Acute bronchitis pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
The pathologic process starts with the inoculation of tracheobranchial epithelium with invading organism which leads to inflammation, thickening and increased mucos production. | |||
==Pathophysiology== | ==Pathophysiology== | ||
*The causative agent, either viruses or bacteria transmitted via airways to the large and medium size airways tract. | *The causative agent, either viruses or bacteria transmitted via airways to the large and medium size airways tract<ref name="pmid11119400">{{cite journal |vauthors=Gonzales R, Sande MA |title=Uncomplicated acute bronchitis |journal=Ann. Intern. Med. |volume=133 |issue=12 |pages=981–91 |year=2000 |pmid=11119400 |doi= |url=}}</ref>. | ||
*Following transmission, the bug starts to inoculate the tracheobronchial epithelium. | *Following transmission, the bug starts to inoculate the tracheobronchial epithelium<ref name="pmid11119400">{{cite journal |vauthors=Gonzales R, Sande MA |title=Uncomplicated acute bronchitis |journal=Ann. Intern. Med. |volume=133 |issue=12 |pages=981–91 |year=2000 |pmid=11119400 |doi= |url=}}</ref>. | ||
*This process leads to inflammation, thickening, and increased mucus production in the airways as shown | *This process leads to inflammation, thickening, and increased mucus production in the airways compared to normal bronchi as shown below<ref name="pmid11119400">{{cite journal |vauthors=Gonzales R, Sande MA |title=Uncomplicated acute bronchitis |journal=Ann. Intern. Med. |volume=133 |issue=12 |pages=981–91 |year=2000 |pmid=11119400 |doi= |url=}}</ref>. | ||
<gallery> | <gallery> | ||
Image:normal bronchi.jpg| | Image:normal bronchi.jpg|Normal Bronchi | ||
Image:acute bronchitis.jpg| | Image:acute bronchitis.jpg|Inflamed Bronchi | ||
</gallery> | </gallery> | ||
*On microscopic analysis,epithelial-cell desquamation and denuding of the airway to the level of the basement membrane in association with the presence of a lymphocytic cellular infiltrate have been demonstrated<ref name="pmid13782910">{{cite journal |vauthors=WALSH JJ, DIETLEIN LF, LOW FN, BURCH GE, MOGABGAB WJ |title=Bronchotracheal response in human influenza. Type A, Asian strain, as studied by light and electron microscopic examination of bronchoscopic biopsies |journal=Arch. Intern. Med. |volume=108 |issue= |pages=376–88 |year=1961 |pmid=13782910 |doi= |url=}}</ref>. | |||
==References== | ==References== | ||
Revision as of 17:35, 8 September 2016
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Seyedmahdi Pahlavani, M.D. [2]
Overview
The pathologic process starts with the inoculation of tracheobranchial epithelium with invading organism which leads to inflammation, thickening and increased mucos production.
Pathophysiology
- The causative agent, either viruses or bacteria transmitted via airways to the large and medium size airways tract[1].
- Following transmission, the bug starts to inoculate the tracheobronchial epithelium[1].
- This process leads to inflammation, thickening, and increased mucus production in the airways compared to normal bronchi as shown below[1].
-
Normal Bronchi
-
Inflamed Bronchi
- On microscopic analysis,epithelial-cell desquamation and denuding of the airway to the level of the basement membrane in association with the presence of a lymphocytic cellular infiltrate have been demonstrated[2].
References
- ↑ 1.0 1.1 1.2 Gonzales R, Sande MA (2000). "Uncomplicated acute bronchitis". Ann. Intern. Med. 133 (12): 981–91. PMID 11119400.
- ↑ WALSH JJ, DIETLEIN LF, LOW FN, BURCH GE, MOGABGAB WJ (1961). "Bronchotracheal response in human influenza. Type A, Asian strain, as studied by light and electron microscopic examination of bronchoscopic biopsies". Arch. Intern. Med. 108: 376–88. PMID 13782910.