Cardiogenic shock resident survival guide: Difference between revisions
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{{Family tree | | | | | | | | | B01 | | | |B01= '''ABCD''' <br> Airway / O2 / 2 wide bore IV access / 12-lead ECG / focused H&P / CXR <br> PA / Arterial line monitoring }} | {{Family tree | | | | | | | | | B01 | | | |B01= '''ABCD''' <br> Airway / O2 / 2 wide bore IV access / 12-lead ECG / focused H&P / CXR <br> PA / Arterial line monitoring }} | ||
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{{Family tree | | | | | | | | | B02 |-|-| B03 | |B02= Hemodynamic optimization | | {{Family tree | | | | | | | | | B02 |-|-|B03 | |B02= Hemodynamic optimization |boxstyle_B03=BACKGROUND:SALMON |B03= '''Do not''' give <br> '''β Blockers''' <br> '''Ca Channel antagonist'''}} | ||
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Revision as of 22:34, 25 December 2013
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ahmed Zaghw, M.D. [2]
Definition
It is a state of end-organ hypoperfusion due to myocardial dysfunction, associated with hemodynamic changes for 30 mins or more which is not due to hypovolemia and not responsive to fluids alone:
- SBP < 80 mmHg or MAP < 30 mmHg than baseline
- Cardiac index ( CI ) < 1.8 L.Min-1·M-2 without vasopressors support or CI 2.0 to 2.2 L.Min-1·M-2 with vasopressors support.
- Elevated filling pressures LVEDP > 18 mmHg, RVEDP > 10 mmHg
Causes cardiogenic shock
- Pump problems: LV failure mainly in anterior wall MI still is the most common cause for CS and cardiomyopathy post-MI is the second most common.[1]
- Mechanical problems: Ventricular septal rupture, contained free wall rupture, and papillary muscle rupture. Mechanical problems are strongly suspected in non anterior MI. However cardiac tamponade, tension pneumothorax) and aortic stenosis are recently considered as obstructive shock.
- Electrical problems: is the least commonly cause for CS and most commonly occurs due toVF or VT and AF.
Prognosis
- Cardiogenic shock occurs in 8% of hospitalized STEMI patient with a mortality rate of 50-60% within 30 days.
- The only way to prevent CS is an early reperfusion therapy for MI
Management
Shock | |||||||||||||||||||||||||||||||||||
ABCD Airway / O2 / 2 wide bore IV access / 12-lead ECG / focused H&P / CXR PA / Arterial line monitoring | |||||||||||||||||||||||||||||||||||
Hemodynamic optimization | Do not give β Blockers Ca Channel antagonist | ||||||||||||||||||||||||||||||||||
ECG evidence of STEMI † | ECG inconclusive | ECG: -ve Clinical history of HF | |||||||||||||||||||||||||||||||||
STEMI | Echocardiography rule out Acute valvular lesions | Heart failure | |||||||||||||||||||||||||||||||||
Echocardiography to associated valvular causes †† | Surgical correction | Treatment of heart failure | |||||||||||||||||||||||||||||||||
PCI capable center | |||||||||||||||||||||||||||||||||||
PCI Non-capable center | |||||||||||||||||||||||||||||||||||
Urgent PCI | |||||||||||||||||||||||||||||||||||
Transfer to PCI center < 120 min | Transfer to PCI center > 120 min | ||||||||||||||||||||||||||||||||||
Urgent Transfer to PCI | Thrombolytics | ||||||||||||||||||||||||||||||||||
Get stable | Still Non stable * Hypotension * ECG evidence | ||||||||||||||||||||||||||||||||||
Transfer to PCI center within 3-24 hrs after Thrombolytics | Urgent Transfer to PCI center | ||||||||||||||||||||||||||||||||||
† New ST elevation at the J point in at least 2 contiguous leads of 2 mm in men or 1.5 mm in women in leads V2-V3 and/or of 1 mm in other contiguous chest leads or the limb leads.
†† Early echocardiography should be done before PCI as long as the patient is not crashing, as it may change the treatment course.
Do's
- Cardiogenic shock occurs in 5-8% of hospitalized STEMI patient.[2]
- Cardiac Echocardiography (Transthoracic) is helpful to rule out mechanical problems when the initial ECG findings are not conclusive or when the cardiogenic shock occurs with the first non anterior MI.[3]
- Echocardiography should be performed early before PCI unless the diagnosis is extensive anterior MI and the patient is undergoing prompt percutaneous coronary intervention (PCI).[3]
- Transfer the STEMI patients with cardiogenic shock to PCI irrespective to time delay from time of presentation.
Don'ts
- Do not routinely use an intraaortic balloon pump (IABP)in all MI patients complicated by cardiogenic shock (CS) whom are planned to have primary percutaneous coronary intervention (PCI) is attempted or performed or in whom fibrinolytic therapy is administered.
References
- ↑ Reynolds, HR.; Hochman, JS. (2008). "Cardiogenic shock: current concepts and improving outcomes". Circulation. 117 (5): 686–97. doi:10.1161/CIRCULATIONAHA.106.613596. PMID 18250279. Unknown parameter
|month=
ignored (help) - ↑ Reynolds, HR.; Hochman, JS. (2008). "Cardiogenic shock: current concepts and improving outcomes". Circulation. 117 (5): 686–97. doi:10.1161/CIRCULATIONAHA.106.613596. PMID 18250279. Unknown parameter
|month=
ignored (help) - ↑ 3.0 3.1 Reynolds, HR.; Hochman, JS. (2008). "Cardiogenic shock: current concepts and improving outcomes". Circulation. 117 (5): 686–97. doi:10.1161/CIRCULATIONAHA.106.613596. PMID 18250279. Unknown parameter
|month=
ignored (help)