17 alpha-hydroxylase deficiency risk factors: Difference between revisions
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__NOTOC__ | __NOTOC__ | ||
{{ | {{17 alpha-hydroxylase deficiency}} | ||
{{CMG}}; {{AE}} {{Ammu}} | {{CMG}}; {{AE}} {{Ammu}}, {{MJ}} | ||
==Overview== | ==Overview== | ||
The most potent risk factor | The most potent risk factor for the development of 17 alpha-hydroxylase deficiency is a positive [[family history]] of 17 alpha-hydroxylase deficiency. | ||
==Risk Factors== | ==Risk Factors== | ||
The most potent risk factor | The most potent risk factor for the development of 17 alpha-hydroxylase deficiency is a positive [[family history]] of 17 alpha-hydroxylase deficiency.<ref name="pmid28476231">{{cite journal |vauthors=Hannah-Shmouni F, Chen W, Merke DP |title=Genetics of Congenital Adrenal Hyperplasia |journal=Endocrinol. Metab. Clin. North Am. |volume=46 |issue=2 |pages=435–458 |year=2017 |pmid=28476231 |doi=10.1016/j.ecl.2017.01.008 |url=}}</ref> | ||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
Latest revision as of 12:07, 23 October 2017
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17 alpha-hydroxylase deficiency Microchapters |
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Differentiating 17 alpha-hydroxylase deficiency from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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17 alpha-hydroxylase deficiency risk factors On the Web |
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American Roentgen Ray Society Images of 17 alpha-hydroxylase deficiency risk factors |
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Risk calculators and risk factors for 17 alpha-hydroxylase deficiency risk factors |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ammu Susheela, M.D. [2], Mehrian Jafarizade, M.D [3]
Overview
The most potent risk factor for the development of 17 alpha-hydroxylase deficiency is a positive family history of 17 alpha-hydroxylase deficiency.
Risk Factors
The most potent risk factor for the development of 17 alpha-hydroxylase deficiency is a positive family history of 17 alpha-hydroxylase deficiency.[1]
References
- ↑ Hannah-Shmouni F, Chen W, Merke DP (2017). "Genetics of Congenital Adrenal Hyperplasia". Endocrinol. Metab. Clin. North Am. 46 (2): 435–458. doi:10.1016/j.ecl.2017.01.008. PMID 28476231.