Sandbox:Cherry: Difference between revisions
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==Overview== | ==Overview== | ||
Pathophysiology | ==Pathophysiology== | ||
Diagnosis | ==Diagnosis== | ||
IBS is a [[diagnosis]] of | IBS is a [[diagnosis]] of <ref name="urlapi.isoa.org">{{cite web |url=https://api.isoa.org/docs/plans/2018/ISO-J_Exchange_Visitors_Plan_2017_2018.pdf |title=api.isoa.org |format= |work= |accessdate=}}</ref> | ||
Visceral hypersensitivity may have been utilized as a clinical marker of IBS and could be represented by signs and symptoms of urgency for bowel movements, bloating and abdominal pain. The mechanisms that result in persistent visceral hypersensitivity in patients who have IBS are unclear. nonetheless, various working models might be considered | Visceral hypersensitivity may have been utilized as a clinical marker of IBS and could be represented by signs and symptoms of urgency for bowel movements, bloating and abdominal pain. The mechanisms that result in persistent visceral hypersensitivity in patients who have IBS are unclear. nonetheless, various working models might be considered | ||
Revision as of 13:47, 18 October 2017
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Sudarshana Datta, MD [2]
Overview
Pathophysiology
Diagnosis
Visceral hypersensitivity may have been utilized as a clinical marker of IBS and could be represented by signs and symptoms of urgency for bowel movements, bloating and abdominal pain. The mechanisms that result in persistent visceral hypersensitivity in patients who have IBS are unclear. nonetheless, various working models might be considered
- ↑ "api.isoa.org" (PDF).