Septic shock: Difference between revisions

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Revision as of 02:06, 17 September 2012

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]

@@ Line 13: / Line 13: @@
 ==[[Sepsis causes|Causes]]==
 ==[[Sepsis differential diagnosis|Differentiating Sepsis from other Diseases]]==
-===Types===
-A subclass of [[distributive shock]], shock refers specifically to decreased tissue [[perfusion]] resulting in end-organ dysfunction.  [[Cytokines]] TNFα, IL-1β, IL-6 released in a large scale inflammatory response results in massive [[vasodilation]], increased [[capillary]] permeability, decreased systemic vascular resistance, and [[hypotension]].  Hypotension reduces tissue perfusion pressure and thus tissue [[Hypoxia (medical)|hypoxia]] ensues.  Finally, in an attempt to offset decreased [[blood pressure]], [[ventricular]] dilatation and [[myocardium|myocardial]] dysfunction will occur.
 ==[[Sepsis medical therapy|Medical therapy]]==