref><ref>{{cite journal|title=The Etiology and Treatment of Diabetes Insipidus|journal=Annals of Internal Medicine|volume=5|issue=5|year=1931|pages=566|issn=0003-4819|doi=10.7326/0003-4819-5-5-566}}</ref>
*In 1913, a historical milestone was achieved when Farini successfully used [[Posterior pituitary gland|posterior pituitary]] extracts to treat [[diabetes insipidus]].
*In the early 1920s the available evidence indicated that diabetes insipidus was a disorder of the [[pituitary gland]].
*In 1928, De Lange first observed that some patients with [[diabetes insipidus]] did not respond to [[posterior pituitary]] extracts.
*In 1945, Forssman and Waring established that the [[kidney]] played a critical role in the forms of diabetes insipidus that were resistant to this treatment.
*In 1947, Williams and Henry introduced the term [[nephrogenic diabetes insipidus]] for the [[Congenital syndromes|congenital syndrome]] characterized by [[polyuria]] and [[renal]] concentrating defect resistant to [[vasopressin]].
*In 1955, du Vigneaud received the 1955 [[Nobel Prize]] in [[chemistry]] for the first synthesis of the hormone [[vasopressin]], which represented a milestone in the development of treatment for [[central diabetes insipidus]].<ref name="pmid26913870">{{cite journal |vauthors=Valenti G, Tamma G |title=History of Diabetes Insipidus |journal=G Ital Nefrol |volume=33 Suppl 66 |issue= |pages=33.S66.1 |year=2016 |pmid=26913870 |doi= |url=}}</ref>


==References==
==References==
{{reflist|2}}
{{reflist|2}}
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Latest revision as of 16:47, 29 October 2020

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Omodamola Aje B.Sc, M.D. [2]

Overview

The history of diabetes insipidus dates as far back as the early 1670s, when Thomas Willis noted that there was a difference in the taste of urine produced by different patients who presented with polyuria and polydipsia. This marked the beginning of the research into the difference between the popularly known diabetes mellitus and diabetes insipidus.

Historical Perspective

References

  1. 1.0 1.1 Valenti G, Tamma G (February 2016). "History of Diabetes Insipidus". G Ital Nefrol. 33 Suppl 66: 33.S66.1. PMID 26913870.
  2. "The Etiology and Treatment of Diabetes Insipidus". Annals of Internal Medicine. 5 (5): 566. 1931. doi:10.7326/0003-4819-5-5-566. ISSN 0003-4819.
  3. Valenti G, Tamma G (2016). "History of Diabetes Insipidus". G Ital Nefrol. 33 Suppl 66: 33.S66.1. PMID 26913870.