Heat stroke pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
==Pathophysiology== | |||
Heat stress means perceived discomfort and physiologic strains during heat exposure. A series of physiologic events happen to adapt heat. These events include thermoregulation (with acclimatization), an acute-phase response, and production of heatshock proteins. | |||
===Thermoregulation=== | |||
The core body temperature is set to 37 °C. Thermoregulatory center is located in hypothalamus, every 1 °C in body temperature activates the peripheral and central receptors to maintain core body temperature at 37 °C. | |||
===Pathogenesis=== | |||
Revision as of 13:41, 25 May 2017
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Heat stroke Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Heat stroke pathophysiology On the Web |
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American Roentgen Ray Society Images of Heat stroke pathophysiology |
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Risk calculators and risk factors for Heat stroke pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Seyedmahdi Pahlavani, M.D. [2],Usama Talib, BSc, MD [3]
Overview
Pathophysiology
Heat stress means perceived discomfort and physiologic strains during heat exposure. A series of physiologic events happen to adapt heat. These events include thermoregulation (with acclimatization), an acute-phase response, and production of heatshock proteins.
Thermoregulation
The core body temperature is set to 37 °C. Thermoregulatory center is located in hypothalamus, every 1 °C in body temperature activates the peripheral and central receptors to maintain core body temperature at 37 °C.