Adiposogenital dystrophy causes: Difference between revisions

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{{Adiposogenital dystrophy}}
{{Adiposogenital Dystrophy}}


{{CMG}} [[Ogechukwu Hannah Nnabude, MD]]
{{CMG}} [[Ogechukwu Hannah Nnabude, MD]]


==Overview==
==Overview==
There are various types of lesion that can lead to adiposogenital dystrophy, all of which result in an injury of a part of the arcuate nucleus and ventromedial nuclei of the [[hypothalamus]].
There are various types of lesion that can lead to adiposogenital dystrophy, most of which result from an injury of a part of the arcuate nucleus and ventromedial nuclei of the [[hypothalamus]]. Deep brain stimulation has also been seen to produce similar symptoms in patients with Parkinson's disease.


==Causes==
==Causes==
Common causes of adiposogenital dystrophy is [[craniopharyngioma]], inflammation due to infections such as [[tuberculosis]], and [[encephalitis]].{{cite web |url=https://rarediseases.org/rare-diseases/froelichs-syndrome/ |title=Froelich Syndrome - NORD (National Organization for Rare Disorders) |format= |work= |accessdate=}}
*The most common cause of [[adiposogenital dystrophy]] are tumors affecting the hypothalamus or pituitary. In children, the most common tumor that would cause [[adiposogenital dystrophy is a [[craniopharyngioma]], a tumor arising from the [[Ratke's pouch]].
*Inflammation due to infections such as [[tuberculosis]], and [[encephalitis]] <ref name="pmid30937075">{{cite journal| author=Aulakh R, Chopra S| title=Pediatric Tubercular Meningitis: A Review. | journal=J Pediatr Neurosci | year= 2018 | volume= 13 | issue= 4 | pages= 373-382 | pmid=30937075 | doi=10.4103/JPN.JPN_78_18 | pmc=6413593 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=30937075  }} </ref> <ref>Babinski-fröhlich syndrome. Bissonnette B, & Luginbuehl I, & Marciniak B, & Dalens B.J.(Eds.), (2006). Syndromes: Rapid Recognition and Perioperative Implications. McGraw Hill. https://accessanesthesiology.mhmedical.com/content.aspx?bookid=852&sectionid=49517244</ref>
 
*Neurosarcoidosis <ref name="pmid12638724">{{cite journal| author=Molina A, Mañá J, Villabona C, Fernández-Castañer M, Soler J| title=Hypothalamic-pituitary sarcoidosis with hypopituitarism. Long-term remission with methylprednisolone pulse therapy. | journal=Pituitary | year= 2002 | volume= 5 | issue= 1 | pages= 33-6 | pmid=12638724 | doi=10.1023/a:1022153401880 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=12638724  }} </ref> <ref>Belojevic G, Maric-Zivkovic J (2005) Sarcoidosis and obesity. Med Pregl 58(Suppl 1):44–45 Medline</ref>
 
*Deep brain stimulation <ref name="pmid15878586">{{cite journal| author=Tuite PJ, Maxwell RE, Ikramuddin S, Kotz CM, Kotzd CM, Billington CJ | display-authors=etal| title=Weight and body mass index in Parkinson's disease patients after deep brain stimulation surgery. | journal=Parkinsonism Relat Disord | year= 2005 | volume= 11 | issue= 4 | pages= 247-52 | pmid=15878586 | doi=10.1016/j.parkreldis.2005.01.006 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=15878586  }} </ref> <ref>Romito LM, Scerrati M, Contarino MF, Iacoangeli M, Bentivoglio AR, Albanese A (2003) Bilateral high frequency subthalamic stimulation in Parkinson’s disease: long-term neurological follow-up. J Neurosurg Sci 47:119–128 Medline</ref>. Some patients were observed to have an increase in weight of up to 20kg<ref name="pmid17535833">{{cite journal| author=Montaurier C, Morio B, Bannier S, Derost P, Arnaud P, Brandolini-Bunlon M | display-authors=etal| title=Mechanisms of body weight gain in patients with Parkinson's disease after subthalamic stimulation. | journal=Brain | year= 2007 | volume= 130 | issue= Pt 7 | pages= 1808-18 | pmid=17535833 | doi=10.1093/brain/awm113 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=17535833  }} </ref>.
 
*Other causes of adiposogenital dystrophy include Friedreich ataxia, demyelinating diseases, and microcephaly<ref>Babinski-fröhlich syndrome. Bissonnette B, & Luginbuehl I, & Marciniak B, & Dalens B.J.(Eds.), (2006). Syndromes: Rapid Recognition and Perioperative Implications. McGraw Hill. https://accessanesthesiology.mhmedical.com/content.aspx?bookid=852&sectionid=49517244</ref>
 
 
{{reflist|2}}
{{reflist|2}}
==References==
==References==

Revision as of 02:28, 26 October 2021

Template:Adiposogenital Dystrophy

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Ogechukwu Hannah Nnabude, MD

Overview

There are various types of lesion that can lead to adiposogenital dystrophy, most of which result from an injury of a part of the arcuate nucleus and ventromedial nuclei of the hypothalamus. Deep brain stimulation has also been seen to produce similar symptoms in patients with Parkinson's disease.

Causes

  • Deep brain stimulation [5] [6]. Some patients were observed to have an increase in weight of up to 20kg[7].
  • Other causes of adiposogenital dystrophy include Friedreich ataxia, demyelinating diseases, and microcephaly[8]


  1. ↑ Aulakh R, Chopra S (2018). "Pediatric Tubercular Meningitis: A Review". J Pediatr Neurosci. 13 (4): 373–382. doi:10.4103/JPN.JPN_78_18. PMC 6413593. PMID 30937075.
  2. ↑ Babinski-fröhlich syndrome. Bissonnette B, & Luginbuehl I, & Marciniak B, & Dalens B.J.(Eds.), (2006). Syndromes: Rapid Recognition and Perioperative Implications. McGraw Hill. https://accessanesthesiology.mhmedical.com/content.aspx?bookid=852&sectionid=49517244
  3. ↑ Molina A, Mañá J, Villabona C, Fernández-Castañer M, Soler J (2002). "Hypothalamic-pituitary sarcoidosis with hypopituitarism. Long-term remission with methylprednisolone pulse therapy". Pituitary. 5 (1): 33–6. doi:10.1023/a:1022153401880. PMID 12638724.
  4. ↑ Belojevic G, Maric-Zivkovic J (2005) Sarcoidosis and obesity. Med Pregl 58(Suppl 1):44–45 Medline
  5. ↑ Tuite PJ, Maxwell RE, Ikramuddin S, Kotz CM, Kotzd CM, Billington CJ; et al. (2005). "Weight and body mass index in Parkinson's disease patients after deep brain stimulation surgery". Parkinsonism Relat Disord. 11 (4): 247–52. doi:10.1016/j.parkreldis.2005.01.006. PMID 15878586.
  6. ↑ Romito LM, Scerrati M, Contarino MF, Iacoangeli M, Bentivoglio AR, Albanese A (2003) Bilateral high frequency subthalamic stimulation in Parkinson’s disease: long-term neurological follow-up. J Neurosurg Sci 47:119–128 Medline
  7. ↑ Montaurier C, Morio B, Bannier S, Derost P, Arnaud P, Brandolini-Bunlon M; et al. (2007). "Mechanisms of body weight gain in patients with Parkinson's disease after subthalamic stimulation". Brain. 130 (Pt 7): 1808–18. doi:10.1093/brain/awm113. PMID 17535833.
  8. ↑ Babinski-fröhlich syndrome. Bissonnette B, & Luginbuehl I, & Marciniak B, & Dalens B.J.(Eds.), (2006). Syndromes: Rapid Recognition and Perioperative Implications. McGraw Hill. https://accessanesthesiology.mhmedical.com/content.aspx?bookid=852&sectionid=49517244

References

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