Benign paroxysmal positional vertigo overview

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Benign paroxysmal positional vertigo Microchapters

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Overview

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Pathophysiology

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Differentiating Benign Paroxysmal Positional Vertigo from other Diseases

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Fahimeh Shojaei, M.D.

Overview

Historical Perspective

BPPV was first dicribed by Adler and Barany, who described it as a problem in the otolith organs. In 1952, Margaret Dix and Charles Hallpike named it positional nystagmus of the benign positional type. They noted nystagmus and vertigo with different headmovements. Hallpike also defined it as a peripheral problem rather than central (brain) problem. In 1962 Harold Schuknecht described theory of detached utricular otoconia (cupulolithiasis). Hall et al and Epley described the theory of free floating particle (canalithiasis). The first treatment strategy suggested for BPPV treatment was cawthorne's exercise (repeatitive head movement which cause vertigo in order to reach central adaption). The newest treatment strategy is to perform Dix Hallpike test to diagnos and induce the vertigo and then performing CRP (Epley) maneuver.

Classification

Benign paroxysmal positional vertigo may be classified according to which semicircular canal the otoconia have migrated to into 3 subtypes including posterior semicircular canal BPPV, lateral semicircular canal BPPV, and superior (anterior) semicircular canal BPPV.

Pathophysiology

Causes

Differentiating benign paroxysmal positional vertigo from Other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications, and Prognosis

Diagnosis

Diagnostic Study of Choice

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

X-ray

Echocardiography and Ultrasound

CT scan

MRI

Other Imaging Findings

Other Diagnostic Studies

Treatment

Medical Therapy

Interventions

Surgery

Primary Prevention

Secondary Prevention

References


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