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{{Beer potomania}}
'''For patient information, click [[Beer potomania (patient information)|here]]'''


'''Editor-in-Chief:''' Suresh Samson, M.D., Yale Bridgeport Hospital
'''Editor-in-Chief:''' Suresh Samson, M.D., Yale Bridgeport Hospital
==[[Beer potomania overview|Overview]]==


{{Editor Join}}
==[[Beer potomania historical perspective|Historical Perspective]]==


== Overview ==
==[[Beer potomania classification|Classification]]==
Beer potomania is defined as the development of [[hyponatremia]] or low levels of salt in the bloodstream, as a result of excess beer intake coupled with poor nutritional intake. This is a syndrome of binge drinking, poor nutritional intake, and profound [[hyponatremia]].


== Pathophysiology & Etiology==
==[[Beer potomania pathophysiology|Pathophysiology]]==
[[Hyponatremia]] is due to a large consumption of beer (which has a poor salt content) together with a minimal intake of ordinary food. Three facts contribute to beer potomania:
#Beer contains a lot of free water with very little salt and protein (one liter of beer contains only 30 mg of sodium).
#The amount of free water we excrete depends upon number of [[osmoles]] that need to be excreted.
#The kidneys can dilute urine to a maximum of 50 mosm/L


The pathophysiologic mechanism of beer potomania can be understood through an example. Imagine someone who binged on 4 liters of beer (which essentially means 4 liters of water) with ingestion of roughly 200 mosm that day. To place this in context, a normal American diet contains 750 mosm/day, and all of it is excreted in urine to maintain a steady state.  As a result of the ingestion of this large amount of beer (i.e.free water), the patient's anti diuretic hormone(ADH) (the hormone that causes the kidney to reabsorb free water) secretion is at its minimum to allow the kidneys to excrete maximally dilute urine. Since the maximal dilution that can be attained is 50mosm/L, the kidneys will excrete all the 200 mosm in the diet in 4 litres of urine (50mosm/L X 4L = 200 mosm). This will indeed maintain his sodium level.If this person in our example drinks his 5th litre of beer without taking any further osmoles, then he will become hyponatremic. This is beer potomania
==[[Beer potomania causes|Causes]]==


===== References =====
==[[Beer potomania differential diagnosis|Differentiating Beer potomania from other Diseases]]==
http://alcalc.oxfordjournals.org/cgi/content/full/35/6/612


=== Differential Diagnosis ===
==[[Beer potomania epidemiology and demographics|Epidemiology and Demographics]]==
See the differential of [[hyponatremia]]


==== Electrolyte and Biomarker Studies ====
==[[Beer potomania risk factors|Risk Factors]]==
A value of Uosm < 100 mOsm/kg is indicative of a complete and appropriate suppression of antidiuretic hormone (ADH) secretion, a finding seen with either primary polydipsia (including ‘beer potomania’) or reset osmostat.
Criteria used to diagnose the ‘beer potomania’ syndrome include
# A history of binge beer drinking
# Poor dietary intake
# Decreased serum sodium levels
# Low serum and urine osmolality
# Absence of other known causes of hyponatremia


== Acknowledgements ==
==[[Beer potomania natural history, complications and prognosis|Natural History, Complications, and Prognosis]]==
The content on this page was first contributed by: {{CMG}}
 
==Diagnosis==
[[Beer potomania history and symptoms|History and Symptoms]] | [[Beer potomania physical examination|Physical Examination]] | [[Beer potomania laboratory findings|Laboratory Findings]] | [[Beer potomania other diagnostic studies|Other Diagnostic Studies]]
 
==Treatment==
[[Beer potomania medical therapy|Medical Therapy]] | [[Beer potomania primary prevention|Primary Prevention]] | [[Beer potomania cost-effectiveness of therapy|Cost-Effectiveness of Therapy]] | [[Beer potomania future or investigational therapies|Future or Investigational Therapies]]
 
==Case Studies==
[[Beer potomania case study one|Case #1]]


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Latest revision as of 19:26, 1 February 2013

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Editor-in-Chief: Suresh Samson, M.D., Yale Bridgeport Hospital

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Beer potomania from other Diseases

Epidemiology and Demographics

Risk Factors

Natural History, Complications, and Prognosis

Diagnosis

History and Symptoms | Physical Examination | Laboratory Findings | Other Diagnostic Studies

Treatment

Medical Therapy | Primary Prevention | Cost-Effectiveness of Therapy | Future or Investigational Therapies

Case Studies

Case #1


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