Hyperaldosteronism
You don't need to be Editor-In-Chief to add or edit content to WikiDoc. You can begin to add to or edit text on this WikiDoc page by clicking on the edit button at the top of this page. Next enter or edit the information that you would like to appear here. Once you are done editing, scroll down and click the Save page button at the bottom of the page.
| Hyperaldosteronism Classification and external resources | |
 | |
|---|---|
| Aldosterone | |
| ICD-10 | E26. |
| ICD-9 | 255.1 |
| DiseasesDB | 6187 |
| MedlinePlus | 000330 |
| eMedicine | radio/354 |
| MeSH | D006929 |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Phone:617-525-6884
Please Join in Editing This Page and Apply to be an Editor-In-Chief for this topic: There can be one or more than one Editor-In-Chief. You may also apply to be an Associate Editor-In-Chief of one of the subtopics below. Please mail us [2] to indicate your interest in serving either as an Editor-In-Chief of the entire topic or as an Associate Editor-In-Chief for a subtopic. Please be sure to attach your CV and or biographical sketch.
Hyperaldosteronism, also aldosteronism, is a medical condition where too much aldosterone is produced by the adrenal glands, which can lead to lowered levels of potassium in blood.
Types
- E26.0: Primary hyperaldosteronism, often caused by an adrenal cortical adenoma (a tumor), is also known as Conn's syndrome.
- E26.1: Secondary hyperaldosteronism is due to overactivity of the renin-angiotensin system.
In endocrinology, the terms primary and secondary are used to describe the abnormality (e.g. elevated aldosterone) in relation to the defect, i.e. the tumor's location. The adjective primary refers to an abnormality that directly leads to pathology, i.e. aldosteronism is caused by the apparatus that generates aldosterone. Secondary refers to an abnormality that indirectly results in pathology through a predictable physiologic pathway, i.e. a renin producing tumor leads to increased aldosterone, as the body's aldosterone production is normally regulated by renin levels.
Symptoms
It can be asymptomatic, but the following symptoms can be present
- Fatigue
- Headache
- High blood pressure
- Hypokalemia
- Intermittent or temporary paralysis
- Muscle spasms
- Muscle weakness
- Numbness
- Polyuria
- Polydipsia
- Tingling
See also
External links
it:Iperaldosteronismo ja:アルドステロン症
Acknowledgement and Attribution Regarding Sources of Content
Some of the initial content on this page may be incorporated in part from copyleft sources in the public domain including wikis such as Wikipedia and AskDrWiki. Drug information for patients came from the The National Library of Medicine. Infectious disease information may have come from the Centers for Disease Control (CDC). Differential Diagnoses are drawn from clinicians as well as an amalgamation of 3 sources: 1.The Disease Database; 2. Kahan, Scott, Smith, Ellen G. In A Page: Signs and Symptoms. Malden, Massachusetts: Blackwell Publishing, 2004:3; 3. Sailer, Christian, Wasner, Susanne. Differential Diagnosis Pocket. Hermosa Beach, CA: Borm Bruckmeir Publishing LLC, 2002:7 .
