Sandbox : anmol: Difference between revisions

Latest revision as of 17:32, 14 January 2019

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Anmol Pitliya, M.B.B.S. M.D.[2]

@@ Line 1: / Line 1: @@
 __NOTOC__
-{{Hyperparathyroidism}}
 {{CMG}}; {{AE}} {{Anmol}}
+==Tables==
+{| class="wikitable"
+|+
+!Diagnosis
+!Lab findings
+!
+!
+|-
+!
+!
+!
+!
+|-
+|
+|
+|
+|
+|-
+|
+|
+|
+|
+|-
+|
+|
+|
+|
+|}
-=Pathogenesis=
-*GSD type 1 results due to defects in the function of microsomal enzyme glucose-6-phosphatase (G6Pase).
-*G6Pase catalyzes the conversion of glucose-6-phosphate to glucose during glycogenolysis and gluconeogenesis.
-*G6Pase is primarily expressed in expressed primarily in the gluconeogenic the liver and kidney. It is also expressed to a lesser extent in the intestine and pancreas.
-*80% Cases of GSD 1 are of GSD type 1a.
 ==References==
 {{reflist|2}}