Heart valve

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Gray's Fig. 498 - Section of the heart showing the ventricular septum.

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]


Overview

In anatomy, the heart valves are valves in the heart that maintain the unidirectional flow of blood by opening and closing depending on the difference in pressure on each side. The mechanical equivalent of the heart valves would be the reed valves.

There are four valves of the heart (not counting the valve of the coronary sinus and valve of the inferior vena cava):

  • The two atrioventricular (AV) valves ensure blood flows from the atria to the ventricles, and not the other way.
  • The two semilunar (SL) valves are present in the arteries leaving the heart, and they prevent blood flowing back from the arteries into the ventricles.

The sound of the heart valves shutting causes the heart sounds.

Atrioventricular valves

The atrioventricular valves are large, multicusped valves that prevent backflow from the ventricles into the atria during systole. They are anchored to the wall of the ventricle by chordae tendinae, that prevent the valve from inverting.

The chordae tendinae are attached to papillary muscles that cause tension to better hold the valve. Together, the papillary muscles and the chordae tendinae are known as the subvalvular apparatus. The function of the subvalvular apparatus is to keep the valves from prolapsing into the atria when they close. The subvalvular apparatus have no effect on the opening and closure of the valves, however. This is caused entirely by the pressure gradient across the valve.

Mitral valve

Also known as the bicuspid valve, the mitral valve gets its name from the resemblance to a bishop's mitre (a type of hat). It prevents blood flowing from the left ventricle into the left atrium. It is on the left side of the heart and has two leaflets.

A common complication of rheumatic fever is thickening and stenosis of the mitral valve.

Tricuspid valve

The tricuspid valve is on the right side of the heart, between the right atrium and the right ventricle.

Semilunar valves

The semilunar vlalves are positioned on the pulmonary artery and the aorta. These valves do not have chordae tendinae, but are more similar to valves in veins.

Aortic valve

The aortic valve lies between the left ventricle and the aorta. The aortic valve has three cusps. During ventricular systole, pressure rises in the left ventricle. When the pressure in the left ventricle rises above the pressure in the aorta, the aortic valve opens, allowing blood to exit the left ventricle into the aorta. When ventricular systole ends, pressure in the left ventricle rapidly drops. When the pressure in the left ventricle decreases, the aortic pressure forces the aortic valve to close. The closure of the aortic valve contributes the A2 component of the second heart sound (S2).

The most common congenital abnormality of the heart is the bicuspid aortic valve. In this condition, instead of three cusps, the aortic valve has two cusps. This condition is often undiagnosed until the person develops calcific aortic stenosis. Aortic stenosis occurs in this condition usually in patients in their 40s or 50s, an average of over 10 years earlier than in people with normal aortic valves.

Pulmonic valve

The pulmonic valve lies between the right ventricle and the pulmonary artery and also has three cusps.

Pathology of the valves

  • Endocarditis - inflammation of the inner layer of the endocardium.
  • Stenosis - a constricture of the heart valve, making it hard for blood to get through.
  • Insufficiency (also regurgitation or incompetence) - the inability of the heart valve to close properly, meaning some blood can flow the wrong way.

See also

3D reconstruction of the heart as viewed from the apex towards the valves, image flipped 180° relative to illustration above. Pulmonary valve not visible, leaflets of the tricuspid and aortic valves only partly visible. To the left two images in 2D from the same dataset, showing tricuspid and mitral valves (above) and aortal and mitral valve (below).

External links

Additional Reading

  • Moss and Adams' Heart Disease in Infants, Children, and Adolescents Hugh D. Allen, Arthur J. Moss, David J. Driscoll, Forrest H. Adams, Timothy F. Feltes, Robert E. Shaddy, 2007 ISBN 0781786843
  • Hurst's the Heart, Fuster V, 12th ed. 2008, ISBN 978-0-07-149928-6
  • Willerson JT, Cardiovascular Medicine, 3rd ed., 2007, ISBN 978-1-84628-188-4


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