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{{Hyperparathyroidism}}


{{CMG}}; {{AE}} {{Anmol}}
{{CMG}}; {{AE}} {{Anmol}}


== Classification ==
==Tables==
{| class="wikitable"
{| class="wikitable"
! colspan="4" |Classification of hyperparathyridism
|+
!Diagnosis
!Lab findings
!
!
|-
|-
|Features
!
|'''Primary hyperparathyroidism'''
!
|'''Secondary hyperparathyroidism'''
!
|'''Tertiary hyperparathyroidism'''
!
|-
|-
|Pathology
|
|Hyperfunction of parathyroid cells due to hyperplasia, adenoma or carcinoma.
|
|Physiological stimulation of parathyroid in response to hypocalcaemia.
|
|Following long term physiological stimulation leading to hyperplasia.
|
|-
|-
|Cause
|
|
|
|
|
|
|
|-
|-
|Associations
|
|May be associated with multiple endocrine neoplasia.
|
|Usually due to chronic renal failure or other causes of Vitamin D deficiency.
|
|Seen in chronic renal failure.
|
|-
|Serum calcium
|High
|Low/Normal
|High
|-
|Serum phosphate
|Low/Normal
|High
|High
|-
|Management
|Usually surgery if symptomatic. Cincacalcet can be considered in those not fit for surgery.
|Treatment of underlying cause.
|Usually cinacalcet or surgery in those that don't respond.
|}
|}


=Epidemiology PE & DVT=
For causes of [[hypoparathyroidism]], [[Hypoparathyroidism causes|click here]].
==References==
==References==
{{reflist|2}}
{{reflist|2}}

Latest revision as of 17:32, 14 January 2019


Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Anmol Pitliya, M.B.B.S. M.D.[2]

Tables

Diagnosis Lab findings

References