Tricuspid regurgitation risk factors: Difference between revisions
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{{CMG}}; {{AE}} | {{CMG}}; {{AE}} | ||
==Overview== | ==Overview== | ||
Common risk factors in the development of [[tricuspid regurgitation]] include infections, endocarditis, ebstein anomaly, tricuspid valve prolapse, carcinoid, papillary muscle dysfunction and medications. Most cases of significant [[tricuspid regurgitation]] are due to [[tricuspid]] annular dilation and leaflet tethering secondary to right [[ventricular]] remodeling from volume and/or pressure overload. | Common risk factors in the development of [[tricuspid regurgitation]] include [[Infection|infections]], [[endocarditis]], [[ebstein anomaly]], [[tricuspid valve prolapse]], [[carcinoid]], [[Papillary muscle dysfunction|papillary muscle]] dysfunction and [[medications]]. Most cases of significant [[tricuspid regurgitation]] are due to [[tricuspid]] annular dilation and leaflet tethering secondary to right [[ventricular]] remodeling from volume and/or pressure overload. | ||
==Risk Factors== | ==Risk Factors== | ||
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*Common risk factors in the development of [[tricuspid regurgitation]] include:<ref name="pmid17400120">{{cite journal| author=Mutlak D, Lessick J, Reisner SA, Aronson D, Dabbah S, Agmon Y| title=Echocardiography-based spectrum of severe tricuspid regurgitation: the frequency of apparently idiopathic tricuspid regurgitation. | journal=J Am Soc Echocardiogr | year= 2007 | volume= 20 | issue= 4 | pages= 405-8 | pmid=17400120 | doi=10.1016/j.echo.2006.09.013 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=17400120 }}</ref> | *Common risk factors in the development of [[tricuspid regurgitation]] include:<ref name="pmid17400120">{{cite journal| author=Mutlak D, Lessick J, Reisner SA, Aronson D, Dabbah S, Agmon Y| title=Echocardiography-based spectrum of severe tricuspid regurgitation: the frequency of apparently idiopathic tricuspid regurgitation. | journal=J Am Soc Echocardiogr | year= 2007 | volume= 20 | issue= 4 | pages= 405-8 | pmid=17400120 | doi=10.1016/j.echo.2006.09.013 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=17400120 }}</ref> | ||
**Rheumatic heart disease | **[[Rheumatic heart disease]] | ||
**Endocarditis is a common risk factor in developing tricuspid regurgitation due to the following: | **[[Endocarditis]] is a common risk factor in developing tricuspid regurgitation due to the following: | ||
***Alcoholism | ***[[Alcoholism]] | ||
***Intravenous drug use | ***[[Intravenous drug use]] | ||
***Cardiac tumours | ***[[Cardiac|Cardiac tumours]] | ||
***Indwelling catheters which might get infected | ***Indwelling [[Catheter|catheters]] which might get infected | ||
***Burns when extensive | ***[[Burn|Burns]] when extensive | ||
**Tricuspid valve prolapse | **[[Tricuspid valve prolapse]] | ||
**Papillary muscle dysfunction | **[[Papillary muscle dysfunction]] | ||
**RV dilatation | **RV dilatation | ||
**Trauma which includes: | **[[Trauma]] which includes: | ||
***Right ventricular [[endomyocardial]] [[biopsy]] | ***[[Right ventricle|Right ventricular]] [[endomyocardial]] [[biopsy]] | ||
***Blunt chest wall trauma like stab wounds and projectiles | ***Blunt chest wall [[trauma]] like stab wounds and projectiles | ||
===Less Common Risk Factors=== | ===Less Common Risk Factors=== | ||
*Less common risk factors in the development of [[tricuspid regurgitation]] include: | *Less common [[Risk factor|risk factors]] in the development of [[tricuspid regurgitation]] include: | ||
**Pregnant mothers who are using lithium are at a risk for developing ebstein anomaly | **Pregnant mothers who are using lithium are at a risk for developing ebstein anomaly | ||
**Connective-tissue diseases | **Connective-tissue diseases | ||
Revision as of 15:16, 12 April 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
Common risk factors in the development of tricuspid regurgitation include infections, endocarditis, ebstein anomaly, tricuspid valve prolapse, carcinoid, papillary muscle dysfunction and medications. Most cases of significant tricuspid regurgitation are due to tricuspid annular dilation and leaflet tethering secondary to right ventricular remodeling from volume and/or pressure overload.
Risk Factors
Common Risk Factors
- Common risk factors in the development of tricuspid regurgitation include:[1]
- Rheumatic heart disease
- Endocarditis is a common risk factor in developing tricuspid regurgitation due to the following:
- Alcoholism
- Intravenous drug use
- Cardiac tumours
- Indwelling catheters which might get infected
- Burns when extensive
- Tricuspid valve prolapse
- Papillary muscle dysfunction
- RV dilatation
- Trauma which includes:
- Right ventricular endomyocardial biopsy
- Blunt chest wall trauma like stab wounds and projectiles
Less Common Risk Factors
- Less common risk factors in the development of tricuspid regurgitation include:
References
- ↑ Mutlak D, Lessick J, Reisner SA, Aronson D, Dabbah S, Agmon Y (2007). "Echocardiography-based spectrum of severe tricuspid regurgitation: the frequency of apparently idiopathic tricuspid regurgitation". J Am Soc Echocardiogr. 20 (4): 405–8. doi:10.1016/j.echo.2006.09.013. PMID 17400120.
- ↑ Baseman DG, O'Suilleabhain PE, Reimold SC, Laskar SR, Baseman JG, Dewey RB (2004). "Pergolide use in Parkinson disease is associated with cardiac valve regurgitation". Neurology. 63 (2): 301–4. doi:10.1212/01.wnl.0000129842.49926.07. PMID 15277624.
- ↑ Pritchett AM, Morrison JF, Edwards WD, Schaff HV, Connolly HM, Espinosa RE (2002). "Valvular heart disease in patients taking pergolide". Mayo Clin Proc. 77 (12): 1280–6. doi:10.4065/77.12.1280. PMID 12479512.