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==Example== | ==Example== | ||
Biochemical stress and neurohumoral factors have been implicated as triggers of cardiac hypertrophy via multiple intracellular pathways involved in protein synthesis and stability.<ref name="Hill-2008">{{Cite journal | last1 = Hill | first1 = JA. | last2 = Olson | first2 = EN. | title = Cardiac plasticity. | journal = N Engl J Med | volume = 358 | issue = 13 | pages = 1370-80 | month = Mar | year = 2008 | doi = 10.1056/NEJMra072139 | PMID = 18367740 }}</ref> | Biochemical stress and neurohumoral factors have been implicated as triggers of cardiac hypertrophy via multiple intracellular pathways involved in protein synthesis and stability.<ref name="Hill-2008">{{Cite journal | last1 = Hill | first1 = JA. | last2 = Olson | first2 = EN. | title = Cardiac plasticity. | journal = N Engl J Med | volume = 358 | issue = 13 | pages = 1370-80 | month = Mar | year = 2008 | doi = 10.1056/NEJMra072139 | PMID = 18367740 }}</ref> | ||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
Revision as of 11:04, 28 November 2013
Example
Biochemical stress and neurohumoral factors have been implicated as triggers of cardiac hypertrophy via multiple intracellular pathways involved in protein synthesis and stability.[1]