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| {{Lyme disease}}
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| {{CMG}}
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| ==Overview==
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| Abnormal [[magnetic resonance imaging]] (MRI) findings are often seen in both early and late Lyme disease. Diffuse white matter pathology can disrupt these ubiquitous [[gray matter]] connections and could account for deficits in attention, memory, visuospatial ability, complex cognition, and emotional status. White matter disease may have a greater potential for recovery than gray matter disease, perhaps because neuronal loss is less common,
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| ==MRI==
| | {{CMG}}; {{AE}} {{Anmol}} |
| Abnormal [[magnetic resonance imaging]] (MRI) findings are often seen in both early and late Lyme disease. MRI scans of patients with neurologic Lyme disease may demonstrate punctated [[white matter]] [[lesions]] on T2-weighted images, similar to those seen in [[demyelinating]] or inflammatory disorders such as [[multiple sclerosis]], [[systemic lupus erythematosus]] (SLE), or cerebrovascular disease.<ref>{{cite conference | last = Fallon | first = BA | title = Review of Lyme Neuroborreliosis | conference = 3th International Scientific Conference on Lyme Disease and other Tick-borne Disorders | url = http://www.medscape.com/viewarticle/412987 | year = 2000}}</ref> [[Cerebral atrophy]] and brainstem [[neoplasm]] has been indicated with Lyme infection as well.<ref>{{cite journal |author=Kalina P, Decker A, Kornel E, Halperin JJ |title=Lyme disease of the brainstem |journal=Neuroradiology |volume=47 |issue=12 |pages=903-7 |year=2005 |pmid=16158278 |doi=10.1007/s00234-005-1440-2}}</ref>
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| Diffuse white matter pathology can disrupt these ubiquitous [[gray matter]] connections and could account for deficits in attention, memory, visuospatial ability, complex cognition, and emotional status. White matter disease may have a greater potential for recovery than gray matter disease, perhaps because neuronal loss is less common. Spontaneous [[remission (medicine)|remission]] can occur in [[multiple sclerosis]], and resolution of MRI white matter hyper-intensities, after antibiotic treatment, has been observed in Lyme disease.<ref>{{cite journal |author=Fallon BA, Keilp J, Prohovnik I, Heertum RV, Mann JJ |title=Regional cerebral blood flow and cognitive deficits in chronic lyme disease |journal=The Journal of neuropsychiatry and clinical neurosciences |volume=15 |issue=3 |pages=326-32 |year=2003 |pmid=12928508}}</ref>
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| ==References== | | ==References== |
| {{reflist|2}} | | {{reflist|2}} |
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| [[Category:Needs overview]]
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| [[Category:Bacterial diseases]]
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| [[Category:Insect-borne diseases]]
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| [[Category:Lyme disease]]
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| [[Category:Zoonoses]]
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| [[Category:Spirochaetes]]
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| [[Category:Disease]]
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| [[Category:Infectious disease]]
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| [[Category:Dermatology]]
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| [[Category:Emergency medicine]]
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| [[Category:Intensive care medicine]]
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