Aortic stenosis physical examination: Difference between revisions

Jump to navigation Jump to search
Line 24: Line 24:
==Signs of Aortic Stenosis==
==Signs of Aortic Stenosis==
*Aortic stenosis murmer. It is mid-systolic ejection murmur that is low-pitched and rough, it is loudest at the base of the heart and transmitted upward along the carotid arteries. The murmer is at least grade III or IV in most patients with severe obstruction, it may be soft in patients with mild degrees of obstruction or in those with heart failure, in whom [[stroke volume is reduced]].
*Aortic stenosis murmer. It is mid-systolic ejection murmur that is low-pitched and rough, it is loudest at the base of the heart and transmitted upward along the carotid arteries. The murmer is at least grade III or IV in most patients with severe obstruction, it may be soft in patients with mild degrees of obstruction or in those with heart failure, in whom [[stroke volume is reduced]].
*Systolic thrill in the same location of murmur.
*Systolic thrill in the same location of murmur.
 
*Early systolic ejection murmer (the opening snap of the aortic valve).
*Paradoxical splitting of S2 from prolongation of LV systole.
*S4 may be audible at the apex.
*S3 generally occurs due to left ventricular dilatation.
Signs associated with peripheral edema include:
Signs associated with peripheral edema include:
* A slow-rising, small volume carotid pulse.
* A slow-rising, small volume carotid pulse.

Revision as of 18:19, 19 October 2011

Aortic Stenosis Microchapters

Home

Patient Information

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Aortic Stenosis from other Diseases

Epidemiology and Demographics

Risk Factors

Natural History, Complications and Prognosis

Diagnosis

History and Symptoms

Physical Examination

Cardiac Stress Test

Electrocardiogram

Chest X Ray

CT

MRI

Echocardiography

Cardiac Catheterization

Aortic Valve Area

Aortic Valve Area Calculation

Treatment

General Approach

Medical Therapy

Surgery

Percutaneous Aortic Balloon Valvotomy (PABV) or Aortic Valvuloplasty

Transcatheter Aortic Valve Replacement (TAVR)

TAVR vs SAVR
Critical Pathway
Patient Selection
Imaging
Evaluation
Valve Types
TAVR Procedure
Post TAVR management
AHA/ACC Guideline Recommendations

Follow Up

Prevention

Precautions and Prophylaxis

Cost-Effectiveness of Therapy

Future or Investigational Therapies

Case Studies

Case #1

Aortic stenosis physical examination On the Web

Most recent articles

Most cited articles

Review articles

CME Programs

Powerpoint slides

Images

American Roentgen Ray Society Images of Aortic stenosis physical examination

All Images
X-rays
Echo & Ultrasound
CT Images
MRI

Ongoing Trials at Clinical Trials.gov

US National Guidelines Clearinghouse

NICE Guidance

FDA on Aortic stenosis physical examination

CDC on Aortic stenosis physical examination

Aortic stenosis physical examination in the news

Blogs on Aortic stenosis physical examination

Directions to Hospitals Treating Aortic stenosis physical examination

Risk calculators and risk factors for Aortic stenosis physical examination

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editors-In-Chief: Claudia P. Hochberg, M.D. [2], Abdul-Rahman Arabi, M.D. [3], Keri Shafer, M.D. [4], Priyamvada Singh, MBBS [5]; Assistant Editor-In-Chief: Kristin Feeney, B.S. [6]

Overview

Upon physical examination, a patient with suspected aortic stenosis may present with signs such as peripheral edema, congestive heart failure, a slow-rising, small volume carotid pulse, lag time between apical and carotid impulses, systolic hypertension, and a distinct systolic murmur.

Physical Examination

The critically ill patient may be in extremis. Peripheral edema may be present in the patient with CHF. Pulmonary rales may be present in the patient with CHF.

Aortic stenosis is most often diagnosed when it is asymptomatic and can sometimes be detected during routine examination of the heart and circulatory system. Good evidence exists to demonstrate that certain characteristics of the peripheral pulse can rule in the diagnosis [1]. In particular, there may be a slow and/or sustained upstroke of the arterial pulse, and the pulse may be of low volume. This is sometimes referred to as pulsus tardus et parvus. There may also be a noticeable delay between the first heart sound (on auscultation) and the corresponding pulse in the carotid artery (so-called 'apical-carotid delay'). Similarly, there may be a delay between the appearance of each pulse in the brachial artery (in the arm) and the radial artery (in the wrist).

An easily heard systolic, crescendo-decrescendo (i.e. 'ejection') murmur is heard loudest at the upper right sternal border, and radiates to the carotid arteries bilaterally. The murmur increases with squatting, decreases with standing and isometric muscular contraction, which helps distinguish it from hypertrophic obstructive cardiomyopathy (HOCM). The murmur is louder during expiration, but is also easily heard during inspiration. The more severe the degree of the stenosis, the later the peak occurs in the crescendo-decrescendo of the murmur.

The 2nd heart sound tends to become softer as the aortic stenosis becomes more severe. This is a result of the increasing calcification of the valve preventing it from "snapping" shut and producing a sharp, loud sound. Due to increases in left ventricular pressure from the stenotic aortic valve, over time the ventricle may hypertrophy, resulting in a diastolic dysfunction. As a result, one may hear a 4th heart sound due to the stiff ventricle. With continued increases in ventricular pressure, dilatation of the ventricle will occur, and a 3rd heart sound may manifest.

Finally, aortic stenosis often co-exists with some degree of aortic insufficiency. Hence, the physical exam in aortic stenosis may also reveal signs of the latter, for example an early diastolic decrescendo murmur. Indeed, when both valve abnormalities are present, the expected findings of either may be modified or may not even be present. Rather, new signs emerge which reflect the presence of simultaneous aortic stenosis and insufficiency, e.g. pulsus bisferiens.

According to a meta analysis, the most useful findings for ruling in aortic stenosis in the clinical setting were slow rate of rise of the carotid pulse(positive likelihood ratio ranged 2.8-130 across studies), mid to late peak intensity of the murmur(positive likelihood ratio, 8.0-101), and decreased intensity of the second heart sound(positive likelihood ratio, 3.1-50) [2].

Murmur in Aortic Stenosis

Murmur in Aortic Stenosis <youtube v=O4bFK3CGLh8/>

Signs of Aortic Stenosis

  • Aortic stenosis murmer. It is mid-systolic ejection murmur that is low-pitched and rough, it is loudest at the base of the heart and transmitted upward along the carotid arteries. The murmer is at least grade III or IV in most patients with severe obstruction, it may be soft in patients with mild degrees of obstruction or in those with heart failure, in whom stroke volume is reduced.
  • Systolic thrill in the same location of murmur.
  • Early systolic ejection murmer (the opening snap of the aortic valve).
  • Paradoxical splitting of S2 from prolongation of LV systole.
  • S4 may be audible at the apex.
  • S3 generally occurs due to left ventricular dilatation.

Signs associated with peripheral edema include:

  • A slow-rising, small volume carotid pulse.
  • Narrowed pulse pressure.
  • Sustained, thrusting apex beat which is usually not displaced unless the stenosis is severe.

Severity of Onset Summary

Severity of Aortic Stenosis
Severity mild moderate severe
Valve area 2.0 - 1.5 1- 1.5 <1
peak velocity (m/s) 2 -3 3-4 >4
Peak gradient (mmHg) <35 35-65 >65
Mean gradient (mmHg) <20 20-40 >40

References

  1. http://jama.ama-assn.org/cgi/content/abstract/277/7/564
  2. Etchells E, Bell C, Robb K (1997). "Does this patient have an abnormal systolic murmur?". JAMA. 277 (7): 564–71. PMID 9032164.


Template:WH Template:WS