Difference between revisions of "Adult bronchiolitis pathophysiology"

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{{Adult bronchiolitis}}
 
{{Adult bronchiolitis}}
 
{{CMG}};{{AE}}{{HM}}
 
{{CMG}};{{AE}}{{HM}}
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==Overview==
 +
The exact pathogenesis of [disease name] is not fully understood.
 +
 +
OR
 +
 +
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
 +
 +
OR
 +
 +
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
 +
 +
OR
 +
 +
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
 +
 +
OR
 +
 +
 +
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
 +
 +
OR
 +
 +
The progression to [disease name] usually involves the [molecular pathway].
 +
 +
OR
 +
 +
The pathophysiology of [disease/malignancy] depends on the histological subtype.
 +
 +
==Pathophysiology==
 +
 +
===Pathogenesis===
 +
*The exact pathogenesis of [disease name] is not fully understood.
 +
OR
 +
*It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
 +
*[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
 +
*Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
 +
*[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
 +
*The progression to [disease name] usually involves the [molecular pathway].
 +
*The pathophysiology of [disease/malignancy] depends on the histological subtype.
 +
 +
==Genetics==
 +
*[Disease name] is transmitted in [mode of genetic transmission] pattern.
 +
*Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
 +
*The development of [disease name] is the result of multiple genetic mutations.
 +
 +
==Associated Conditions==
 +
 +
==Gross Pathology==
 +
*On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
 +
 +
==Microscopic Pathology==
 +
*On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
  
 
==References==
 
==References==

Revision as of 21:20, 22 February 2018

Adult bronchiolitis Microchapters

Home

Patient Information

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Occupational lung disease from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Study of Choice

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

X Ray

CT

MRI

Echocardiography or Ultrasound

Other Imaging Findings

Other Diagnostic Studies

Treatment

Medical Therapy

Surgery

Primary Prevention

Secondary Prevention

Cost-Effectiveness of Therapy

Future or Investigational Therapies

Case Studies

Case #1

Adult bronchiolitis pathophysiology On the Web

Most recent articles

Most cited articles

Review articles

CME Programs

Powerpoint slides

Images

American Roentgen Ray Society Images of Adult bronchiolitis pathophysiology

All Images
X-rays
Echo & Ultrasound
CT Images
MRI

Ongoing Trials at Clinical Trials.gov

US National Guidelines Clearinghouse

NICE Guidance

FDA on Adult bronchiolitis pathophysiology

CDC on Adult bronchiolitis pathophysiology

Adult bronchiolitis pathophysiology in the news

Blogs on Adult bronchiolitis pathophysiology


Risk calculators and risk factors for Adult bronchiolitis pathophysiology

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Hadeel Maksoud M.D.[2]

Overview

The exact pathogenesis of [disease name] is not fully understood.

OR

It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].

OR

[Pathogen name] is usually transmitted via the [transmission route] route to the human host.

OR

Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.

OR


[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].

OR

The progression to [disease name] usually involves the [molecular pathway].

OR

The pathophysiology of [disease/malignancy] depends on the histological subtype.

Pathophysiology

Pathogenesis

  • The exact pathogenesis of [disease name] is not fully understood.

OR

  • It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
  • [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
  • Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
  • [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
  • The progression to [disease name] usually involves the [molecular pathway].
  • The pathophysiology of [disease/malignancy] depends on the histological subtype.

Genetics

  • [Disease name] is transmitted in [mode of genetic transmission] pattern.
  • Genes involved in the pathogenesis of [disease name] include [gene1], [gene2], and [gene3].
  • The development of [disease name] is the result of multiple genetic mutations.

Associated Conditions

Gross Pathology

  • On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

Microscopic Pathology

  • On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

References


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